scholarly journals Role of different pathways of the complement cascade in experimental bullous pemphigoid

2006 ◽  
Vol 116 (11) ◽  
pp. 2892-2900 ◽  
Author(s):  
Kelly C. Nelson ◽  
Minglang Zhao ◽  
Pamela R. Schroeder ◽  
Ning Li ◽  
Rick A. Wetsel ◽  
...  
2007 ◽  
Vol 44 (1-3) ◽  
pp. 158
Author(s):  
Kelly E. Carter ◽  
Pamela A. Rose ◽  
Minglang Zhao ◽  
Rick A. Wetsel ◽  
Luis A. Diaz ◽  
...  

1975 ◽  
Vol 37 (4) ◽  
pp. 532-540 ◽  
Author(s):  
Zenro IKEZAWA ◽  
Yo KAMEDA ◽  
Mitsuaki UCHIYAMA ◽  
Hiroshi NAKAJIMA ◽  
Toru BABA

2013 ◽  
Vol 2013 ◽  
pp. 1-7 ◽  
Author(s):  
Hani Sinno ◽  
Satya Prakash

Wound healing is a complex pathway of regulated reactions and cellular infiltrates. The mechanisms at play have been thoroughly studied but there is much still to learn. The health care system in the USA alone spends on average 9 billion dollars annually on treating of wounds. To help reduce patient morbidity and mortality related to abnormal or prolonged skin healing, an updated review and understanding of wound healing is essential. Recent works have helped shape the multistep process in wound healing and introduced various growth factors that can augment this process. The complement cascade has been shown to have a role in inflammation and has only recently been shown to augment wound healing. In this review, we have outlined the biology of wound healing and discussed the use of growth factors and the role of complements in this intricate pathway.


1996 ◽  
Vol 9 (1) ◽  
pp. 34-46 ◽  
Author(s):  
T R Kozel

Fungi have been studied as prototype activators of the complement cascade since the early 1900s. More recently, attention has focused on the role of the complement system in the pathogenesis of fungal infections. The interactions of Cryptococcus neoformans and Candida albicans with the complement system are the most widely characterized; however, all pathogenic fungi examined to date have the ability to initiate the complement cascade. The molecular mechanisms for initiation and regulation of the complement cascade differ from one fungus to another, most likely reflecting differences in the structure of the outer layers of the cell wall. The molecular bases for such differences remain to be identified. Studies of mycoses in experimental animals with induced or congenital deficiencies in the complement system demonstrate that complement is an important innate system for control of fungal infection. Contributions to host resistance include opsonization and generation of inflammatory mediators. Inflammation induced by chemotactic products of the complement system may contribute to the pathogenesis of some fungal infections.


Author(s):  
A. Papadopoulou ◽  
E. Zafiriou ◽  
G.K. Koukoulis ◽  
A.V. Roussaki‐Schulze

The Lancet ◽  
1981 ◽  
Vol 318 (8250) ◽  
pp. 806 ◽  
Author(s):  
Marianne Kieffer ◽  
R.StC. Barnetson
Keyword(s):  

2018 ◽  
Vol 5 ◽  
Author(s):  
Kyle T. Amber ◽  
Manuel Valdebran ◽  
Khalaf Kridin ◽  
Sergei A. Grando
Keyword(s):  

2001 ◽  
Vol 7 (6) ◽  
pp. 367-382 ◽  
Author(s):  
Anthony L. D’Ambrosio ◽  
David J. Pinsky ◽  
E. Sander Connolly

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