The immobility produced by intermittent swim stress is not mediated by serotonin

2008 ◽  
Vol 89 (3) ◽  
pp. 412-423 ◽  
Author(s):  
John P. Christianson ◽  
Sarah Rabbett ◽  
Jennifer Lyckland ◽  
Robert C. Drugan
Keyword(s):  
Swim Stress

Attenuation of the habituation of neophobia by swim stress

1991 ◽  
Author(s):  
T. R. Schachtman ◽  
B. R. Butchart ◽  
J. L. Calton ◽  
S. Reilly
Keyword(s):  
Swim Stress

Anticonvulsive Effect of Swim Stress in Mice

2000 ◽  
Vol 66 (4) ◽  
pp. 879-886 ◽  
Author(s):  
Danka Peričić ◽  
Dubravka Švob ◽  
Maja Jazvinšćak ◽  
Kety Mirković
Keyword(s):  
Swim Stress ◽  
Anticonvulsive Effect

Fluoxetine increases the activity of the ERK-CREB signal system and alleviates the depressive-like behavior in rats exposed to chronic forced swim stress

2008 ◽  
Vol 31 (2) ◽  
pp. 278-285 ◽  
Author(s):  
Xiaoli Qi ◽  
Wenjuan Lin ◽  
Junfa Li ◽  
Huanhuan Li ◽  
Weiwen Wang ◽  
...  
Keyword(s):  
Signal System ◽  
Swim Stress ◽  
Forced Swim ◽  
Forced Swim Stress

Repeated swim stress increases pain-induced expression of c-Fos in the rat lumbar cord

2003 ◽  
Vol 965 (1-2) ◽  
pp. 259-268 ◽  
Author(s):  
Luis Quintero ◽  
Maria Cecilia Cuesta ◽  
Jose Antonio Silva ◽  
Jose Luis Arcaya ◽  
Lorena Pinerua-Suhaibar ◽  
...  
Keyword(s):  
Swim Stress ◽  
Induced Expression ◽  
Lumbar Cord

Hypothalamic noradrenergic and sympathoadrenal control of glycemia after stress

1989 ◽  
Vol 256 (2) ◽  
pp. E231-E235
Author(s):  
G. A. Smythe ◽  
W. S. Pascoe ◽  
L. H. Storlien
Keyword(s):  
Insulin Release ◽  
Acute Stress ◽  
Serum Insulin ◽  
Elevated Serum ◽  
Serum Glucose ◽  
Neural Pathways ◽  
Swim Stress ◽  
Insulin Levels ◽  
Glucose Levels ◽  
Positive Correlations

Central noradrenergic pathways play a significant role in mediating blood glucose levels after neuroglycopenia. To further investigate hypothalamic noradrenergic neuronal activity (NNA) and sympathoadrenal influences in glucoregulation, we studied the effects of acute stress on glycemia and insulin release in normal and adrenalectomized (ADRX) rats. Within 5 min of exposure of rats to ether or cold-swim stress, significant positive correlations were evident between hypothalamic NNA and serum glucose levels (r = 0.70, P less than 0.001; at 15 min r = 0.78, P less than 0.0001). Five minutes after stress in the intact rat, insulin release was inhibited and serum insulin levels inversely correlated to hypothalamic NNA (r = 0.45, P less than 0.05). This relationship between insulin and NNA was no longer present 15 min after stress, but the levels of insulin remained inappropriately low with respect to the elevated serum glucose levels (approximately 30% above basal). Blockade of sympathetic noradrenergic pathways by treatment of intact rats with guanethidine prevented the rise in glucose after cold-swim stress but did not prevent the inhibition of insulin release. Fifteen minutes after exposure of ADRX rats to cold-swim stress their hypothalamic NNA and serum glucose levels were similar to intact animals. However, in contrast to their intact counterparts, serum insulin levels were significantly elevated (P less than 0.01). These data are consistent with central noradrenergic neural pathways directly mediating hepatic glucose release and indirectly inhibiting pancreatic insulin release via activation of adrenal medullary catecholamines.

CART expression in limbic regions of rat brain following forced swim stress: Sex differences

Neuropeptides ◽  
2006 ◽  
Vol 40 (3) ◽  
pp. 185-193 ◽  
Author(s):  
Burcu Balkan ◽  
Oguz Gozen ◽  
Gorkem Yararbas ◽  
Ersin O. Koylu ◽  
Serra Akinturk ◽  
...  
Keyword(s):  
Sex Differences ◽  
Rat Brain ◽  
Swim Stress ◽  
Forced Swim ◽  
Forced Swim Stress ◽  
Limbic Regions

Levorphanol and swim stress-induced analgesia in selectively bred mice: evidence for genetic commonalities

1993 ◽  
Vol 608 (2) ◽  
pp. 353-357 ◽  
Author(s):  
Przemyslaw Marek ◽  
Jeffrey S. Mogil ◽  
John K. Belknap ◽  
Bogdan Sadowski ◽  
John C. Liebeskind
Keyword(s):  
Swim Stress
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