INDUCED SEED TREATMENT WITH HYDROGEN PEROXIDE (H2O2) PROMOTES PHYSIOLOGICAL, BIOLOGICAL CHANGES AND SALT-TOLERANCE IN WHEAT (TRITICUM AESTIVUM L.)

The present study was undertaken to study the effect of exogenously applied six hydrogen peroxide H2O2 concentrations (0, 20, 40, 60, 80 and 100 µM) as seed primer on two wheat varieties (Khirman and Inqalab) under salt and non-salt water levels (0 and 100 mM NaCl). The oxidizing effects were measured and estimated using the wheat index of different salt and non-salt water levels, as well as H2O2 soaking seed at varying fixations and concentrations. The H2O2 as a seed primer impacted on growth, yield and physiological and biochemical aspects such as moisture content, sodium potassium substance and sodium potassium content under H2O2 and NaCl levels. The results revealed that the exogenous application of hydrogen peroxide was effective in increasing tolerance of wheat under salt stress. The Khirman and Inqalab varieties could be established and cultivated under saline conditions. 60?M treatment of H2O2 is seen with the strongest impacts. Progress has contributed to enhance physiological and biochemical features of stress outflow, which promote growth.

Glutathione content of the small intestine: regulation and function

In ad lib.-fed rats the epithelium of the small intestine, like the liver, contains large quantities of glutathione, 17.0 and 32.4 nmol/mg protein respectively. Following 24 h food restriction the glutathione content in both tissues fell 53 and 69% respectively. Unlike the liver, however, the glutathione content of the intestinal mucosa is not regulated to a diurnal rhythm, suggesting that the liver may provide glutathione or glutathione precursors to maintain intestinal glutathione levels. Intestinal epithelial cell preparations obtained from 24 h food-deprived rats had depleted glutathione stores (50%) and as a consequence were more susceptible to the oxidizing effects of cumene hydroperoxide. These results suggest that if glutathione plays a major role in the defence of the intestinal mucosa from ingested toxins then depletion of this defence during periods of food restriction could significantly increase the susceptibility of the individual to toxins present in the diet.