scholarly journals Role of Collateral Vein Occlusion in Autologous Dysfunctional Hemodialysis Fistulas

Author(s):  
Iftikhar Ahmad
2021 ◽  
pp. 112067212110006
Author(s):  
Manuel Marques ◽  
Francisco Alves ◽  
Miguel Leitão ◽  
Catarina Rodrigues ◽  
Joana Tavares Ferreira

The role of polymorphisms of methylenetetrahydrofolate reductase (MTHFR) gene in retinal vein occlusion (RVO) is a theme of discussion since the first reports of RVO in patients with MTHFR C677T mutation and without classic acquired risk factors for retinal vascular disease. The association between MTHFR polymorphisms and RVO has been studied over the last 20 years producing conflicting results. This review aims to summarize the literature concerning the role MTHFR polymorphisms as risk factors for RVO.


Author(s):  
Alessia Viscardi ◽  
Antonio Travaglino ◽  
Luca del Guercio ◽  
Maria D'Armiento ◽  
Michele Santangelo ◽  
...  

2018 ◽  
Vol 59 (11) ◽  
pp. 4701 ◽  
Author(s):  
Anri Nishinaka ◽  
Yuki Inoue ◽  
Shinichiro Fuma ◽  
Yoshifumi Hida ◽  
Shinsuke Nakamura ◽  
...  

2020 ◽  
pp. 112067212096873
Author(s):  
Lorenzo Iuliano ◽  
Eleonora Corbelli ◽  
Francesco Bandello ◽  
Marco Codenotti

Purpose: To hypothesize the protective role of vitrectomy on the natural course of diabetic retinopathy. Methods: Case report. Results: A 66-year-old lady with a history of type 1 diabetes mellitus since the age of 10 was referred for evaluation. She received laser treatment in her left eye for proliferative diabetic retinopathy 6 years earlier, while her right eye underwent vitrectomy for retinal detachment 17 years before. Diabetic retinopathy was found to be substantially asymmetrical, as the vitrectomized right eye had only mild macular edema without significant vascular abnormalities, whereas the left eye had a complete confluent panretinal photocoagulation with advanced atrophic macular changes. Other reasons for this asymmetry, such as axial length disparity, carotid circulation impairment, or history of branch retinal vein occlusion, were not identified. Conclusions: Vitreous removal, performed for a reason other than diabetic retinopathy, may inhibit the retinal complications often associated with longstanding diabetes. Further clinical studies should be considered to ascertain such evidence.


2014 ◽  
Vol 27 (4) ◽  
pp. E48-E50
Author(s):  
Paweł Stróżecki ◽  
Mariusz Flisiński ◽  
Zbigniew Serafin ◽  
Joanna Wiechecka-Korenkiewicz ◽  
Jacek Manitius

QJM ◽  
2015 ◽  
Vol 109 (2) ◽  
pp. 97-102 ◽  
Author(s):  
J.J. Napal ◽  
S. Neila ◽  
R. Pérez-Montes ◽  
I. Sierra ◽  
S. Ruiz ◽  
...  

2014 ◽  
Vol 2014 ◽  
pp. 1-6 ◽  
Author(s):  
Francisco J. Ascaso ◽  
Valentín Huerva ◽  
Andrzej Grzybowski

Macular edema (ME) is a nonspecific sign of numerous retinal vascular diseases. This paper is an updated overview about the role of inflammatory processes in the genesis of both diabetic macular edema (DME) and ME secondary to retinal vein occlusion (RVO). We focus on the inflammatory mediators implicated, the effect of the different intravitreal therapies, the recruitment of leukocytes mediated by adhesion molecules, and the role of retinal Müller glial (RMG) cells.


2018 ◽  
Vol 38 (5) ◽  
Author(s):  
Xinyue Yang ◽  
Yan Cheng ◽  
Guanfang Su

Angiopoietin-like protein 4 (ANGPTL4) is a multifunctional cytokine regulating vascular permeability, angiogenesis, and inflammation. Dysregulations in these responses contribute to the pathogenesis of ischemic retinopathies such as diabetic retinopathy (DR), age-related macular degeneration (AMD), retinal vein occlusion, and sickle cell retinopathy (SCR). However, the role of ANGPTL4 in these diseases remains controversial. Here, we summarize the functional mechanisms of ANGPTL4 in several diseases. We highlight original studies that provide detailed data about the mechanisms of action for ANGPTL4, its applications as a diagnostic or prognostic biomarker, and its use as a potential therapeutic target. Taken together, the discussions in this review will help us gain a better understanding of the molecular mechanisms by which ANGPTL4 functions in eye disease and will provide directions for future research.


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