Host Susceptibility to Paralysis by the Tick Dermacentor andersoni Stiles (Acarina: Ixodidae)

1958 ◽  
Vol 90 (7) ◽  
pp. 421-424 ◽  
Author(s):  
J. D. Gregson
Keyword(s):  
British Columbia ◽  
Laboratory Animal ◽  
Domestic Animals ◽  
Host Susceptibility ◽  
Dermacentor Andersoni ◽  
Tick Paralysis

One of the objectives in the study of tick paralysis at Kamloops was to discover a species of laboratory animal that is consistently susceptible to this disease. Such an animal was needed to explore inconsistencies probably caused by differences in tick virulence or host susceptibility. Observations on infestations of adults of Dermacentor andersoni in tick paralysis areas in British Columbia, and experiments with similar ticks on large and small wild and domestic animals, have revealed the following.

Experiments on Oral Secretion of the Rocky Mountain Wood Tick, Dermacentor andersoni Stiles (Acarina: Ixodidae)

1957 ◽  
Vol 89 (1) ◽  
pp. 1-5 ◽  
Author(s):  
J. D. Gregson
Keyword(s):  
British Columbia ◽  
Pacific Northwest ◽  
Rocky Mountain ◽  
Dermacentor Variabilis ◽  
Oral Secretion ◽  
Amblyomma Maculatum ◽  
Dermacentor Andersoni ◽  
The Pacific ◽  
Tick Paralysis ◽  
Rocky Mountain Wood Tick

Tick paralysis continues to be one of the most baffling and fascinating tickborne diseases in Canada. It was first reported in this country by Todd in 1912. Since then about 250 human cases, including 28 deaths, have been recorded from British Columbia. Outbreaks in cattle have affected up to 400 animals at a time, with losses in a herd as high as 65 head. Although the disease is most common in the Pacific northwest, where it is caused by the Rocky Mountain wood tick, Dermacentor andersoni Stiles, it has lately been reported as far south as Florida and has been produced by Dermacentor variabilis Say, Amblyomma maculatum Koch, and A. americanum (L.) (Gregson, 1953). The symptoms include a gradual ascending symmetrical flaccid paralysis. Apparently only man, sheep, cattle, dogs, and buffalo (one known instance) are susceptible, but even these may not necessarily be paralysed.

Ticks in Relation to Certain Diseases of Man and Animals in Canada

1964 ◽  
Vol 96 (1-2) ◽  
pp. 112-112
Author(s):  
J. D. Gregson
Keyword(s):  
British Columbia ◽  
Ixodes Pacificus ◽  
Dermacentor Andersoni ◽  
Dermacentor Albipictus ◽  
Tick Paralysis

In Canada eight species of ticks are implicated with ten and possibly seventeen diseases of man and animals. The most important of these, tick paralysis, is caused by Dermacentor andersoni Stiles, and of three hundred human cases in British Columbia since 1910, thirty have been fatal. In livestock, outbreaks have involved up to four hundred cattle in a single herd, with losses of up to fifty head. Other diseases resulting from tick toxins include a paralysis of cattle by Oyobius megnini Dugès, which has caused twelve deaths since 1955; occasional deaths in deer and moose from heavy infestations of Dermacentor albipictus (Packard); and slow-healing sores in man resulting from the bites of Ixodes pacificus Cooley and Kohls.

Conservation and health of Lumholtz’s tree-kangaroo (Dendrolagus lumholtzi)

2019 ◽  
Vol 41 (1) ◽  
pp. 57 ◽  
Author(s):  
Amy L. Shima ◽  
Lee Berger ◽  
Lee F. Skerratt
Keyword(s):  
Domestic Animals ◽  
Home Range Size ◽  
Population Viability ◽  
Population Status ◽  
Far North ◽  
Wild Tree ◽  
Wild Dogs ◽  
Tick Paralysis ◽  
Dendrolagus Lumholtzi ◽  
Conservation And Management

Lumholtz’s tree-kangaroo (Dendrolagus lumholtzi) is an iconic species in far north Queensland yet little is known about its health or population status. Studies on this species have been conducted in a limited number of locations and focused primarily on ecology, habitat use and home-range size. The species is relatively common in the Atherton Tablelands but habitat loss, predation by domestic, feral and wild dogs, vehicle strike, low fecundity, and disease have been identified as threats to the population. We review knowledge of population ecology and threats for this species, and include a novel collation of disease reports on all tree-kangaroos with particular reference to Lumholtz’s tree-kangaroo. Health of Lumholtz’s tree-kangaroo appears to be impacted by the increase in humans and domestic animals in their range. There have been reports of melioidosis, toxoplasmosis, tick paralysis and blindness in wild tree-kangaroos. We identify where increased information on health and population viability will improve conservation and management of the species.

scholarly journals Host Susceptibility toBrucella abortusInfection Is More Pronounced in IFN-γknockout than IL-12/β2-Microglobulin Double-Deficient Mice

2012 ◽  
Vol 2012 ◽  
pp. 1-7 ◽  
Author(s):  
Ana Paula M. S. Brandão ◽  
Fernanda S. Oliveira ◽  
Natalia B. Carvalho ◽  
Leda Q. Vieira ◽  
Vasco Azevedo ◽  
...  
Keyword(s):  
T Cells ◽  
Immune Responses ◽  
Cd8 T Cells ◽  
Bacterial Load ◽  
Bacterial Pathogen ◽  
Domestic Animals ◽  
Host Susceptibility ◽  
Mouse Splenocytes ◽  
Host Immune Responses ◽  
Deficient Mice

Brucella abortusis a facultative intracellular bacterial pathogen that causes abortion in domestic animals and undulant fever in humans. IFN-γ, IL-12, and CD8+ T lymphocytes are important components of host immune responses againstB. abortus. Herein, IFN-γand IL-12/β2-microglobulin (β2-m) knockout mice were used to determine whether CD8+ T cells and IL-12-dependent IFN-γdeficiency would be more critical to controlB. abortusinfection compared to the lack of endogenous IFN-γ. At 1 week after infection, IFN-γKO and IL-12/β2-m KO mice showed increased numbers of bacterial load in spleens; however, at 3 weeks postinfection (p.i.), only IFN-γKO succumbed toBrucella. All IFN-γKO had died at 16 days p.i. whereas death within the IL-12/β2-m KO group was delayed and occurred at 32 days until 47 days postinfection. Susceptibility of IL-12/β2-m KO animals toBrucellawas associated to undetectable levels of IFN-γin mouse splenocytes and inability of these cells to lyseBrucella-infected macrophages. However, the lack of endogenous IFN-γwas found to be more important to control brucellosis than CD8+ T cells and IL-12-dependent IFN-γdeficiencies.

Variation in the Susceptibility of Lodgepole Pine Provenances to Sirococcus Shoot Blight

1973 ◽  
Vol 3 (4) ◽  
pp. 585-589 ◽  
Author(s):  
K. Illingworth
Keyword(s):  
British Columbia ◽  
Genetic Variation ◽  
Entire Range ◽  
Lodgepole Pine ◽  
Host Susceptibility ◽  
Marked Variation ◽  
Shoot Blight

Sirococcusstrobilinus Preuss. was identified as the cause of a shoot blight of lodgepole pine (Pinuscontorta Dougl. ex Loud.) seedlings grown in a nursery in central British Columbia. Marked variation in susceptibility to this fungus was noted among the 153 provenances representing the entire range of lodgepole pine. Genetic variation in host susceptibility is inferred.

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