Influence of the gulf stream upon the short-term evolution of a warm-core ring

1983 ◽  
Vol 34 (4) ◽  
pp. 515 ◽  
Author(s):  
TM Joyce ◽  
RW Schmitt ◽  
MC Stalcup

A large anticyclonic, Gulf Stream ring was surveyed during September 1981; two expendable bathythemographic (XBT) surveys, a conductivity-temperature-depth-oxygen (CTD-02) survey, and continuous underway measurements of velocity in the upper 100 m using an acoustic doppler velocimeter were conducted. The initial XBT survey revealed an elliptically shaped ring, over 240 km in diameter, with maximum surface velocities near 2 m s-1, situated well away from the Gulf Stream. However, the later CTD-02 and XBT surveys showed that over a 12-day period a northward meander of the Gulf Stream enveloped part of the ring. This caused a loss of water from the ring, resulting in both a decrease in ring diameter and a shoaling of the thermocline. Using the close relationship between geopotential anomaly and isotherm depths, approximately 33% of the geopotential anomaly signature of the ring is estimated to have been lost due to this event. Clearly, where such interactions with the Gulf Stream occur, they play an important role in the evolution of the warm-core rings.

Author(s):  
Saroj KARKI ◽  
Yuji HASEGAWA ◽  
Masakazu HASHIMOTO ◽  
Hajime NAKAGAWA ◽  
Kenji KAWAIKE

Nephron ◽  
1991 ◽  
Vol 58 (1) ◽  
pp. 13-16 ◽  
Author(s):  
S. Camara ◽  
J.P. de la Cruz ◽  
M.A. Frutos ◽  
P. Sanchez ◽  
Lopez de Novales ◽  
...  
Keyword(s):  

1988 ◽  
Vol 23 (1-2) ◽  
pp. 181-203 ◽  
Author(s):  
Alfred K Hanson ◽  
Carole M Sakamoto-Arnold ◽  
Douglas L Huizenga ◽  
Dana R Kester

2010 ◽  
Vol 37 (8-9) ◽  
pp. 777-789 ◽  
Author(s):  
M. Di Risio ◽  
I. Lisi ◽  
G.M. Beltrami ◽  
P. De Girolamo

2018 ◽  
Author(s):  
Brigitta Kurenbach ◽  
Amy M Hill ◽  
William Godsoe ◽  
Sophie van Hamelsveld ◽  
Jack A Heinemann

Antibiotic resistance is medicine’s climate change: caused by human activity, and resulting in more extreme outcomes. Resistance emerges in microbial populations when antibiotics act on phenotypic variance within the population. This can arise from either genotypic diversity (resulting from a mutation or horizontal gene transfer), or from ‘adaptive’ differences in gene expression due to environmental variation. Adaptive changes can increase fitness allowing bacteria to survive at higher concentrations of the antibiotic. They can also decrease fitness, potentially leading to selection for antibiotic resistance at lower concentrations. There are opportunities for other environmental stressors to promote antibiotic resistance in ways that are hard to predict using conventional assays. Exploiting our observation that commonly used herbicides can increase or decrease the minimum inhibitory concentration (MIC) of different antibiotics, we provide the first comprehensive test of the hypothesis that the rate of antibiotic resistance evolution under specified conditions can increase, regardless of whether a herbicide increases or decreases the antibiotic MIC. Short term evolution experiments were used for various herbicide and antibiotic combinations. We found conditions where acquired resistance arises more frequently regardless of whether the exogenous non-antibiotic agent increased or decreased antibiotic effectiveness. This “damned if you do/damned if you don’t” outcome suggests that the emergence of antibiotic resistance is exacerbated by additional environmental factors that influence competition between bacteria. Our work demonstrates that bacteria may acquire antibiotic resistance in the environment at rates substantially faster than predicted from laboratory conditions.


2011 ◽  
Vol 193 (11) ◽  
pp. 823-832 ◽  
Author(s):  
Marko Wassmann ◽  
Ralf Moeller ◽  
Günther Reitz ◽  
Petra Rettberg

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