scholarly journals Variable susceptibility and response to estrogenic chemicals in Menidia menidia

2009 ◽  
Vol 380 ◽  
pp. 245-254 ◽  
Author(s):  
TA Duffy ◽  
AE McElroy ◽  
DO Conover
Keyword(s):  
Menidia Menidia ◽  
Estrogenic Chemicals ◽  
Variable Susceptibility

scholarly journals Developmental toxicity of estrogenic chemicals on rodents and other species

2002 ◽  
Vol 42 (2) ◽  
pp. 94-105 ◽  
Author(s):  
Taisen Iguchi ◽  
Hajime Watanabe ◽  
Yoshinao Katsu ◽  
Takeshi Mizutani ◽  
Shinichi Miyagawa ◽  
...  
Keyword(s):  
Developmental Toxicity ◽  
Estrogenic Chemicals

Pancreatic Adenocarcinoma Cell Lines Show Variable Susceptibility to TRAIL-Mediated Cell Death

Pancreas ◽  
2001 ◽  
Vol 23 (1) ◽  
pp. 72-79 ◽  
Author(s):  
Saleh M. Ibrahim ◽  
Jörg Ringel ◽  
Christian Schmidt ◽  
Bruno Ringel ◽  
Petra Müller ◽  
...  
Keyword(s):  
Cell Death ◽  
Pancreatic Adenocarcinoma ◽  
Cell Lines ◽  
Adenocarcinoma Cell ◽  
Variable Susceptibility

scholarly journals You Better Repeat It: Complex CO2 × Temperature Effects in Atlantic Silverside Offspring Revealed by Serial Experimentation

Diversity ◽  
2018 ◽  
Vol 10 (3) ◽  
pp. 69 ◽  
Author(s):  
Christopher Murray ◽  
Hannes Baumann
Keyword(s):  
Early Life ◽  
Larval Growth ◽  
Menidia Menidia ◽  
Embryo Survival ◽  
Single Experiment ◽  
Growth And Survival ◽  
Atlantic Silverside ◽  
Co2 Effects ◽  
Experimental Approaches ◽  
Temperature Interactions

Concurrent ocean warming and acidification demand experimental approaches that assess biological sensitivities to combined effects of these potential stressors. Here, we summarize five CO2 × temperature experiments on wild Atlantic silverside, Menidia menidia, offspring that were reared under factorial combinations of CO2 (nominal: 400, 2200, 4000, and 6000 µatm) and temperature (17, 20, 24, and 28 °C) to quantify the temperature-dependence of CO2 effects in early life growth and survival. Across experiments and temperature treatments, we found few significant CO2 effects on response traits. Survival effects were limited to a single experiment, where elevated CO2 exposure reduced embryo survival at 17 and 24 °C. Hatch length displayed CO2 × temperature interactions due largely to reduced hatch size at 24 °C in one experiment but increased length at 28 °C in another. We found no overall influence of CO2 on larval growth or survival to 9, 10, 15 and 13–22 days post-hatch, at 28, 24, 20, and 17 °C, respectively. Importantly, exposure to cooler (17 °C) and warmer (28 °C) than optimal rearing temperatures (24 °C) in this species did not appear to increase CO2 sensitivity. Repeated experimentation documented substantial inter- and intra-experiment variability, highlighting the need for experimental replication to more robustly constrain inherently variable responses. Taken together, these results demonstrate that the early life stages of this ecologically important forage fish appear largely tolerate to even extreme levels of CO2 across a broad thermal regime.

Darwinian fishery science: lessons from the Atlantic silverside (Menidia menidia)

2005 ◽  
Vol 62 (4) ◽  
pp. 730-737 ◽  
Author(s):  
David O Conover ◽  
Stephen A Arnott ◽  
Matthew R Walsh ◽  
Stephan B Munch
Keyword(s):  
Life History ◽  
Theory And Practice ◽  
Winter Mortality ◽  
Menidia Menidia ◽  
Adaptive Genetic Variation ◽  
Atlantic Silverside ◽  
Fishery Science ◽  
Evolutionary Changes ◽  
In The Wild ◽  
Contrary Evidence

The potential of fishing mortality to cause rapid evolutionary changes in life history has received relatively little attention. By focusing only on ecological responses, standard fisheries theory and practice implicitly assume either that genetic influences on life history in the wild are negligible or that natural selection and adaptation is a slow process that can be effectively ignored. Lack of contrary evidence has allowed these assumptions to persist. Drawing upon >25 years of research on the Atlantic silverside (Menidia menidia), we show that adaptive genetic variation in many traits is finely tuned to natural variation in climate. Much of this variation is caused by a gradient in size-selective winter mortality and involves two- to threefold changes in physiological traits that influence population productivity. Many other species are now known to display similar patterns. Harvest experiments show that these traits can evolve rapidly in response to size-selective fishing. Hence, the pool of genotypes that code for life history traits is a highly dynamic property of populations. We argue that the lessons from Menidia are applicable to many exploited species where similar observations would be difficult to obtain and advocate greater use of species models to address fundamental questions in fishery science.

Prostate Development: Mechanisms for Opposite Effects of Low and High Doses of Estrogenic Chemicals

2004 ◽  
pp. 379-410 ◽  
Author(s):  
Catherine A. Richter ◽  
Barry G. Timms ◽  
Frederick S. vom Saal
Keyword(s):  
Prostate Development ◽  
Estrogenic Chemicals ◽  
High Doses
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