Ascorbic acid prevents vascular dysfunction induced by oral glucose load in healthy subjects

2012 ◽  
Vol 23 (1) ◽  
pp. 54-57 ◽  
Author(s):  
Sergio De Marchi ◽  
Manlio Prior ◽  
Anna Rigoni ◽  
Sara Zecchetto ◽  
Fanny Rulfo ◽  
...  
Keyword(s):  
Ascorbic Acid ◽  
Healthy Subjects ◽  
Vascular Dysfunction ◽  
Oral Glucose ◽  
Oral Glucose Load ◽  
Glucose Load

The disposal of an oral glucose load in healthy subjects. A quantitative study

Diabetes ◽  
1985 ◽  
Vol 34 (6) ◽  
pp. 580-588 ◽  
Author(s):  
E. Ferrannini ◽  
O. Bjorkman ◽  
G. A. Reichard ◽  
A. Pilo ◽  
M. Olsson ◽  
...  
Keyword(s):  
Quantitative Study ◽  
Healthy Subjects ◽  
Oral Glucose ◽  
Oral Glucose Load ◽  
Glucose Load

Growth hormone nadir during oral glucose load depends on waist circumference, gender and age: normative data in 231 healthy subjects

2011 ◽  
Vol 74 (2) ◽  
pp. 234-240 ◽  
Author(s):  
Annamaria Colao ◽  
Rosario Pivonello ◽  
Renata S. Auriemma ◽  
Ludovica F. S. Grasso ◽  
Mariano Galdiero ◽  
...  
Keyword(s):  
Growth Hormone ◽  
Waist Circumference ◽  
Healthy Subjects ◽  
Normative Data ◽  
Oral Glucose ◽  
Oral Glucose Load ◽  
Glucose Load ◽  
Gender And Age

scholarly journals Glucose tolerance and insulin responsiveness in Gitelman syndrome patients

2017 ◽  
Vol 6 (4) ◽  
pp. 243-252 ◽  
Author(s):  
Tao Yuan ◽  
Lanping Jiang ◽  
Chen Chen ◽  
Xiaoyan Peng ◽  
Min Nie ◽  
...  
Keyword(s):  
Insulin Secretion ◽  
Insulin Sensitivity ◽  
Glucose Metabolism ◽  
Healthy Subjects ◽  
Potassium Level ◽  
Serum Glucose ◽  
Oral Glucose ◽  
Oral Glucose Load ◽  
Glucose Load

Objective Impaired glucose metabolism and insulin sensitivity have been reported in patients with Gitelman syndrome (GS), but insulin secretion and the related mechanisms are not well understood. Design and methods The serum glucose levels, insulin secretion and insulin sensitivity were evaluated in patients with GS (n = 28), patients with type 2 diabetes mellitus (DM) and healthy individuals (n = 20 in both groups) using an oral glucose tolerance test. Serum and urine sodium, potassium and creatinine levels were measured at 0, 30, 60, 120 and 180 min after an oral glucose load was administered. Results The areas under the serum glucose curves were higher in the GS patients than those in the healthy controls (17.4 ± 5.1 mmol·h/L vs 14.5 ± 2.8 mmol·h/L, P = 0.02) but lower than those in the DM patients (24.8 ± 5.3 mmol·h/L, P < 0.001). The areas under the serum insulin curves and the insulin secretion indexes in GS patients were higher than those in DM patients and lower than those in healthy subjects. The insulin secretion-sensitivity index of GS patients was between that of healthy subjects and DM patients, but the insulin sensitivity indices were not different among the three groups. After one hour of glucose administration, the serum potassium level significantly decreased from baseline, and the urinary potassium-to-creatinine ratio increased gradually and peaked at 2 h. Conclusions Glucose metabolism and insulin secretion were impaired in GS patients, but insulin sensitivity was comparable between GS patients and patients with type 2 DM. After administration of an oral glucose load, the plasma potassium level decreased in GS patients due to the increased excretion of potassium in the urine.

The Disposal of an Oral Glucose Load in Healthy Subjects: A Quantitative Study

Diabetes ◽  
1985 ◽  
Vol 34 (6) ◽  
pp. 580-588 ◽  
Author(s):  
E. Ferrannini ◽  
O. Bjorkman ◽  
G. A. Reichard ◽  
A. Pilo ◽  
M. Olsson ◽  
...  
Keyword(s):  
Quantitative Study ◽  
Healthy Subjects ◽  
Oral Glucose ◽  
Oral Glucose Load ◽  
Glucose Load

A new concept for the evaluation of the blood glucose and insulin responses of healthy subjects following an oral glucose load

1974 ◽  
Vol 11 (4) ◽  
pp. 265-276 ◽  
Author(s):  
Olga Szabo ◽  
Werner Oppermann ◽  
Rafael A. Camerini-Dávalos ◽  
Carl Victor
Keyword(s):  
Blood Glucose ◽  
Healthy Subjects ◽  
Oral Glucose ◽  
Oral Glucose Load ◽  
Glucose Load ◽  
Insulin Responses

scholarly journals Administration of tauroursodeoxycholic acid prevents endothelial dysfunction caused by an oral glucose load

2016 ◽  
Vol 130 (21) ◽  
pp. 1881-1888 ◽  
Author(s):  
Lauren K. Walsh ◽  
Robert M. Restaino ◽  
Martha Neuringer ◽  
Camila Manrique ◽  
Jaume Padilla
Keyword(s):  
Endoplasmic Reticulum ◽  
Single Dose ◽  
Endothelial Dysfunction ◽  
Oral Administration ◽  
Vascular Dysfunction ◽  
Oral Glucose ◽  
Oral Glucose Load ◽  
Glucose Load ◽  
Tauroursodeoxycholic Acid ◽  
Dose Oral

The key finding of the present study is that vascular dysfunction caused by an oral glucose load can be prevented by a single dose oral administration of tauroursodeoxycholic acid (TUDCA), a compound known to alleviate endoplasmic reticulum stress (ER).

Reduction of hyperglycemia after oral glucose load by the new α2-adrenergic receptor antagonist SL 84.0418 in healthy subjects

1994 ◽  
Vol 55 (3) ◽  
pp. 338-345 ◽  
Author(s):  
Ivan Berlin ◽  
Pierre Rosenzweig ◽  
Stephan Chalon ◽  
Eliane Fuseau ◽  
Catherine Landault ◽  
...  
Keyword(s):  
Receptor Antagonist ◽  
Adrenergic Receptor ◽  
Healthy Subjects ◽  
Oral Glucose ◽  
Oral Glucose Load ◽  
Glucose Load

scholarly journals Incretin responses to oral glucose load in Japanese non-obese healthy subjects

2011 ◽  
Vol 2 (1) ◽  
pp. 20-28 ◽  
Author(s):  
Etsuko Nagai ◽  
Tomoyuki Katsuno ◽  
Jun-ichiro Miyagawa ◽  
Kosuke Konishi ◽  
Masayuki Miuchi ◽  
...  
Keyword(s):  
Healthy Subjects ◽  
Oral Glucose ◽  
Oral Glucose Load ◽  
Glucose Load
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