scholarly journals GEOMETRY OF THE BOOSTER INJECTION REGION

1993 ◽  
Author(s):  
E. Bleser
1971 ◽  
Vol 15 (3) ◽  
pp. 273-275 ◽  
Author(s):  
Naohide Takayama ◽  
Nobuo Sakurai ◽  
Muneharu Nakayama

2012 ◽  
Vol 36 (5) ◽  
pp. S46-S47
Author(s):  
Leszek Nosek ◽  
Hans-Veit Coester ◽  
Henrik F. Thomsen ◽  
Carsten Roepstorff ◽  
Hanne L. Haahr ◽  
...  

2005 ◽  
Vol 23 (3) ◽  
pp. 877-884 ◽  
Author(s):  
T. Sarris ◽  
X. Li

Abstract. One manifestation of energetic particle acceleration during magnetospheric substorms is the sudden appearance of particle injections into the inner magnetosphere, often observed near geosynchronous orbit. Injections that show simultaneous flux increases in all energy ranges of a detector are called dispersionless injections, and are most often observed in a narrow region around local midnight. In these events it is assumed that the satellite is located close to or inside the region where acceleration and/or transport processes are taking place, called the injection region. We present a study of the location, extent and temporal evolution of the injection region, based on simulation results of a model of the expansion of the electric and magnetic fields associated with a substorm. The model simulates the fields during a substorm onset with an electric field and consistent magnetic field pulse that propagates towards the Earth with a decreasing speed. Our simulation shows that the dispersionless injection boundary can be considered coincident with the leading edge of the pulse field, which transports particles toward the Earth across a certain range of local time. Under the same model field, the dispersionless injection boundary shifts eastward for electrons and westward for protons, consistent with the observation results deduced from statistical analysis of multiple spacecraft measurements.


1968 ◽  
Vol 127 (2) ◽  
pp. 307-325 ◽  
Author(s):  
Vera S. Byers ◽  
Eli E. Sercarz

A set of conditions has been described under which primed rabbit lymph nodes produce a secondary antibody response upon in vivo stimulation with a large dose of antigen, but are subsequently "exhausted;" that is, lymph node cultures prepared at intervals following the booster injection cannot be re-stimulated to display tertiary responses. Rabbits given 100-fold less antigen in the booster inoculum were able to give a tertiary response upon in vitro challenge. The system used permits neither induction nor continuation of a primary response to BSA in vitro. Since it could be demonstrated that no memory cells were generated by the booster injection within the intervals between in vivo injection and culture, the tertiary response in nonexhausted nodes must have been due to residual memory cells which remained untriggered by the in vivo booster injection. The unresponsive state was not caused by antibody feedback. These results are interpreted to mean that a population of memory cells can be exhausted by a supraoptimal dose of antigen, rendering the node temporarily incapable of further response. This implies that long-lived memory is not due to asymmetric division of memory cells. The source and fate of memory cells is discussed with regard to this evidence.


2013 ◽  
Vol 868 ◽  
pp. 15-19
Author(s):  
Xing Hua Zhuang ◽  
Da Hong Zhang ◽  
Jun Xie

The Hei47 block, typical tectonic reservoir of the Daqingzi oil field, the west fault of which has great influences on distribution of the remaining oil. Basing on the foundation of 3D geological model of Hei47 block, using the black oil E100 of ECLIPSE software to build the numerical simulation, we found that the remaining oil distribute in the non-injection region,and the peak area is mainly near the west fault.By means of integration of 3D geological modeling and reservoir numerical simulation technology,we can forecast the results of different injection-production ratio and production speed, and choose the best reservoir development scheme. This can be fine guidances for oilfields to establish the potential solutions and improve the recovery ratio.


Vaccine ◽  
2006 ◽  
Vol 24 (16) ◽  
pp. 3084-3086 ◽  
Author(s):  
P KHAWPLOD ◽  
H WILDE ◽  
S SIRIKWIN ◽  
M BENJAWONGKULCHAI ◽  
S LIMUSANNO ◽  
...  

1983 ◽  
Vol 97 (6) ◽  
pp. 1724-1736 ◽  
Author(s):  
M Pacifici ◽  
R Soltesz ◽  
G Thal ◽  
D J Shanley ◽  
D Boettiger ◽  
...  

Polyclonal antibodies were raised in a rabbit against the major proteoglycan of chick sternal cartilage. A total of six antisera was obtained, three after the first booster injection (A1, A2, and A3) and three after the second booster injection (A4, A5, and A6). The A1 antiserum, which was characterized in most detail, immunoprecipitated native as well as chondroitinase ABC-digested or chondroitinase ABC/keratanase-digested cartilage proteoglycan synthesized by cultured chick chondroblasts, but failed to immunoprecipitate the major proteoglycan synthesized by chick skin fibroblasts. This antiserum was also able to immunoprecipitate the cartilage proteoglycan core protein newly synthesized by cultured chondroblasts, but no other major cell protein. However, the late bleed antisera obtained from the same rabbit after a second booster injection reacted with a new chondroblast-specific polypeptide(s) of approximately 60,000 mol wt in addition to the cartilage proteoglycan. By immunofluorescence procedures, the A1 antiserum stained the extracellular proteoglycan matrix of cultured chondroblasts but not that of skin fibroblasts. Following enzymatic removal of the extracellular matrix and cell membrane permeabilization, this antiserum stained primarily a large, juxtanuclear structure. Additional radioautographic evidence suggests that this structure represents the Golgi complex. Similar immunofluorescent staining with antibodies to the cartilage-characteristic Type II collagen revealed that type II procollagen was localized in numerous cytoplasmic, vacuole-like structures which were scattered throughout most of the chondroblast cytoplasm but were notably scanty in the Golgi complex area. In conclusion, our data suggest the transit of the major cartilage proteoglycan through the Golgi complex of cultured chondroblasts and possible differences in the intracellular distribution of newly synthesized cartilage proteoglycan and Type II procollagen.


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