scholarly journals Effect of the novel calcium antagonist AE0047 on responses of isolated dog blood vessels to vasoconstricting agents and to nerve stimulation.

1995 ◽  
Vol 106 (1) ◽  
pp. 19-29 ◽  
Author(s):  
Masakuni NISHIKAWA ◽  
Tomio OKAMURA ◽  
Noboru TODA
Keyword(s):  
Calcium Antagonist ◽  
Blood Vessels ◽  
Nerve Stimulation ◽  
The Novel ◽  
Dog Blood

Hemodynamic and humoral effects of the novel calcium antagonist Ro 40-5967 in patients with hypertension

1992 ◽  
Vol 52 (3) ◽  
pp. 314-323 ◽  
Author(s):  
Rita Schmitt ◽  
Cornells H Kleinbloesem ◽  
Gustav G Belz ◽  
Volkmar Schroeter ◽  
Ulrich Feifel ◽  
...  
Keyword(s):  
Calcium Antagonist ◽  
The Novel

scholarly journals First interventional exchange of a left transvenous phrenic nerve stimulation lead from the novel remedē system

2020 ◽  
Vol 31 (11) ◽  
pp. 3056-3060
Author(s):  
Klaus‐Jürgen Gutleben ◽  
Thomas Eitz ◽  
Randy Westlund ◽  
Jan F. Gummert ◽  
Philipp Sommer ◽  
...  
Keyword(s):  
Nerve Stimulation ◽  
Phrenic Nerve ◽  
The Novel ◽  
Phrenic Nerve Stimulation

scholarly journals Effect of amlodipine, a new calcium antagonist on isolated blood vessels.

1991 ◽  
Vol 97 (3) ◽  
pp. 167-178 ◽  
Author(s):  
Kyozo YAMANAKA ◽  
Makoto SUZUKI ◽  
Shiro MUNEHASU ◽  
Jun-ichi ISHIKO
Keyword(s):  
Calcium Antagonist ◽  
Blood Vessels

scholarly journals Heterogeneity of α1-adrenoceptor subtypes involved in noradrenaline-induced contractions of dog blood vessels.

1993 ◽  
Vol 61 ◽  
pp. 258
Author(s):  
Yoko Kohno ◽  
Hitoshi Saito ◽  
Manabu Takita ◽  
Shigeru Kigoshi ◽  
Ikunobu Muramatsu
Keyword(s):  
Blood Vessels ◽  
Dog Blood

scholarly journals Effect of Hemoglobin on Neurogenic Responses and Cholinergic Parameters in Porcine Cerebral Arteries

1989 ◽  
Vol 9 (2) ◽  
pp. 219-225 ◽  
Author(s):  
Matthew D. Linnik ◽  
Tony Jer-Fu Lee
Keyword(s):  
Blood Vessels ◽  
Nerve Stimulation ◽  
Cerebral Arteries ◽  
Choline Uptake ◽  
Cholinergic Transmission ◽  
Cerebral Blood Vessels ◽  
Mixed Response ◽  
Low Concentrations ◽  
Release Of Acetylcholine ◽  
Neurogenic Vasodilation

Electrically stimulated neurogenic vasodilation and endothelial-dependent cholinergic vasodilation in cerebral arteries are both blocked by hemoglobin. To determine if neurogenic vasodilation has a cholinergic component, we examined the effect of hemoglobin on neurogenic responses and perivascular cholinergic parameters in isolated porcine cerebral arteries. The perfused circle of Willis has a mixed response to transmural nerve stimulation (TNS) that is predominately vasodilation. Exposure to hemoglobin (5 μM) causes constriction of this preparation while simultaneously blocking TNS-induced vasodilation. At similar concentrations, however, hemoglobin did not alter electrically stimulated, tetrodotoxin-sensitive release of acetylcholine. Hemoglobin also had no effect on neuronal choline uptake or esteratic inactivation of acetylcholine. These results demonstrate the ability of low concentrations of hemoglobin to alter cerebral neurogenic vasodilation. The failure of hemoglobin to affect any aspect of cholinergic transmission, however, provides further evidence against a direct vasodilatory role for acetylcholine as a terminal transmitter in isolated cerebral blood vessels.

Relaxant Effects of Butylidenephthalide in Isolated Dog Blood Vessels

Planta Medica ◽  
2002 ◽  
Vol 68 (11) ◽  
pp. 1004-1009 ◽  
Author(s):  
Wun Chang Ko ◽  
Chao-Chiun Liao ◽  
Chih-Hsien Shih ◽  
Chien-Bang Lei ◽  
Chi-Ming Chen
Keyword(s):  
Blood Vessels ◽  
Dog Blood

scholarly journals on the Nervous Regulation of Gill Blood Flow in the Dogfish (Scyliorhinus Canicula)

1984 ◽  
Vol 113 (1) ◽  
pp. 253-267 ◽  
Author(s):  
J. D. METCALFE ◽  
P. J. BUTLER
Keyword(s):  
Blood Vessels ◽  
Nerve Stimulation ◽  
Striated Muscle ◽  
Cranial Nerves ◽  
Striated Muscles ◽  
Motor Innervation ◽  
Scyliorhinus Canicula ◽  
Motor End Plate ◽  
Motor End Plates ◽  
Nervous Transmission

The motor innervation of the gill blood vessels of the dogfish Scyliorhinus canicula L. has been investigated by electrical stimulation of (1) the branchial branches of the IXth and Xth cranial nerves in isolated perfused 1st holobranch preparations and (2) both Xth cranial nerves in whole anaesthetized fish. The observed vascular responses to nerve stimulation appear to be entirely due to contraction of the striated muscles of the gill arch and not to any direct motor innervation of the major gill blood vessels since the responses were blocked only by the drug pancuronium, which blocks striated muscle motor end-plates. The specificity of pancuronium for the motor end-plate of striated muscle in the dogfish was established by showing that it did not block nervous transmission across the cardiac ganglia. The results from the nerve stimulation studies have been investigated further by pharmacological studies on isolated perfused gill preparations. Acetylcholine produces an atropine-sensitive increase in resistance to perfusion, while both adrenalin and noradrenalin decrease the resistance to perfusion.

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