Fusarium oxysporum f.sp. cucumerinum. [Descriptions of Fungi and Bacteria].

1970 ◽  
Author(s):  
P. Holliday
Keyword(s):  
Fusarium Oxysporum ◽  
Cucumis Sativus ◽  
Geographical Distribution ◽  
Vascular System ◽  
Damping Off ◽  
Soil P ◽  
The North ◽  
Whole Plant ◽  
Single Crown

Abstract A description is provided for Fusarium oxysporum f.sp. cucumerinum. Information is included on the disease caused by the organism, its transmission, geographical distribution, and hosts. HOSTS: Cucumis sativus. DISEASE: A temperate and subtropical wilt of cucumber which caused up to 40% losses in Florida in 1949. The pathogen causes pre-emergence and post-emergence damping-off and wilt of older plants. In Florida mature plants with runners may first show wilt in a single crown branch, followed by collapse of the whole plant. Vascular necrosis may extend into the vine for 6-8 nodes (35: 265, 266). A cortical decay, absent from older plants, occurs in seedlings. In England, wilt symptoms have been described as beginning in the lower leaves and the vascular system of the lower nodes becomes prominent, standing out as white lines (45, 3013). GEOGRAPHICAL DISTRIBUTION: In U.S.A., in glasshouses in the north and in the field in California,. Florida, Mich., N. Mex., Texas and Wis. The disease has also been reported from: Africa (S. Africa); Asia (Iraq, Japan, Thailand); Australasia (Australia); Europe (England, France, Germany, Netherlands, Norway, U.S.S.R.); North Amenca (Canada). TRANSMISSION: Presumably through soil.

Fusarium oxysporum f.sp. melonis. [Descriptions of Fungi and Bacteria].

1970 ◽  
Author(s):  
P. Holliday
Keyword(s):  
North America ◽  
Fusarium Oxysporum ◽  
Cucumis Sativus ◽  
Geographical Distribution ◽  
Cucumis Melo ◽  
Cucurbita Pepo ◽  
Citrullus Lanatus ◽  
Damping Off ◽  
Leaf Chlorosis ◽  
Vascular Elements

Abstract A description is provided for Fusarium oxysporum f.sp. melonis. Information is included on the disease caused by the organism, its transmission, geographical distribution, and hosts. HOSTS: Cucumis melo. DISEASE: Causes a destructive wilt of muskmelon and cantaloupe. The casaba or winter melon is resistant (e.g. var. Honey Dew). Infection occurs on seedlings (pre-emergence and post-emergence damping-off) and older plants (leaf chlorosis, stunting and generai wilt). Streaks appear on the stems up to 0.6 m long; they become necrotic and bear the salmon-pink sporulating masses. In some cases stem cracks develop and a brownish exudate forms. The vascular elements become orange-red and the fruit is much reduced in size. The formae speciales of F. oxysporum from muskmelon and watermelon (Citrullus lanatus) will each attack the seedlings of both hosts, but older plants are susceptible only to their own f.sp. Generally the virulence of a given isolate is greater on its original host (12: 744; 27: 307; 31: 473; Leach, 1936). Cucumber (Cucumis sativus) and marrow (Cucurbita pepo) are resistant (39: 117). GEOGRAPHICAL DISTRIBUTION: Generai in North America (U.S.A., Canada) and Europe. Also reported from Asia (Formosa, Iraq, Japan, Philippines) and Australasia (Australia). TRANSMISSION: Through soil and seed (Leach, 1936).

Fusarium oxysporum f.sp. niveum. [Descriptions of Fungi and Bacteria].

1970 ◽  
Author(s):  
P. Holliday
Keyword(s):  
Fusarium Oxysporum ◽  
Geographical Distribution ◽  
Vascular System ◽  
Citrullus Lanatus ◽  
Seed Infection ◽  
Damping Off ◽  
Important Host ◽  
Wet Weather ◽  
North And South ◽  
South Europe

Abstract A description is provided for Fusarium oxysporum f.sp. niveum. Information is included on the disease caused by the organism, its transmission, geographical distribution, and hosts. HOSTS: Citrullus lanatus. DISEASE: Causes a widespread wilt of watermelon which is the only important host, also damping-off, cortical rot and stunting of seedlings and sudden or progressive wilt of older plants. Necrotic lesions occur on the roots and browning, gum and tyloses are found in the vascular system. In mature plants the wilt may be confined to a particular part, depending on which portion of the root system has been invaded from the soil. Chlorosis and stunting in maturing plants can occur and sometimes there is temporary recovery from wilt. Sporulation is found on dead stems in wet weather. GEOGRAPHICAL DISTRIBUTION: Throughout North America (U.S.A., Canada); South America (Argentina, Chile), South Europe including U.S.S.R. ; North and South Africa; Asia (India, Iraq, Japan, Pakistan, Philippines); Australasia (Australia, New Zealand). TRANSMISSION: Through soil; also seed infection has been reported (7: 760; 43, 2170).

Fusarium oxysporum f.sp. elaeidis. [Descriptions of Fungi and Bacteria].

1970 ◽  
Author(s):  
P. Holliday
Keyword(s):  
Fusarium Oxysporum ◽  
Geographical Distribution ◽  
Oil Palm ◽  
Vascular Tissue ◽  
Elaeis Guineensis ◽  
Vascular Wilt ◽  
Soil P ◽  
Acute Form ◽  
Leaf Necrosis ◽  
Internal Necrosis

Abstract A description is provided for Fusarium oxysporum f.sp. elaeidis. Information is included on the disease caused by the organism, its transmission, geographical distribution, and hosts. HOSTS: Elaeis guineensis. DISEASE: A vascular wilt of the oil palm was first reported from Africa (26: 103; 28: 124). In young palms the first symptom is an extensive chlorosis in some of the central leaves (4th-15th). This is followed by leaf necrosis (giving a flat-topped appearance) and death within a year. In mature palms leaf wilt and necrosis and breaking of the rachis may be rapid, causing death in a few months (acute form). But in the chronic form the progressive dying of the crown inwards may be very slow with new, though smaller, leaves being produced. The vascular tissue in roots and stems becomes orange, darkening progressively to black. It is very characteristic of the disease that the internal necrosis is restricted to the xylem region. Elaeis madagascariensis and E. melanococca have also been found to be affected (37: 52). GEOGRAPHICAL DISTRIBUTION: First found in the Congo, then later in Nigeria (27: 523), Cameroon (40: 148) and Colombia (46, 3389). TRANSMISSION: Presumably through soil.

Fusarium oxysporum f. sp. cubense. [Descriptions of Fungi and Bacteria].

1992 ◽  
Author(s):  
D. Brayford
Keyword(s):  
Fusarium Oxysporum ◽  
Sierra Leone ◽  
Geographical Distribution ◽  
Vascular Wilt ◽  
Distribution Map ◽  
Plant Debris ◽  
Panama Disease ◽  
Soil P ◽  
Planting Material ◽  
South Brazil

Abstract A description is provided for Fusarium oxysporum f. sp. cubense. Information is included on the disease caused by the organism, its transmission, geographical distribution, and hosts. HOSTS: Musa sp., Heliconia sp. DISEASE: Panama disease (vascular wilt) of banana. Also vascular wilt of abaca (Musa textilis). GEOGRAPHICAL DISTRIBUTION: Widespread in tropical and subtropical regions. Africa: Burundi, Cameroun, Canary Is., Ghana, Kenya, Madagascar, Malawi, Mauritius, Mozambique (50, 3049), Nigeria, Republic of South Africa, Rwanda, Sierra Leone, Tanzania (incl. Zanzibar), Uganda, Zaire. America: North: Florida (68, 905); South: Brazil, Costa Rica, Cuba (56, 4615), Dominica, Guadeloupe, Jamaica. Australasia: Australia, Brunei (51, 3059), Guam (60, 3258), India (68, 5699; 69, 657), Indonesia, Philippines, Sri Lanka, Taiwan, Thailand. IMI Distribution Map 31. TRANSMISSION: Through human transportation of infected planting material, plant debris or soil.

Fusarium oxysporum f.sp. cucumerinum. [Distribution map].

1984 ◽  
Author(s):  
Keyword(s):  
South Africa ◽  
New Mexico ◽  
North America ◽  
Fusarium Oxysporum ◽  
Central America ◽  
Cucumis Sativus ◽  
Geographical Distribution ◽  
Distribution Map ◽  
New Distribution

Abstract A new distribution map is provided for Fusarium oxysporum f. sp.cucumerinum Owen. Host: Cucumber (Cucumis sativus). Information is given on the geographical distribution in AFRICA, Kenya, Libya, South Africa, ASIA, Israel, Japan, Korea, Iraq, Turkey, Thailand, USSR, AUSTRALASIA, Australia, Queensland, Tasm., Southern Australia, EUROPE, Britain, France (SW), Germany, Greece, Netherlands, Norway, Romania, USSR (Armenia, Leningrad), NORTH AMERICA, Canada, USA (California, Florida, Ken., Michigan, New Mexico, Oklahoma, Texas, Wisconsin), CENTRAL AMERICA, Panama.

Phomopsis sclerotioides. [Descriptions of Fungi and Bacteria].

1975 ◽  
Author(s):  
E. Punithalingam
Keyword(s):  
British Columbia ◽  
Cucumis Sativus ◽  
Geographical Distribution ◽  
Black Rot ◽  
Cortical Tissue ◽  
Soil P ◽  
Phomopsis Sclerotioides ◽  
Lateral Shoots ◽  
Necrotic Spots

Abstract A description is provided for Phomopsis sclerotioides. Information is included on the disease caused by the organism, its transmission, geographical distribution, and hosts. HOSTS: On members of Cucurbitaceae. DISEASE: Black rot of cucumber (Cucumis sativus). On finer roots, small isolated sunken necrotic spots. Later infections on older roots greyish black or brown areas (not necessarily sunken), bounded by darker areas (black wavy lines); coalescence occurs. Roots may be girdled and killed and the cortical tissue sloughs off. Stem infected at base from several rotted roots; stems show elongate brown (sometimes black) lesions with amber, gummy exudations. Plants are stunted, have fewer lateral shoots and small, downward cupped, leaves; partially developed fruit does not mature. Irreversible wilt (non-vascular) often reached as fruiting begins (46, 3612; 48, 3235). GEOGRAPHICAL DISTRIBUTION: Britain, Canada (British Columbia), Denmark, France, Germany, Malaysia (Sabah), Netherlands and Norway (CMI Map 509, ed. 1, 1975). TRANSMISSION: Through soil.

Fusarium oxysporum f. sp. cepae. [Descriptions of Fungi and Bacteria].

1996 ◽  
Author(s):  
D. Brayford
Keyword(s):  
South Africa ◽  
South America ◽  
Fusarium Oxysporum ◽  
Geographical Distribution ◽  
Damping Off ◽  
Planting Material ◽  
Basal Rot ◽  
Japanese Bunching Onion ◽  
Bunching Onion ◽  
Local Dispersal

Abstract A description is provided for Fusarium oxysporum f. sp. cepae. Information is included on the disease caused by the organism, its transmission, geographical distribution, and hosts. HOSTS: Allium cepa L. (onion, shallots), A. sativum L. (garlic) (see notes below concerning F. oxysporum f.sp. garlic), A. fistulosum L. (Japanese bunching onion), A. chinense G. Don (rakkyo); Asparagus (68, 2953); Oxalis spp. (56, 1835). DISEASE: Basal rot of bulbs. Damping off of seedlings. GEOGRAPHICAL DISTRIBUTION: Africa: Egypt, Ethiopia, South Africa, Zambia. Australasia: India, Israel, Japan, Philippines, Tasmania. Europe: Greece, Hungary, Italy. North America: USA. South America: Brazil. TRANSMISSION: Via seed, infected planting material or movement of soil. Local dispersal is by slimy micro- and macroconidia moved by water flow and splash droplets.

Fusarium oxysporum f. sp. batatas. [Descriptions of Fungi and Bacteria].

1992 ◽  
Author(s):  
D. Brayford
Keyword(s):  
Fusarium Oxysporum ◽  
Geographical Distribution ◽  
Plant Material ◽  
Ipomoea Batatas ◽  
Infected Plant ◽  
Vascular Wilt ◽  
Tropical Regions ◽  
Soil P ◽  
Wide Range ◽  
Infected Plant Material

Abstract A description is provided for Fusarium oxysporum f. sp. batatas. Information is included on the disease caused by the organism, its transmission, geographical distribution, and hosts. HOSTS: Ipomoea batatas (sweet potato), Nicotiana (tobacco). The fungus may infect a wide range of other plants in Convolvulaceae (63, 1065) and other families, sometimes without causing wilt symptoms. DISEASE: Vascular wilt, sometimes called stem rot. GEOGRAPHICAL DISTRIBUTION: Brazil (62, 525), China, Hawaii, India, Japan, Malawi, New Zealand. The disease occurs in temperate rather than tropical regions. TRANSMISSION: The fungus may survive in soil for many years as chlamydospores. Transmission may occur by means of infected plant material used for propagation, or through contaminated soil.

Fusarium oxysporum f.sp. batatas. [Descriptions of Fungi and Bacteria].

1970 ◽  
Author(s):  
P. Holliday
Keyword(s):  
Fusarium Oxysporum ◽  
Sweet Potato ◽  
Geographical Distribution ◽  
Ipomoea Batatas ◽  
Potato Production ◽  
Soil P ◽  
The Pacific ◽  
Race 2 ◽  
The Tropics ◽  
Water Melon

Abstract A description is provided for Fusarium oxysporum f.sp. batatas. Information is included on the disease caused by the organism, its transmission, geographical distribution, and hosts. HOSTS: Ipomoea batatas. DISEASE: An important wilt disease of sweet potato in U.S.A. but apparently of little importance in the tropics. Interveinal yellowing of the leaves is followed by distortion and stunting and the old leaves fall. There is extensive vascular necrosis which may appear purplish below soil level; the cortex may rupture. Infected tubers may rot in storage. Fusaria that cause surface rots are probably different (41: 329). The fungus infects the roots of many plants without causing any external symptoms, viz. cabbage, cotton, cowpea, maize, okra potato, sage, snap bean, soyabean, tobacco, tomato and water melon (28: 189; 37: 593). But race 2 causes wilt in Burley and flue cured tobacco (38: 421). GEOGRAPHICAL DISTRIBUTION: General in U.S.A., especially in the northern range of sweet potato production, including the Pacific coast and western states (40: 511). Also reported from Africa (Malawi); Asia (China, Formosa, India, Japan) Australasia & Oceania (Hawaii, New Zealand). TRANSMISSION: Through plant material and by any means through soil.

scholarly journals Development of specific primers for Fusarium oxysporum causing damping off of Lilium formolongi

2020 ◽  
Vol 18 (1) ◽  
Author(s):  
Takeshi Toda ◽  
Shun Hanesaka ◽  
Kuniaki Shishido ◽  
Shin-ichi Fuji ◽  
Hiromitsu Furuya
Keyword(s):  
Fusarium Oxysporum ◽  
Pathogenic Fungus ◽  
Primer Design ◽  
Damping Off ◽  
Specific Primers ◽  
Forma Specialis

AbstractPrimers specific for the hypothetical forma specialis of Fusarium oxysporum were designed to amplify DNA from this pathogenic fungus that infects plants including lilies. The F. oxysporum sequence between the transposal elements han and hop was used for primer design. Three primer pairs designed from this region were confirmed as specific for 24 isolates of F. oxysporum pathogenic to lilies, except for one pathogenic isolates as extraordinary. No amplification was observed from F. oxysporum non-pathogenic to lily, from 12 forma specialis, and 14 fungi and oomycetes concerned with Liliaceae plants. We propose that specific primers designed from this region will be useful to detect isolates of F. oxysporum that are pathogenic to lilies.

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