scholarly journals Boron Induces Hyperpolarization of Sunflower Root Cell Membranes and Increases Membrane Permeability to K+

1990 ◽  
Vol 93 (2) ◽  
pp. 566-571 ◽  
Author(s):  
Mary K. Schon ◽  
Anton Novacky ◽  
Dale G. Blevins
2014 ◽  
Vol 49 (4) ◽  
pp. 357-361 ◽  
Author(s):  
Maria Podbielkowska ◽  
Alicja Zobel ◽  
Maria Wałęza

A specific influence of cyclophosphamide, an oncostatic drug of the group of alkylating agents, has been demonstrated on cellular respiration and the permeability of cell membranes. The tested drug under the experimental conditions inhibites cell respiration by about 20-30 per cent as compared with the control. The permeability of the plasmalemma and tonoplast de-creased markedly under the action of cyclophosphamide.


Author(s):  
Stefano Romeo

Cholesterol is the most abundant steroid in animals. Not only is it a vital constituent of cell membranes, where it establishes proper membrane permeability and fluidity, but it is also the immediate metabolic precursor of all known steroid hormones and bile acids. Synthesized de novo in cells or absorbed from the diet, cholesterol circulates in the body in association with lipoproteins and is ultimately degraded into bile acids by the liver. Every perturbation of the numerous enzymes involved in cholesterol metabolism leads to impairment in the development and function of the gastrointestinal, cardiovascular, skeletal, and nervous systems.


Blood ◽  
1976 ◽  
Vol 47 (1) ◽  
pp. 121-131 ◽  
Author(s):  
MR Clark ◽  
SB Shohet

Abstract A hybrid erythrocyte model for membrane studies in sickle cell disease has been developed. The model consists of normal red cell membranes containing hemoglobin S and sickle cell membranes containing hemoglobin A. In hybrids, complete hemoglobin exchange has been achieved together with restoration of low membrane permeability to potassium. Normal membranes containing HbS sickle upon deoxygenation and assume the characteristic appearance of irreversibly sickled cells (ISC) after prolonged anoxia. It is suggested that the hybrid model will be useful in defining further the process of ISC formation and in studying the influence of sickle hemoglobin upon the function of the surrounding membrane.


Author(s):  
Helén Svenningsson ◽  
Maria Andersson ◽  
Conny Liljenberg

Author(s):  
Magdalena Wytrwal ◽  
Paulina Knobloch ◽  
Sławomir Lasota ◽  
Marta Michalik ◽  
M. Nowakowska ◽  
...  

The interaction of nanometric synthetic materials with cell membranes is one of the key factors determining their possible cytotoxicity. This work investigated the interaction of polycation nanostructures with lipid and...


2012 ◽  
Vol 93 (5) ◽  
pp. 789-791
Author(s):  
O V Orlova ◽  
V N Oslopov ◽  
S A Sidullina

Aim. To study the influence of triphenyltetradecylphosphonium bromide [(PPh)3P+C14H29] Br- on the cell membranes permeability to Na+ by determining the rate of Na+-Li+-counter transport in erythrocyte membrane depending on it’s variable initial condition. Methods. Blood samples of 10 healthy volunteers with different Na+-Li+-counter transport rate distribution in erythrocyte membrane were analyzed: I quartile (5 subjects) - low permeability, III quartile (5 subjects) - moderately high permeability. Results. Na+-Li+-counter transport rate change in erythrocyte membrane under the influence of triphenyltetradecylphosphonium bromide depends on genetically determined initial membrane permeability. In subjects belonging to III quartile of Na+-Li+-counter transport the initial membrane permeability is high, and after administration of 0.001 and 0.005 μM concentrations of [(PPh)3P+C14H29] Br- membrane permeability to Na+ increased. In subjects belonging to I quartile of Na+-Li+-counter transport (low initial permeability) [(PPh)3P+C14H29] Br- did not alter membrane permeability to Na+. Conclusion. Triphenyltetradecylphosphonium bromide increases erythrocyte membrane permeability to Na+ in subjects belonging to III quartile of Na+-Li+-counter transport and dose not influence subjects belonging to I quartile of Na+-Li+-counter transport.


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