EFFECTS OF DIETARY ASPIRIN (ACETYLSALICYLIC ACID) ON THE INCIDENCE OF SUDDEN DEATH SYNDROME AND THE GENERAL PERFORMANCE OF BROILER CHICKENS

1983 ◽  
Vol 63 (2) ◽  
pp. 469-471 ◽  
Author(s):  
F. G. PROUDFOOT ◽  
H. W. HULAN
Keyword(s):  
Growth Rate ◽  
Sudden Death ◽  
Deleterious Effect ◽  
Broiler Chickens ◽  
Dietary Supplementation ◽  
Sudden Death Syndrome ◽  
The Other ◽  
Reduced Body ◽  
Body Weights ◽  
Overall Mortality

A total of 2400 broiler chickens were used in four experiments to estimate the effects of using 0.04, 0.08 and 0.16% aspirin (ASA) as dietary supplements. Not only did the ASA dietary supplementation have no significant (P < 0.05) beneficial effect on the incidence of sudden death syndrome but it also had a deleterious effect on overall mortality which was significant (P < 0.01) in two of the four experiments. Furthermore, the addition of ASA at 0.16% to the diet resulted in reduced body weights which were significant at P < 0.05 in two experiments and at P < 0.10 in the other two experiments. Key words: Aspirin, sudden death syndrome, broiler chickens, growth rate

scholarly journals Interrelationships between biotin, choline and other B-vitamins and the occurrence of fatty liver and kidney syndrome and sudden death syndrome in broiler chickens

1982 ◽  
Vol 48 (1) ◽  
pp. 177-184 ◽  
Author(s):  
C. C. Whitehead ◽  
C. J. Randall
Keyword(s):  
Sudden Death ◽  
Fatty Liver ◽  
Broiler Chickens ◽  
Dietary Supplementation ◽  
B Vitamins ◽  
Sudden Death Syndrome ◽  
Post Mortem ◽  
Deficient Diet ◽  
Liver And Kidney ◽  
B Vitamin

1. Addition of supplemental choline to a biotin-deficient diet decreased the biotin status of chicks and increased mortality from fatty liver and kidney syndrome (FLKS).2. Mortality was also increased by dietary supplementation with a mixture of other B-vitamins, excluding biotin, and was highest when the choline and B-vitamin supplements were combined.3. The occurrence of sudden death syndrome (SDS) was unaffected by dietary biotin concentration.4. A previously unreported condition was observed in which birds died showing post-mortem signs characteristic of both FLKS and SDS and whose occurrence was related to the biotin status of the chicks.

FEED TEXTURE EFFECTS ON THE PERFORMANCE OF ROASTER CHICKENS

1989 ◽  
Vol 69 (3) ◽  
pp. 801-807 ◽  
Author(s):  
F. G. PROUDFOOT ◽  
H. W. HULAN
Keyword(s):  
Particle Size ◽  
Sudden Death ◽  
Key Words ◽  
Broiler Chickens ◽  
Sudden Death Syndrome ◽  
Feed Conversion ◽  
Body Weights ◽  
Four Blocks ◽  
Dietary Treatments

Two experiments were conducted, each experiment involved 1600 day-old Arbor Acre male broiler chickens which were assigned to 20 pens divided into four blocks of five pens each with five feed texture treatments assigned to the pens within each block. The five treatments were (1) All mash — corn and wheat finely ground, (2) All mash — corn and wheat coarsely ground, (3) All mash — corn and wheat very coarsely rolled, (4) same as no. 1 but crumbled or pelleted and (5) same as no. 2 but crumbled or pelleted. Although feed texture had no overall significant (P > 0.05) effect on mortality, mortality was higher among birds fed the crumble-pellet diets compared with those fed the all-mash diets, this higher mortality was due to a higher incidence of Sudden Death Syndrome. Finely and very coarsely ground diets fed as mash resulted in significantly poorer feed conversion at both 21 d (P < 0.001) and 49 d of age (P < 0.01); however, there was no significant (P < 0.05) effect of feed texture on feed conversion at 63 d of age. Body weights were significantly (P < 0.01 to P < 0.001) affected by feed texture at 21, 49 and 63 d of age with the feeding of finely ground mash resulting in the lowest body weights with the heaviest final body weights exhibited by birds fed diets which were either fine or coarse ground mashes which had gone through the crumble and/or pellet process. Feed texture dietary treatments had no effect on the incidence of leg abnormalities. Monetary returns per bird started were significantly (P < 0.05) depressed by the feeding of finely ground mash to chickens while other feed texture treatments were not significantly (P > 0.05) different. Key words: Chicken (roaster), feed texture, ingredient particle size

scholarly journals Effects of the physical form of diet on growth performance, ascites and sudden death syndrome incidences in broiler chickens

2020 ◽  
Vol 71 (2) ◽  
pp. 2087
Author(s):  
M. AZIZIAN ◽  
A.A. SAKI
Keyword(s):  
Weight Gain ◽  
Sudden Death ◽  
Broiler Chickens ◽  
Low Density Lipoprotein ◽  
Density Lipoprotein ◽  
Carcass Characteristics ◽  
Sudden Death Syndrome ◽  
The Other ◽  
Low Density ◽  
Physical Form

This experiment was conducted to determine the effects of the diet physical form, Mash (M), Pellet (P) and Extruded (E) on the growth performance, carcass characteristics and metabolic disorders ascites (AS) and as well as sudden death syndrome (SDS) in the broiler chickens. In this respect, feed intake (FI), weight gain (WG) and mortalitywere recorded throughout the experiment and biochemical parameters, hematology and carcass characteristics were tested at 35 and 42 days of age respectively. The results showed that with the increase of the average daily weight gain (ADWG) (p<0.01), the relative breast weight to the carcass weight (p<0.05), better feed conversion ratio (p<0.01), the lower relative cecum weight and gizzard to the carcass weight (p<0.05) were observed by applying the E diet form, as compared with the other treatments. A significant increase in the average daily feed intake (ADFI) was also observed by using the P diet form (p<0.01). Hematological parameters including hemoglobin (Hb), hematocrit (HCT), urea, uric acid, triglyceride, the ratio of low-density lipoprotein to high-density lipoprotein (LDL/HDL), very low-density lipoprotein (VLDL), enzyme activities of aspartate aminotransferase(AST) and alanine aminotransferase (ALT) of the blood serum were lowered by applying the M treatment, as compared with the other treatments (p<0.05). The results indicated that the performance and carcass characteristics were improved by the E and P diet forms; also, with raising the hematology parameters in these treatments, the mortality of ascites and SDS was increased.

Diet, total plasma homocysteine concentrations and mortality rates in broiler chickens

2003 ◽  
Vol 83 (3) ◽  
pp. 601-604 ◽  
Author(s):  
S. E. Samuels
Keyword(s):  
Cardiovascular Disease ◽  
Sudden Death ◽  
Broiler Chickens ◽  
Independent Risk Factor ◽  
Total Mortality ◽  
Mortality Rates ◽  
Plasma Homocysteine ◽  
Sudden Death Syndrome ◽  
Total Plasma ◽  
Elevated Plasma

The aim of this study was to determine if total plasma homocysteine (HCY) concentrations and mortality rates due to ascites syndrome and (AS) sudden death syndrome (SDS) in broiler chickens could be lowered by diet. Elevated plasma HCY is an independent risk factor for cardiovascular disease in humans. A total of 828 day-old male broiler chickens (Arbor Acre) were fed, for 6 wk, either a basal practical diet or one supplemented with excess vitamins B6 and B12, folic acid and betaine to stimulate the degradation of HCY. The supplemented diet decreased plasma HCY by 17% (P < 0.05; n = 16 per diet). Total mortality due to AS and SDS was 18% lower in the supplemented diet but this difference was not statistically significant. Key words: Homocysteine, folate, chickens, cardiovascular disease, ascites, sudden death syndrome

Role of cardiac hypoxia in the pathogenesis of sudden death syndrome in broiler chickens – A metabolic and molecular study

2021 ◽  
Author(s):  
Pegah Safaei ◽  
Gholamhossein Khadjeh ◽  
Mohammad Reza Tabandeh ◽  
Keramat Asasi
Keyword(s):  
Sudden Death ◽  
Cardiac Muscle ◽  
Internal Control ◽  
Broiler Chickens ◽  
Sudden Death Syndrome ◽  
Pyruvate Dehydrogenase Kinase ◽  
Cardiac Insufficiency ◽  
Monocarboxylate Transporter ◽  
Pcr Analysis ◽  
Glucose Transporter 1

AbstractSudden death syndrome (SDS) is an economically important disorder in broiler chickens with unknown aetiology. The aim of the present study was to evaluate the metabolic and molecular alterations related to hypoxia in the myocardium of broiler chickens with SDS. Samples from the cardiac muscle of internal control broiler chickens (ICs) (n = 36) and chickens having died of SDS (n = 36) were obtained during the rearing period. The activities of lactate dehydrogenase (LDH) and creatine phosphokinase (CPK) and the concentration of lactate were measured in the cardiac tissue using available commercial kits. The expression of hypoxia-inducing factor 1α (HIF1α), glucose transporter 1 (GLUT1), pyruvate dehydrogenase kinase 4 (PDHK4) and monocarboxylate transporter 4 (MCT4) genes was determined in the myocardium by real-time PCR analysis. The results showed the elevation of lactate level and activities of LDH and CPK in the cardiac muscle of SDS-affected chickens compared with the IC birds (P < 0.05). The cardiac muscle expression of HIF1α, MCT4 and GLUT1 genes was increased, while the PDHK4 mRNA level was decreased in the SDS-affected group compared to those in the IC chickens (P < 0.05). Our results showed that metabolic remodelling associated with hypoxia in the cardiac tissues may have an important role in the pathogenesis of cardiac insufficiency and SDS in broiler chickens.

Concentrations of glucose, albumin, inorganic phosphate and sodium in the blood of beef calves at 2 ½ months of age and their relationships with subsequent weight gain

1980 ◽  
Vol 94 (1) ◽  
pp. 95-104 ◽  
Author(s):  
G. J. Rowlands
Keyword(s):  
Growth Rate ◽  
Blood Glucose ◽  
Growth Rates ◽  
Inorganic Phosphate ◽  
The Body ◽  
The Other ◽  
Growth Depression ◽  
Blood Samples ◽  
Beef Calves ◽  
Body Weights

SummaryBlood samples were taken at 9, 10 and 11 weeks of age from 230 male Hereford × Friesian calves, the progeny of 12 Hereford bulls. Concentrations of blood glucose, serum albumin, inorganic phosphate and sodium were measured and correlated with body weights and growth rates until slaughter at 19½ months of age.Correlations between growth rates and glucose concentrations (0·44) and between growth rates and albumin concentrations (0·38) were observed until 4 months of age. Similar correlations between body weights and blood glucose or albumin concentrations persisted until 6 months of age. By 10 months, however, the correlations had begun to decline, and by slaughter they had become insignificant.Correlations between growth rates and inorganic phosphate or sodium concentrations were smaller, and also decreased with age.The effect of hypoglycaemia on growth rate was compared with the effect of enzootic pneumonia. Although the 10% of calves with the lowest glucose concentrations were growing 24% more slowly than the other calves at the time of sampling, this growth depression was not related to pneumonia, and they subsequently made up for most of the early loss of growth.Three blood samples were also taken from 22 Hereford bulls. No significant correlations were observed between the blood composition of the bulls and the body weights or weight gains of their progeny.

scholarly journals A Note on Fatty Acids Profile of Meat from Broiler Chickens Supplemented with Inorganic or Organic Selenium

2017 ◽  
Vol 2017 ◽  
pp. 1-8 ◽  
Author(s):  
Marta del Puerto ◽  
M. Cristina Cabrera ◽  
Ali Saadoun
Keyword(s):  
Fatty Acids ◽  
Broiler Chickens ◽  
Sodium Selenite ◽  
Dietary Supplementation ◽  
Potential Effect ◽  
The Other ◽  
Fatty Acids Composition ◽  
Organic Selenium ◽  
Fatty Acids Profile ◽  
Health Promoting

This investigation evaluated, in broiler chickensPectoralisandGastrocnemiusmuscles, the effect of the dietary supplementation with sodium selenite (0.3 ppm) versus selenomethionine (0.3 ppm), on the fatty acids composition, lipids indices, and enzymes indexes for desaturase, elongase, and thioesterase. The selenium reduced, in both muscles, the content of atherogenic fatty acids, C14:0 and C16:0, while it increased the C18:1 level. On the other hand, selenium increased, in both muscles, the content of C18:3n3 and EPA, but not DPA and DHA. No selenium effect was detected for PUFA/SFA, n-6, n-3, n-6/n-3, and atherogenic and thrombogenic indices. As for the enzyme indexes, a selenium effect is only detected for thioesterase. Taken together, the results highlight the potential effect of dietary selenium, mainly selenomethionine, in the modulation of the composition of fatty acids in chicken meat, in particular, reducing the content of atherogenic fatty acids and increasing the health promoting n-3 PUFA.

THE EFFECTS OF DIETARY MICRONUTRIENT, FAT AND PROTEIN COMPONENTS IN PELLETED FEEDS ON THE INCIDENCE OF SUDDEN DEATH SYNDROME AND OTHER TRAITS AMONG MALE BROILER CHICKENS

1984 ◽  
Vol 64 (1) ◽  
pp. 159-164 ◽  
Author(s):  
F. G. PROUDFOOT ◽  
H. W. HULAN ◽  
K. B. McRAE
Keyword(s):  
Sudden Death ◽  
Key Words ◽  
Soybean Meal ◽  
Dietary Protein ◽  
Broiler Chickens ◽  
Sudden Death Syndrome ◽  
Canola Meal ◽  
Protein Supplements ◽  
Protein Components

Three experiments involving 11 600 male broiler chickens sought to determine if the pelleting process affects the dietary micronutrient, fat and protein components to increase the incidence of sudden death syndrome. Processed dietary micronutrient and fat components were not significantly associated with an increase in sudden death syndrome among broiler chickens. In one of the experiments, the incidence of sudden death syndrome was reduced (P < 0.01) when the dietary protein supplements (soybean meal, canola meal and fishmeal) bypassed the pelleting process. Key words: Sudden death syndrome, broiler chickens, feed pelleting, fat, micronutrients, protein supplements

scholarly journals Research Note: Effect of Feed Restriction on Feed Efficiency and Incidence of Sudden Death Syndrome in Broiler Chickens

1988 ◽  
Vol 67 (7) ◽  
pp. 1102-1104 ◽  
Author(s):  
VICTORIA A. BOWES ◽  
R.J. JULIAN ◽  
STEVEN LEESON ◽  
TANYA STIRTZINGER
Keyword(s):  
Sudden Death ◽  
Feed Efficiency ◽  
Broiler Chickens ◽  
Sudden Death Syndrome ◽  
Research Note ◽  
Feed Restriction

scholarly journals Effect of Dietary Lactate and Glucose on the Incidence of Sudden Death Syndrome in Male Broiler Chickens

1990 ◽  
Vol 69 (9) ◽  
pp. 1529-1532 ◽  
Author(s):  
J.P. JACOB ◽  
R. BLAIR ◽  
E.E. GARDINER
Keyword(s):  
Sudden Death ◽  
Broiler Chickens ◽  
Sudden Death Syndrome
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