Chemically induced change in nuclear decay rate as a tool for calibratingFe57isomer shifts

1974 ◽  
Vol 9 (9) ◽  
pp. 3666-3669 ◽  
Author(s):  
R. N. Verma ◽  
G. T. Emery
Keyword(s):  
Decay Rate ◽  
Nuclear Decay ◽  
Chemically Induced

scholarly journals Rrp6p/Rrp47p constitutes an independent nuclear turnover system of mature small non-coding RNAs in Saccharomyces cerevisiae

2020 ◽  
Author(s):  
Anusha Chaudhuri ◽  
Subhadeep Das ◽  
Mayukh Banerjea ◽  
Biswadip Das
Keyword(s):  
Saccharomyces Cerevisiae ◽  
Steady State ◽  
Decay Rate ◽  
Nuclear Decay ◽  
Steady State Levels ◽  
Nuclear Exosome ◽  
Non Coding Rnas ◽  
Selective Enhancement

In Saccharomyces cerevisiae, the nuclear exosome/Rrp6p/TRAMP participates in the 3'-end processing of several precursor non-coding RNAs. Here we demonstrate that the depletion of nucleus-specific 3'to 5' exoribonuclease Rrp6p and its co-factor Rrp47p led to the specific and selective enhancement of steady-state levels of mature small non-coding RNAs (sncRNAs) that include 5S and 5.8S rRNAs, snRNAs and snoRNAs, but not 18S and 25S rRNAs. Most importantly, their steady-state enhancement does not require the exosome, TRAMP, CTEXT or Rrp6p-associated Mpp6p. Rrp6p/47p-dependent enhancement of the steady-state levels of sncRNAs is associated with the diminution of their nuclear decay-rate and requires their polyadenylation before targeting by Rrp6p, which is catalyzed by both the canonical and non-canonical poly(A) polymerases, Pap1p and Trf4p. Consistent with this finding, the Rrp6p and Rrp47p were demonstrated to exist as an exosome-independent complex. Thus, Rrp6p-Rrp47p defines a core nuclear exosome-independent novel turnover system that targets the small non-coding RNAs.

Electron capture nuclear decay rate under compression in a confined environment

2021 ◽  
Vol 75 (4) ◽  
Author(s):  
A. Ray ◽  
P. Das ◽  
A. K. Sikdar ◽  
S. Pathak ◽  
N. Aquino ◽  
...  
Keyword(s):  
Decay Rate ◽  
Electron Capture ◽  
Nuclear Decay ◽  
Confined Environment

scholarly journals Observation of enhanced orbital electron-capture nuclear decay rate in a compact medium

2009 ◽  
Vol 679 (2) ◽  
pp. 106-110 ◽  
Author(s):  
A. Ray ◽  
P. Das ◽  
S.K. Saha ◽  
A. Goswami ◽  
A. De
Keyword(s):  
Decay Rate ◽  
Electron Capture ◽  
Nuclear Decay ◽  
Orbital Electron

scholarly journals Laser-Based Means for Accelerating Nuclear Decay Rate

2020 ◽  
Author(s):  
Robert Lascola ◽  
Michael Thomas ◽  
Simona Murph ◽  
David DiPrete ◽  
Kalee Fenker
Keyword(s):  
Decay Rate ◽  
Nuclear Decay

Variations in Nuclear Decay Rates

1977 ◽  
Vol 32 (3-4) ◽  
pp. 345-361 ◽  
Author(s):  
K.-P. Dostal ◽  
M. Nagel ◽  
D. Pabst
Keyword(s):  
Decay Rate ◽  
Subject Matter ◽  
Decay Rates ◽  
Order Effects ◽  
Nuclear Decay ◽  
Pertinent Literature ◽  
The Subject ◽  
Higher Order Effects ◽  
Theoretical Foundations ◽  
Introductory Review

Abstract This paper is intended to provide a concise introductory review of the fundamental principles underlying the higher-order effects of nuclear decay rate variations which may be produced by physical or chemical means. The first part of the paper embraces the theoretical foundations of the subject matter, and the second deals with methodological and experimental questions. Several tables summarize published experimental results and the pertinent literature.

Biological and Surface Properties of C-Type Virus Particles Produced by Novikoff Ascites Hepatoma Cells

1977 ◽  
Vol 35 ◽  
pp. 388-389
Author(s):  
D.C. Hixson ◽  
J.C. Chan ◽  
J.M. Bowen ◽  
E.F. Walborg
Keyword(s):  
Surface Properties ◽  
Ricinus Communis ◽  
Rat Kidney ◽  
Leukemia Virus ◽  
Viral Envelope ◽  
Virus Particles ◽  
Chemically Induced ◽  
Sarcoma Virus ◽  
Hepatocarcinoma Cells ◽  
Type C

Several years ago Karasaki (1) reported the production of type C virus particles by Novikoff ascites hepatocarcinoma cells. More recently, Weinstein (2) has reported the presence of type C virus particles in cell cultures derived from transplantable and primary hepatocellular carcinomas. To date, the biological function of these virus and their significance in chemically induced hepatocarcinogenesis are unknown. The present studies were initiated to determine a possible role for type C virus particles in chemically induced hepatocarcinogenesis. This communication describes results of studies on the biological and surface properties of type C virus associated with Novikoff hepatocarcinoma cells.Ecotropic and xenotropic murine leukemia virus (MuLV) activity in ascitic fluid of Novikoff tumor-bearing rats was assayed in murine sarcoma virus transformed S+L- mouse cells and S+L- mink cells, respectively. The presence of sarcoma virus activity was assayed in non-virus-producing normal rat kidney (NRK) cells. Ferritin conjugates of concanavalin A (Fer-Con wheat germ agglutinin (Fer-WGA), and Ricinus communis agglutinins I and II (Fer-RCAI and Fer-RCAII) were used to probe the structure and topography of saccharide determinants present on the viral envelope.

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