EFFECT OF DIETARY TAURINE SUPPLEMENTATION ON SUDDEN DEATH SYNDROME IN BROILER CHICKENS

1989 ◽  
Vol 69 (2) ◽  
pp. 509-512 ◽  
Author(s):  
G. L. CAMPBELL ◽  
H. L. CLASSEN
Keyword(s):  
Body Weight ◽  
Sudden Death ◽  
Key Words ◽  
Broiler Chickens ◽  
Total Mortality ◽  
Sudden Death Syndrome ◽  
Taurine Supplementation ◽  
Gain Ratio

Dietary taurine supplementation (0, 0.05, 0.10, or 0.20%) was examined as a means of reducing sudden death syndrome (SDS) in male broiler chickens (624 per treatment) raised to market weight. Total mortality and sudden death syndrome were lower for broilers fed taurine-supplemented diets. The feed-to-gain ratio from 3 to 6 wk decreased linearly with increasing dietary taurine while taurine addition had no effect on 3-wk or 6-wk body weight, or feed-to-gain ratio from 0 to 3 wk. Key words: Sudden death syndrome, taurine, broiler chickens

Diet, total plasma homocysteine concentrations and mortality rates in broiler chickens

2003 ◽  
Vol 83 (3) ◽  
pp. 601-604 ◽  
Author(s):  
S. E. Samuels
Keyword(s):  
Cardiovascular Disease ◽  
Sudden Death ◽  
Broiler Chickens ◽  
Independent Risk Factor ◽  
Total Mortality ◽  
Mortality Rates ◽  
Plasma Homocysteine ◽  
Sudden Death Syndrome ◽  
Total Plasma ◽  
Elevated Plasma

The aim of this study was to determine if total plasma homocysteine (HCY) concentrations and mortality rates due to ascites syndrome and (AS) sudden death syndrome (SDS) in broiler chickens could be lowered by diet. Elevated plasma HCY is an independent risk factor for cardiovascular disease in humans. A total of 828 day-old male broiler chickens (Arbor Acre) were fed, for 6 wk, either a basal practical diet or one supplemented with excess vitamins B6 and B12, folic acid and betaine to stimulate the degradation of HCY. The supplemented diet decreased plasma HCY by 17% (P < 0.05; n = 16 per diet). Total mortality due to AS and SDS was 18% lower in the supplemented diet but this difference was not statistically significant. Key words: Homocysteine, folate, chickens, cardiovascular disease, ascites, sudden death syndrome

THE EFFECTS OF DIETARY MICRONUTRIENT, FAT AND PROTEIN COMPONENTS IN PELLETED FEEDS ON THE INCIDENCE OF SUDDEN DEATH SYNDROME AND OTHER TRAITS AMONG MALE BROILER CHICKENS

1984 ◽  
Vol 64 (1) ◽  
pp. 159-164 ◽  
Author(s):  
F. G. PROUDFOOT ◽  
H. W. HULAN ◽  
K. B. McRAE
Keyword(s):  
Sudden Death ◽  
Key Words ◽  
Soybean Meal ◽  
Dietary Protein ◽  
Broiler Chickens ◽  
Sudden Death Syndrome ◽  
Canola Meal ◽  
Protein Supplements ◽  
Protein Components

Three experiments involving 11 600 male broiler chickens sought to determine if the pelleting process affects the dietary micronutrient, fat and protein components to increase the incidence of sudden death syndrome. Processed dietary micronutrient and fat components were not significantly associated with an increase in sudden death syndrome among broiler chickens. In one of the experiments, the incidence of sudden death syndrome was reduced (P < 0.01) when the dietary protein supplements (soybean meal, canola meal and fishmeal) bypassed the pelleting process. Key words: Sudden death syndrome, broiler chickens, feed pelleting, fat, micronutrients, protein supplements

scholarly journals Relationships Between Age, Body Weight, and Season of the Year and the Incidence of Sudden Death Syndrome in Male Broiler Chickens

1988 ◽  
Vol 67 (9) ◽  
pp. 1243-1249 ◽  
Author(s):  
E.E. GARDINER ◽  
J.R. HUNT ◽  
R.C. NEWBERRY ◽  
J.W. HALL
Keyword(s):  
Body Weight ◽  
Sudden Death ◽  
Broiler Chickens ◽  
Sudden Death Syndrome

FEED TEXTURE EFFECTS ON THE PERFORMANCE OF ROASTER CHICKENS

1989 ◽  
Vol 69 (3) ◽  
pp. 801-807 ◽  
Author(s):  
F. G. PROUDFOOT ◽  
H. W. HULAN
Keyword(s):  
Particle Size ◽  
Sudden Death ◽  
Key Words ◽  
Broiler Chickens ◽  
Sudden Death Syndrome ◽  
Feed Conversion ◽  
Body Weights ◽  
Four Blocks ◽  
Dietary Treatments

Two experiments were conducted, each experiment involved 1600 day-old Arbor Acre male broiler chickens which were assigned to 20 pens divided into four blocks of five pens each with five feed texture treatments assigned to the pens within each block. The five treatments were (1) All mash — corn and wheat finely ground, (2) All mash — corn and wheat coarsely ground, (3) All mash — corn and wheat very coarsely rolled, (4) same as no. 1 but crumbled or pelleted and (5) same as no. 2 but crumbled or pelleted. Although feed texture had no overall significant (P > 0.05) effect on mortality, mortality was higher among birds fed the crumble-pellet diets compared with those fed the all-mash diets, this higher mortality was due to a higher incidence of Sudden Death Syndrome. Finely and very coarsely ground diets fed as mash resulted in significantly poorer feed conversion at both 21 d (P < 0.001) and 49 d of age (P < 0.01); however, there was no significant (P < 0.05) effect of feed texture on feed conversion at 63 d of age. Body weights were significantly (P < 0.01 to P < 0.001) affected by feed texture at 21, 49 and 63 d of age with the feeding of finely ground mash resulting in the lowest body weights with the heaviest final body weights exhibited by birds fed diets which were either fine or coarse ground mashes which had gone through the crumble and/or pellet process. Feed texture dietary treatments had no effect on the incidence of leg abnormalities. Monetary returns per bird started were significantly (P < 0.05) depressed by the feeding of finely ground mash to chickens while other feed texture treatments were not significantly (P > 0.05) different. Key words: Chicken (roaster), feed texture, ingredient particle size

EFFECT OF DIETARY RESERPINE ON THE INCIDENCE OF SUDDEN DEATH SYNDROME IN CHICKENS

1984 ◽  
Vol 64 (4) ◽  
pp. 1015-1018 ◽  
Author(s):  
E. E. GARDINER ◽  
J. R. HUNT
Keyword(s):  
Body Weight ◽  
Sudden Death ◽  
Body Weight Gain ◽  
Total Mortality ◽  
Sudden Death Syndrome ◽  
Lower Body ◽  
Feed Conversion ◽  
Lower Body Weight ◽  
Percent Mortality ◽  
Meat Type

Two experiments were conducted to determine the effects of various levels of dietary reserpine on growth, feed conversion, percent mortality and percent mortality due to sudden death syndrome (SDS) of meat-type chickens to 10 wk of age. In one experiment, dietary reserpine (0.0, 0.5, 1.0 and 1.5 mg/kg) had no significant effect on body weight gain but reduced feed:gain ratios with the two highest levels at 3 wk and at all levels at 7 and 10 wk. In the second experiment (0.0, 0.75, 1.5 and 3.0 mg/kg), dietary reserpine resulted in lower body weight at 3, 7 and 10 wk of age while feed:gain ratios at 7 or 10 wk were not affected. In both experiments neither total mortality nor mortality due to SDS was influenced by dietary reserpine. Key words: Reserpine, sudden death syndrome, broilers, roasters, growth, feed:gain

Role of cardiac hypoxia in the pathogenesis of sudden death syndrome in broiler chickens – A metabolic and molecular study

2021 ◽  
Author(s):  
Pegah Safaei ◽  
Gholamhossein Khadjeh ◽  
Mohammad Reza Tabandeh ◽  
Keramat Asasi
Keyword(s):  
Sudden Death ◽  
Cardiac Muscle ◽  
Internal Control ◽  
Broiler Chickens ◽  
Sudden Death Syndrome ◽  
Pyruvate Dehydrogenase Kinase ◽  
Cardiac Insufficiency ◽  
Monocarboxylate Transporter ◽  
Pcr Analysis ◽  
Glucose Transporter 1

AbstractSudden death syndrome (SDS) is an economically important disorder in broiler chickens with unknown aetiology. The aim of the present study was to evaluate the metabolic and molecular alterations related to hypoxia in the myocardium of broiler chickens with SDS. Samples from the cardiac muscle of internal control broiler chickens (ICs) (n = 36) and chickens having died of SDS (n = 36) were obtained during the rearing period. The activities of lactate dehydrogenase (LDH) and creatine phosphokinase (CPK) and the concentration of lactate were measured in the cardiac tissue using available commercial kits. The expression of hypoxia-inducing factor 1α (HIF1α), glucose transporter 1 (GLUT1), pyruvate dehydrogenase kinase 4 (PDHK4) and monocarboxylate transporter 4 (MCT4) genes was determined in the myocardium by real-time PCR analysis. The results showed the elevation of lactate level and activities of LDH and CPK in the cardiac muscle of SDS-affected chickens compared with the IC birds (P < 0.05). The cardiac muscle expression of HIF1α, MCT4 and GLUT1 genes was increased, while the PDHK4 mRNA level was decreased in the SDS-affected group compared to those in the IC chickens (P < 0.05). Our results showed that metabolic remodelling associated with hypoxia in the cardiac tissues may have an important role in the pathogenesis of cardiac insufficiency and SDS in broiler chickens.

scholarly journals Research Note: Effect of Feed Restriction on Feed Efficiency and Incidence of Sudden Death Syndrome in Broiler Chickens

1988 ◽  
Vol 67 (7) ◽  
pp. 1102-1104 ◽  
Author(s):  
VICTORIA A. BOWES ◽  
R.J. JULIAN ◽  
STEVEN LEESON ◽  
TANYA STIRTZINGER
Keyword(s):  
Sudden Death ◽  
Feed Efficiency ◽  
Broiler Chickens ◽  
Sudden Death Syndrome ◽  
Research Note ◽  
Feed Restriction

scholarly journals Effect of Dietary Lactate and Glucose on the Incidence of Sudden Death Syndrome in Male Broiler Chickens

1990 ◽  
Vol 69 (9) ◽  
pp. 1529-1532 ◽  
Author(s):  
J.P. JACOB ◽  
R. BLAIR ◽  
E.E. GARDINER
Keyword(s):  
Sudden Death ◽  
Broiler Chickens ◽  
Sudden Death Syndrome

Whole canola/pea and whole canola/canola meal blends in diets for broiler chickens 2. Determination of optimal inclusion levels

1997 ◽  
Vol 77 (2) ◽  
pp. 191-195 ◽  
Author(s):  
Y. O. Fasina ◽  
G. L. Campbell
Keyword(s):  
Body Weight ◽  
Weight Gain ◽  
Key Words ◽  
Feed Intake ◽  
Broiler Chickens ◽  
Body Weight Gain ◽  
Canola Meal ◽  
Canola Seed ◽  
Finisher Diets

Whole canola/pea (WCP, 1:1 wt/wt) or whole canola/canola meal (WCC, 3:1 wt/wt) were heat-processed using a commercial pellet mill prior to incorporation in broiler starter (0–20 d) and finisher diets (20–40 d). The pelleted blends readily disintegrated in the preparation of the mash diets. Body weight gain of broilers (0–40 d) fed WCP (0, 10, 20, or 30%) or WCC (0, 5, 10, or 15%) declined linearly with level of dietary inclusion (P < 0.01). Feed intake declined curvilinearly (P < 0.01) with both blends, with lower intake most apparent at the higher inclusion levels. Feed/gain increased for broilers fed WCP or WCC (P < 0.01). Overall broiler mortality declined incrementally with inclusion of WCP (P < 0.07) or WCC (P < 0.03) in the diet. Although there was no distinct limitation on the dietary inclusion levels of either blend, the low feed intake with broilers fed 15% WCC indicated that its level should not exceed 10% of the diet. Key words: Canola seed, canola meal, peas, broiler chickens

scholarly journals Evaluation of lighting programs, diet density, and short term use of mash as compared to crumbled starter to reduce incidence of sudden death syndrome in broiler chicks to 35 d of age

2002 ◽  
Vol 82 (3) ◽  
pp. 375-383 ◽  
Author(s):  
T. A. Scott
Keyword(s):  
Body Weight ◽  
Sudden Death ◽  
Early Growth ◽  
High Density ◽  
The Body ◽  
Sudden Death Syndrome ◽  
Broiler Chicks ◽  
Feed Conversion ◽  
Lower Body Weight ◽  
M 12

Restricted lighting is used commercially to regulate early growth and limit losses associated with sudden death syndrome (SDS), ascites and leg problems. Standard lighting programs may not be suited for broilers marketed at 35 d of age, and promote injury (scratching, i.e., cellulitis) by creating peak feeding periods. This study evaluates four lighting programs, three phases of mash or crumble starter feeding and two diet densities on performance and mortality of broilers marketed at 35 d of age. Two trials each utilized 7200 male broiler (Cobb × Cobb) chicks randomly housed in six (2.75 × 4.90 m) floor pens in each of eight rearing rooms. Four lighting programs were replicated in each trial and consisted of: (a) 23C (23L:1D 0–35 d); (b) 16C (23L:1D 0–3 d, 16L:8D 4–35 d); (c) DDI (delayed decreasing increasing) (23L:1D 0–3 d; 20L:4D 4–6 d, 16L:8D 7–10 d; 8L:16D 11–15 d; 12L:12D 16–20 d; 16L:8 D 21–28 d; and 20L:4 D 29–35 d); and (d) IL (intermittent lighting, provided in four cycles of 6 h d-1 the same total daily hours of light as the DDI program). Two diet densities were applied for the starter and grower diets; high (ME 3170/3200 kcal; CP 251/210 g) or low (ME 3100/3060 kcal; CP 235/195 g kg-1 starter/grower diet, respectively). The two starter types were fed as combinations of mash (M) or crumble (C): (1) M 0–12 d followed by C 12–21 d; (2) C 0–12 d followed by M 12–21 d; and 3) C 0–21 d. The respective grower/finisher density diets were fed in a pelleted form. By 35 d of age the body weight (BW) of broilers maintained under the DDI lighting program was significantly lower than BW of broilers held under the other lighting programs (16Ca > ILab > 23Cb). Overall (0–35 d) feed conversion (corrected for mortality, but not market weight at 35 d) was highest for broilers reared under the 23Ca > ILab > DDIb > 16Cc lighting programs, respectively. SDS (%) mortality was highest for IL (1.68a) = 23C (1.53a) ≥ 16C (1.02ab) ≥ DDI (0.80b) lighting programs. There were no significant differences in 35 d BW or feed conversion of broilers fed mash or crumble starter. Feeding mash diets did lower 21 d BW, but this decrease in early growth did not significantly reduce overall mortality. Broilers fed the low as compared to the high density diets weighed less at 21 d, but weighed significantly more at 35 d, and had a higher (P < 0.05) 0–35 d feed conversion. Low as compared to high density diets did reduce SDS mortality significantly (0.72 vs. 1.81%, respectively). Lighting programs that do provide variable extended dark periods will alleviate broiler mortality as compared to constant (23 or 16 h) or intermittent lighting programs. However, these broilers had a significantly lower body weight when marketed at 35 or less days of age. Key words: Lighting, broilers, mash, diet density, sudden death, ascites

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