scholarly journals Hyperbaric Oxygen Exposures at 3 and 4 Atmospheres Absolute Pressure for Experimental Gas Gangrene: Succinate Protection Against Oxygen Toxicity

1972 ◽  
Vol 2 (5) ◽  
pp. 384-389
Author(s):  
G. B. Hill
Keyword(s):  
Hyperbaric Oxygen ◽  
Oxygen Toxicity ◽  
Gas Gangrene ◽  
Absolute Pressure

Hyperoxic myopia: a case series of four divers

2020 ◽  
pp. 261-265
Author(s):  
Jonathan W. Brügger ◽  
Glenn A. Rauscher ◽  
John P. Florian ◽  
Keyword(s):  
Hyperbaric Oxygen ◽  
Prolonged Exposure ◽  
Case Series ◽  
Oxygen Toxicity ◽  
Hyperbaric Oxygen Treatment ◽  
Subjective Data ◽  
Residual Symptoms ◽  
Oxygen Treatment ◽  
Blurry Vision ◽  
Record Review

Hyperoxic myopia is a phenomenon reported in individuals who have prolonged exposure to an increased partial pressure of oxygen (PO2) and subsequently have a myopic (nearsighted) change in their vision. To date, there are numerous accounts of hyperoxic myopia in dry hyperbaric oxygen treatment patients; however, there have been only three confirmed cases reported in wet divers. This case series adds four confirmed cases of hyperoxic myopia in wet divers using 1.35 atmospheres (ATM) PO2 at the Navy Experimental Diving Unit (NEDU). The four divers involved were the first author’s patients at NEDU. Conditions for two divers were confirmed via record review, whereas the other two divers were diagnosed by the first author. All subjects were interviewed to correlate subjective data with objective findings. Each subject completed five consecutive six-hour hyperoxic (PO2 of 1.35 ATM) dives with 18-hour surface intervals. Each individual was within the U. S. Navy Dive Manual’s standards for general health. Visual acuity was measured prior to diving. Within three to four days after diving, the individuals reported blurry vision with an associated myopic refraction shift. Each diver had spontaneous resolution of his myopia over the next two to three weeks, with no significant residual symptoms. The divers in this case series were exposed to an increased PO2 (1.35 ATM for 30 hours over five days), a lesser exposure than that in other reports of hyperoxic myopia in wet divers diagnosed with hyperoxic myopia (1.3-1.6 ATM for 45-85 hours in 12-18 days). Furthermore, this pulse of exposure was more concentrated than typically seen with traditional hyperbaric oxygen therapy. Hyperoxic myopia continues to be a risk for those conducting intensive diving with a PO2 between 1.3-1.6 ATM. Additional investigation is warranted to better define risk factors and PO2 limits regarding ocular oxygen toxicity.

Succinate: Protective Agent against Hyperbaric Oxygen Toxicity

Science ◽  
1965 ◽  
Vol 150 (3705) ◽  
pp. 1830-1831 ◽  
Author(s):  
A. P. Sanders ◽  
I. H. Hall ◽  
B. Woodhall
Keyword(s):  
Hyperbaric Oxygen ◽  
Oxygen Toxicity ◽  
Protective Agent

Hyperbaric oxygen in anaerobic infection of the mediastinum

1975 ◽  
Vol 89 (11) ◽  
pp. 1147-1150 ◽  
Author(s):  
I. Monies-Chass ◽  
H. Z. Joachims ◽  
M. M. Altman
Keyword(s):  
Hyperbaric Oxygen ◽  
Anaerobic Bacteria ◽  
Gas Gangrene ◽  
Causative Organism ◽  
Anaerobic Infection ◽  
Road Accidents ◽  
War Wounds ◽  
Broken Bones ◽  
Surgical Measures ◽  
Oxygenation Level

AbstractIN the last decade hyperbaric oxygen has been recognized as an important therapeutic tool in a variety of instances in which either destruction of anaerobic bacteria is urgent or an improvement in the oxygenation level is mandatory. We have usede it successfully in a case of mediastinal anaerobic infection (gas gangrene) after medical and surgical measures had failed to eradicate the disease.The causative organism of gas gangrene, Clostridium perfringens (Welchii), is widely distributed. It may be cultured from the soil, house dust, human skin and faeces, etc. For this reason infection with Clostridium is practically inevitable whenever suitable conditions arise. As an anaerobic bacterium Clostridium Welchii multiplies readily in damaged tissues without cantact with the air and devoid of a normal blood supply. This occurs especially in road accidents and war wounds in which broken bones and crushed muscles provide a suitable medium for the infection (Altmeier 1965). Sometimes this infection can occur too after abdominal or gynaecological operations (Hitchcock, 1965).We present here a case of mediastinal gas gangrene which was caused by perforation of the oesophagus by a swallowed foreign body.

HYPERBARIC OXYGEN USED IN THE TREATMENT OF GAS GANGRENE

1967 ◽  
Vol 2 (22) ◽  
pp. 991-993
Author(s):  
G. J. A. Bayliss ◽  
Cecil Cass
Keyword(s):  
Hyperbaric Oxygen ◽  
Gas Gangrene

Modification of hyperbaric oxygen toxicity by experimental venous admixture.

1967 ◽  
Vol 23 (6) ◽  
pp. 954-963 ◽  
Author(s):  
P M Winter ◽  
R K Gupta ◽  
A H Michalski ◽  
E H Lanphier
Keyword(s):  
Hyperbaric Oxygen ◽  
Oxygen Toxicity ◽  
Venous Admixture

Hyperbaric oxygen used in the treatment of gas gangrene in a dog

1976 ◽  
Vol 17 (11) ◽  
pp. 759-764 ◽  
Author(s):  
N. A. COOPER ◽  
I. P. UNSWORTH ◽  
D. M. TURNER ◽  
J. E. ILKIW
Keyword(s):  
Hyperbaric Oxygen ◽  
Gas Gangrene

scholarly journals Sudden death after oxygen toxicity seizure during hyperbaric oxygen treatment: Case report

2020 ◽  
Vol 50 (2) ◽  
pp. 185-188
Author(s):  
Rutger C Lalieu ◽  
◽  
René D Bol Raap ◽  
Emile FL Dubois ◽  
Rob A van Hulst ◽  
...  
Keyword(s):  
Case Report ◽  
Sudden Death ◽  
Hyperbaric Oxygen ◽  
Oxygen Toxicity ◽  
Hyperbaric Oxygen Treatment ◽  
Oxygen Treatment
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