scholarly journals Relationships Between Age, Body Weight, and Season of the Year and the Incidence of Sudden Death Syndrome in Male Broiler Chickens

1988 ◽  
Vol 67 (9) ◽  
pp. 1243-1249 ◽  
Author(s):  
E.E. GARDINER ◽  
J.R. HUNT ◽  
R.C. NEWBERRY ◽  
J.W. HALL
Keyword(s):  
Body Weight ◽  
Sudden Death ◽  
Broiler Chickens ◽  
Sudden Death Syndrome

EFFECT OF DIETARY TAURINE SUPPLEMENTATION ON SUDDEN DEATH SYNDROME IN BROILER CHICKENS

1989 ◽  
Vol 69 (2) ◽  
pp. 509-512 ◽  
Author(s):  
G. L. CAMPBELL ◽  
H. L. CLASSEN
Keyword(s):  
Body Weight ◽  
Sudden Death ◽  
Key Words ◽  
Broiler Chickens ◽  
Total Mortality ◽  
Sudden Death Syndrome ◽  
Taurine Supplementation ◽  
Gain Ratio

Dietary taurine supplementation (0, 0.05, 0.10, or 0.20%) was examined as a means of reducing sudden death syndrome (SDS) in male broiler chickens (624 per treatment) raised to market weight. Total mortality and sudden death syndrome were lower for broilers fed taurine-supplemented diets. The feed-to-gain ratio from 3 to 6 wk decreased linearly with increasing dietary taurine while taurine addition had no effect on 3-wk or 6-wk body weight, or feed-to-gain ratio from 0 to 3 wk. Key words: Sudden death syndrome, taurine, broiler chickens

Diet, total plasma homocysteine concentrations and mortality rates in broiler chickens

2003 ◽  
Vol 83 (3) ◽  
pp. 601-604 ◽  
Author(s):  
S. E. Samuels
Keyword(s):  
Cardiovascular Disease ◽  
Sudden Death ◽  
Broiler Chickens ◽  
Independent Risk Factor ◽  
Total Mortality ◽  
Mortality Rates ◽  
Plasma Homocysteine ◽  
Sudden Death Syndrome ◽  
Total Plasma ◽  
Elevated Plasma

The aim of this study was to determine if total plasma homocysteine (HCY) concentrations and mortality rates due to ascites syndrome and (AS) sudden death syndrome (SDS) in broiler chickens could be lowered by diet. Elevated plasma HCY is an independent risk factor for cardiovascular disease in humans. A total of 828 day-old male broiler chickens (Arbor Acre) were fed, for 6 wk, either a basal practical diet or one supplemented with excess vitamins B6 and B12, folic acid and betaine to stimulate the degradation of HCY. The supplemented diet decreased plasma HCY by 17% (P < 0.05; n = 16 per diet). Total mortality due to AS and SDS was 18% lower in the supplemented diet but this difference was not statistically significant. Key words: Homocysteine, folate, chickens, cardiovascular disease, ascites, sudden death syndrome

Role of cardiac hypoxia in the pathogenesis of sudden death syndrome in broiler chickens – A metabolic and molecular study

2021 ◽  
Author(s):  
Pegah Safaei ◽  
Gholamhossein Khadjeh ◽  
Mohammad Reza Tabandeh ◽  
Keramat Asasi
Keyword(s):  
Sudden Death ◽  
Cardiac Muscle ◽  
Internal Control ◽  
Broiler Chickens ◽  
Sudden Death Syndrome ◽  
Pyruvate Dehydrogenase Kinase ◽  
Cardiac Insufficiency ◽  
Monocarboxylate Transporter ◽  
Pcr Analysis ◽  
Glucose Transporter 1

AbstractSudden death syndrome (SDS) is an economically important disorder in broiler chickens with unknown aetiology. The aim of the present study was to evaluate the metabolic and molecular alterations related to hypoxia in the myocardium of broiler chickens with SDS. Samples from the cardiac muscle of internal control broiler chickens (ICs) (n = 36) and chickens having died of SDS (n = 36) were obtained during the rearing period. The activities of lactate dehydrogenase (LDH) and creatine phosphokinase (CPK) and the concentration of lactate were measured in the cardiac tissue using available commercial kits. The expression of hypoxia-inducing factor 1α (HIF1α), glucose transporter 1 (GLUT1), pyruvate dehydrogenase kinase 4 (PDHK4) and monocarboxylate transporter 4 (MCT4) genes was determined in the myocardium by real-time PCR analysis. The results showed the elevation of lactate level and activities of LDH and CPK in the cardiac muscle of SDS-affected chickens compared with the IC birds (P < 0.05). The cardiac muscle expression of HIF1α, MCT4 and GLUT1 genes was increased, while the PDHK4 mRNA level was decreased in the SDS-affected group compared to those in the IC chickens (P < 0.05). Our results showed that metabolic remodelling associated with hypoxia in the cardiac tissues may have an important role in the pathogenesis of cardiac insufficiency and SDS in broiler chickens.

THE EFFECTS OF DIETARY MICRONUTRIENT, FAT AND PROTEIN COMPONENTS IN PELLETED FEEDS ON THE INCIDENCE OF SUDDEN DEATH SYNDROME AND OTHER TRAITS AMONG MALE BROILER CHICKENS

1984 ◽  
Vol 64 (1) ◽  
pp. 159-164 ◽  
Author(s):  
F. G. PROUDFOOT ◽  
H. W. HULAN ◽  
K. B. McRAE
Keyword(s):  
Sudden Death ◽  
Key Words ◽  
Soybean Meal ◽  
Dietary Protein ◽  
Broiler Chickens ◽  
Sudden Death Syndrome ◽  
Canola Meal ◽  
Protein Supplements ◽  
Protein Components

Three experiments involving 11 600 male broiler chickens sought to determine if the pelleting process affects the dietary micronutrient, fat and protein components to increase the incidence of sudden death syndrome. Processed dietary micronutrient and fat components were not significantly associated with an increase in sudden death syndrome among broiler chickens. In one of the experiments, the incidence of sudden death syndrome was reduced (P < 0.01) when the dietary protein supplements (soybean meal, canola meal and fishmeal) bypassed the pelleting process. Key words: Sudden death syndrome, broiler chickens, feed pelleting, fat, micronutrients, protein supplements

scholarly journals Research Note: Effect of Feed Restriction on Feed Efficiency and Incidence of Sudden Death Syndrome in Broiler Chickens

1988 ◽  
Vol 67 (7) ◽  
pp. 1102-1104 ◽  
Author(s):  
VICTORIA A. BOWES ◽  
R.J. JULIAN ◽  
STEVEN LEESON ◽  
TANYA STIRTZINGER
Keyword(s):  
Sudden Death ◽  
Feed Efficiency ◽  
Broiler Chickens ◽  
Sudden Death Syndrome ◽  
Research Note ◽  
Feed Restriction

scholarly journals Effect of Dietary Lactate and Glucose on the Incidence of Sudden Death Syndrome in Male Broiler Chickens

1990 ◽  
Vol 69 (9) ◽  
pp. 1529-1532 ◽  
Author(s):  
J.P. JACOB ◽  
R. BLAIR ◽  
E.E. GARDINER
Keyword(s):  
Sudden Death ◽  
Broiler Chickens ◽  
Sudden Death Syndrome

scholarly journals Evaluation of lighting programs, diet density, and short term use of mash as compared to crumbled starter to reduce incidence of sudden death syndrome in broiler chicks to 35 d of age

2002 ◽  
Vol 82 (3) ◽  
pp. 375-383 ◽  
Author(s):  
T. A. Scott
Keyword(s):  
Body Weight ◽  
Sudden Death ◽  
Early Growth ◽  
High Density ◽  
The Body ◽  
Sudden Death Syndrome ◽  
Broiler Chicks ◽  
Feed Conversion ◽  
Lower Body Weight ◽  
M 12

Restricted lighting is used commercially to regulate early growth and limit losses associated with sudden death syndrome (SDS), ascites and leg problems. Standard lighting programs may not be suited for broilers marketed at 35 d of age, and promote injury (scratching, i.e., cellulitis) by creating peak feeding periods. This study evaluates four lighting programs, three phases of mash or crumble starter feeding and two diet densities on performance and mortality of broilers marketed at 35 d of age. Two trials each utilized 7200 male broiler (Cobb × Cobb) chicks randomly housed in six (2.75 × 4.90 m) floor pens in each of eight rearing rooms. Four lighting programs were replicated in each trial and consisted of: (a) 23C (23L:1D 0–35 d); (b) 16C (23L:1D 0–3 d, 16L:8D 4–35 d); (c) DDI (delayed decreasing increasing) (23L:1D 0–3 d; 20L:4D 4–6 d, 16L:8D 7–10 d; 8L:16D 11–15 d; 12L:12D 16–20 d; 16L:8 D 21–28 d; and 20L:4 D 29–35 d); and (d) IL (intermittent lighting, provided in four cycles of 6 h d-1 the same total daily hours of light as the DDI program). Two diet densities were applied for the starter and grower diets; high (ME 3170/3200 kcal; CP 251/210 g) or low (ME 3100/3060 kcal; CP 235/195 g kg-1 starter/grower diet, respectively). The two starter types were fed as combinations of mash (M) or crumble (C): (1) M 0–12 d followed by C 12–21 d; (2) C 0–12 d followed by M 12–21 d; and 3) C 0–21 d. The respective grower/finisher density diets were fed in a pelleted form. By 35 d of age the body weight (BW) of broilers maintained under the DDI lighting program was significantly lower than BW of broilers held under the other lighting programs (16Ca > ILab > 23Cb). Overall (0–35 d) feed conversion (corrected for mortality, but not market weight at 35 d) was highest for broilers reared under the 23Ca > ILab > DDIb > 16Cc lighting programs, respectively. SDS (%) mortality was highest for IL (1.68a) = 23C (1.53a) ≥ 16C (1.02ab) ≥ DDI (0.80b) lighting programs. There were no significant differences in 35 d BW or feed conversion of broilers fed mash or crumble starter. Feeding mash diets did lower 21 d BW, but this decrease in early growth did not significantly reduce overall mortality. Broilers fed the low as compared to the high density diets weighed less at 21 d, but weighed significantly more at 35 d, and had a higher (P < 0.05) 0–35 d feed conversion. Low as compared to high density diets did reduce SDS mortality significantly (0.72 vs. 1.81%, respectively). Lighting programs that do provide variable extended dark periods will alleviate broiler mortality as compared to constant (23 or 16 h) or intermittent lighting programs. However, these broilers had a significantly lower body weight when marketed at 35 or less days of age. Key words: Lighting, broilers, mash, diet density, sudden death, ascites

EFFECTS OF DIETARY ASPIRIN (ACETYLSALICYLIC ACID) ON THE INCIDENCE OF SUDDEN DEATH SYNDROME AND THE GENERAL PERFORMANCE OF BROILER CHICKENS

1983 ◽  
Vol 63 (2) ◽  
pp. 469-471 ◽  
Author(s):  
F. G. PROUDFOOT ◽  
H. W. HULAN
Keyword(s):  
Growth Rate ◽  
Sudden Death ◽  
Deleterious Effect ◽  
Broiler Chickens ◽  
Dietary Supplementation ◽  
Sudden Death Syndrome ◽  
The Other ◽  
Reduced Body ◽  
Body Weights ◽  
Overall Mortality

A total of 2400 broiler chickens were used in four experiments to estimate the effects of using 0.04, 0.08 and 0.16% aspirin (ASA) as dietary supplements. Not only did the ASA dietary supplementation have no significant (P < 0.05) beneficial effect on the incidence of sudden death syndrome but it also had a deleterious effect on overall mortality which was significant (P < 0.01) in two of the four experiments. Furthermore, the addition of ASA at 0.16% to the diet resulted in reduced body weights which were significant at P < 0.05 in two experiments and at P < 0.10 in the other two experiments. Key words: Aspirin, sudden death syndrome, broiler chickens, growth rate

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