scholarly journals Advanced Glycation End Products Inhibit Both Infection and TransmissionIn Transof HIV-1 from Monocyte-Derived Dendritic Cells to Autologous T Cells

2011 ◽  
Vol 186 (10) ◽  
pp. 5687-5695 ◽  
Author(s):  
Nadine Nasreddine ◽  
Chloé Borde ◽  
Joël Gozlan ◽  
Laurent Bélec ◽  
Vincent Maréchal ◽  
...  
Keyword(s):  
T Cells ◽  
Dendritic Cells ◽  
Advanced Glycation End Products ◽  
Advanced Glycation ◽  
Glycation End Products ◽  
End Products ◽  
Hiv 1

Advanced Glycation End Products Modulate the Maturation and Function of Peripheral Blood Dendritic Cells

Diabetes ◽  
2004 ◽  
Vol 53 (6) ◽  
pp. 1452-1458 ◽  
Author(s):  
C. L. Price ◽  
P. S. Sharp ◽  
M. E. North ◽  
S. J. Rainbow ◽  
S. C. Knight
Keyword(s):  
Dendritic Cells ◽  
Advanced Glycation End Products ◽  
Peripheral Blood ◽  
Advanced Glycation ◽  
Glycation End Products ◽  
End Products ◽  
And Function

scholarly journals Release of High Mobility Group Box 1 by Dendritic Cells Controls T Cell Activation via the Receptor for Advanced Glycation End Products

2005 ◽  
Vol 174 (12) ◽  
pp. 7506-7515 ◽  
Author(s):  
Ingrid E. Dumitriu ◽  
Paramita Baruah ◽  
Barbara Valentinis ◽  
Reinhard E. Voll ◽  
Martin Herrmann ◽  
...  
Keyword(s):  
Dendritic Cells ◽  
T Cell ◽  
T Cell Activation ◽  
Advanced Glycation End Products ◽  
Cell Activation ◽  
High Mobility ◽  
High Mobility Group ◽  
Advanced Glycation ◽  
Glycation End Products ◽  
End Products

scholarly journals Placental Morphology and Cellular Characteristics in Stillbirths in Women With Diabetes and Unexplained Stillbirths

2020 ◽  
Vol 145 (1) ◽  
pp. 82-89
Author(s):  
Alan Kerby ◽  
Daniel Shingleton ◽  
Gauri Batra ◽  
Megan C. Sharps ◽  
Bernadette C. Baker ◽  
...  
Keyword(s):  
T Cells ◽  
Advanced Glycation End Products ◽  
Advanced Glycation ◽  
Phenotypic Differences ◽  
Live Births ◽  
Glycation End Products ◽  
End Products ◽  
Hofbauer Cells ◽  
Placental Macrophages ◽  
And Function

Context.— Women with diabetes have increased stillbirth risk. Although the underlying pathophysiological processes are poorly understood, stillbirth is frequently related to abnormal placental structure and function. Objective.— To investigate placental morphology and cellular characteristics in the placentas of women with diabetes who had stillbirths and stillbirths of unexplained cause. Design.— Placentas from women with uncomplicated live births, live births in women with diabetes, unexplained stillbirths, and stillbirths related to diabetes (n = 10/group) underwent clinical histopathologic assessment and were also investigated using immunohistochemical staining to quantify syncytial nuclear aggregates, proliferation, trophoblast area, vascularization, T cells, placental macrophages (Hofbauer cells), and the receptor for advanced glycation end products. Results.— Ki67+ cells were decreased in unexplained stillbirths compared with live births in women with diabetes. Both stillbirth groups had increased cytokeratin 7+/nuclear area compared with controls. Blood vessels/villi were decreased in unexplained stillbirth compared with live births from women with diabetes. Compared with uncomplicated controls, CD163+ macrophages were increased in live births in women with diabetes and unexplained stillbirths, and further increased in stillbirths related to diabetes. There was no change in CD3+ T cells or syncytial nuclear aggregates. Receptor for advanced glycation end products–positive cells were decreased in both stillbirth groups compared with diabetes-related live births. Co-localization of receptor for advanced glycation end products in macrophages was increased in both stillbirth groups compared with live birth groups. Conclusions.— Stillbirths related to diabetes exhibit placental phenotypic differences compared with live births. Further investigation of these parameters may provide understanding of the pathologic mechanisms of stillbirth and aid the development of stillbirth prevention strategies.

scholarly journals Receptor for Advanced Glycation End Products Expression on T Cells Contributes to Antigen-Specific Cellular Expansion In Vivo

2007 ◽  
Vol 179 (12) ◽  
pp. 8051-8058 ◽  
Author(s):  
Bernhard Moser ◽  
Dharmesh D. Desai ◽  
Matthew P. Downie ◽  
Yali Chen ◽  
Shi Fang Yan ◽  
...  
Keyword(s):  
T Cells ◽  
Advanced Glycation End Products ◽  
Advanced Glycation ◽  
Glycation End Products ◽  
End Products
Sign in / Sign up

Export Citation Format

Share Document