Identification of Avian Paramyxovirus Serotype-1 in Wild Birds in the USA

2016 ◽  
Vol 52 (3) ◽  
pp. 657 ◽  
Author(s):  
Kerri Pedersen ◽  
David R. Marks ◽  
Claudio L. Afonso ◽  
Scott R. Stopak ◽  
Dawn Williams-Coplin ◽  
...  
2016 ◽  
Vol 11 (2) ◽  
pp. 50-58
Author(s):  
A. V. Glushchenko ◽  
K. S. Yurchenko ◽  
A. K. Yurlov ◽  
Yu. G. Yushkov ◽  
M. Yu. Shchelkanov ◽  
...  

2017 ◽  
Vol 14 (1) ◽  
Author(s):  
Andrew M. Ramey ◽  
Iryna V. Goraichuk ◽  
Joseph T. Hicks ◽  
Kiril M. Dimitrov ◽  
Rebecca L. Poulson ◽  
...  

2013 ◽  
Vol 158 (12) ◽  
pp. 2495-2503 ◽  
Author(s):  
Andrew M. Ramey ◽  
Andrew B. Reeves ◽  
Haruko Ogawa ◽  
Hon S. Ip ◽  
Kunitoshi Imai ◽  
...  

2016 ◽  
Vol 7 ◽  
Author(s):  
Dilan A. Satharasinghe ◽  
Kavitha Murulitharan ◽  
Sheau W. Tan ◽  
Swee K. Yeap ◽  
Muhammad Munir ◽  
...  

Author(s):  
Fedri Rell ◽  
Anak Agung Ayu Mirah Adi ◽  
I Gusti Ngurah Kade Mahardika

Newcastle disease (ND) merupakan penyakit kontagius yang disebabkan virus Avian paramyxovirus serotype 1 yang menginfeksi bangsa unggas. Penelitian ini bertujuan untuk menganalisis pohon filogeni berdasarkan sekuan gen daerah pemotonan protein fusion dari virus ND pada peternakan ayam di provinsi Bali dari tahun 2013 sampai 2014. Sebanyak empat isolat virus dari kasus ayam sakit/mati yang dicurigai terinfeksi oleh virus Newcastle disease. Sekuen potongan gen F disejajarkan dan dianalisis dengan program MEGA5. Analisis sekuen asam amino daerah pemotongan protein F keempat isolat memiliki sekuen 112R-R-Q-K-R-F117 dan dilanjutkan dengan analisis pohom filogeni yang menunjukan bahwa keempat isolat merupakan virus Newcastle disease yang virulen. Penelitian ini menunjukan bahwa keempat isolat lapang Bali tahun 2013 sampai 2014 masuk ke dalam kelompok virus Newcastle disease genotipe VII.


2012 ◽  
Vol 2012 ◽  
pp. 1-17 ◽  
Author(s):  
Nichole L. Hines ◽  
Cathy L. Miller

Avian paramyxovirus serotype-1 (APMV-1) is capable of infecting a wide range of avian species leading to a broad range of clinical symptoms. Ease of transmission has allowed the virus to spread worldwide with varying degrees of virulence depending on the virus strain and host species. Classification systems have been designed to group isolates based on their genetic composition. The genetic composition of the fusion gene cleavage site plays an important role in virulence. Presence of multiple basic amino acids at the cleavage site allows enzymatic cleavage of the fusion protein enabling virulent viruses to spread systemically. Diagnostic tests, including virus isolation, real-time reverse-transcription PCR, and sequencing, are used to characterize the virus and identify virulent strains. Genetic diversity within APMV-1 demonstrates the need for continual monitoring for changes that may arise requiring modifications to the molecular assays to maintain their usefulness for diagnostic testing.


1992 ◽  
Vol 39 (1-10) ◽  
pp. 153-158 ◽  
Author(s):  
U. Wernery ◽  
J. D. Remple ◽  
U. Neumann ◽  
D. J. Alexander ◽  
Ruth J. Manvell ◽  
...  

2013 ◽  
Vol 17 ◽  
pp. 260-268 ◽  
Author(s):  
Yee Ling Chong ◽  
Tommy Tsan-Yuk Lam ◽  
Oekyung Kim ◽  
Huaguang Lu ◽  
Patty Dunn ◽  
...  

2012 ◽  
Vol 97 (12) ◽  
pp. 1070-1072 ◽  
Author(s):  
Matthew F Thomas ◽  
Carmen L Sheppard ◽  
Malcolm Guiver ◽  
Mary P E Slack ◽  
Robert C George ◽  
...  

IntroductionInvasive pneumococcal disease due to serotype 19A has become a major concern, particularly in the USA and Asia. We describe the characteristics of pneumococcal serotype 19A related empyema and changes in its incidence in the UK.MethodsData from paediatric empyema patients between September 2006 and March 2011 were collected from 17 respiratory centres in the UK. Pneumococcal serotypes were identified as part of the Health Protection Agency enhanced paediatric empyema surveillance programme.ResultsFour serotypes accounted for over 80% of 136 cases (Serotype 1 : 43%, 3 : 21%, 7 : 11% and 19A:10%). The incidence of empyema due to serotype 19A quadrupled from 0.48 (0.16–1.13) cases per million children in 2006/2007 to 2.02 (1.25–3.09) in 2010/2011. Severity of disease was significantly increased in children with 19A infection when compared to other serotypes.ConclusionsThe incidence of empyema due to pneumococcal serotype 19A infection has increased significantly and is associated with substantial morbidity.


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