The influence of dietary fat on the lipogenic activity and fatty acid composition of rat white adipose tissue

Lipids ◽  
1987 ◽  
Vol 22 (5) ◽  
pp. 338-344 ◽  
Author(s):  
Gary J. Nelson ◽  
Darshan S. Kelley ◽  
Perla C. Schmidt ◽  
Claire M. Serrato
Keyword(s):  
Adipose Tissue ◽  
Fatty Acid Composition ◽  
Fatty Acid ◽  
Acid Composition ◽  
White Adipose Tissue ◽  
Dietary Fat ◽  
Lipogenic Activity

scholarly journals The Combination of Resveratrol and Quercetin Attenuates Metabolic Syndrome in Rats by Modifying the Serum Fatty Acid Composition and by Upregulating SIRT 1 and SIRT 2 Expression in White Adipose Tissue

2015 ◽  
Vol 2015 ◽  
pp. 1-9 ◽  
Author(s):  
Ana Elena Peredo-Escárcega ◽  
Verónica Guarner-Lans ◽  
Israel Pérez-Torres ◽  
Sergio Ortega-Ocampo ◽  
Elizabeth Carreón-Torres ◽  
...  
Keyword(s):  
Metabolic Syndrome ◽  
Fatty Acids ◽  
Drinking Water ◽  
Adipose Tissue ◽  
Fatty Acid Composition ◽  
Fatty Acid ◽  
Acid Composition ◽  
White Adipose Tissue ◽  
Serum Fatty Acid ◽  
Serum Fatty Acid Composition

Resveratrol (RSV) and quercetin (QRC) modify energy metabolism and reduce cardiovascular risk factors included in the metabolic syndrome (MetS). These natural compounds upregulate and activate sirtuins (SIRTs), a family of NAD-dependent histone deacetylases. We analyzed the effect of two doses of a commercial combination of RSV and QRC on serum fatty acid composition and their regulation of SIRTs 1–3 and PPAR-γexpression in white adipose tissue. MetS was induced in Wistar rats by adding 30% sucrose to drinking water for five months. Rats were divided into control and two groups receiving the two different doses of RSV and QRC in drinking water daily for 4 weeks following the 5 months of sucrose treatment. Commercial kits were used to determine serum parameters and the expressions of SIRTs in WAT were analysed by western blot. In MetS rats body mass, central adiposity, insulin, triglycerides, non-HDL-C, leptin, adiponectin, monounsaturated fatty acids (MUFAs), and nonesterified fatty acids (NEFAs) were increased, while polyunsaturated fatty acids (PUFAs) and HDL-C were decreased. SIRT 1 and SIRT 2 were downregulated, while PPAR-γwas increased. RSV + QRC administration improved the serum health parameters modified by MetS and upregulate SIRT 1 and SIRT 2 expression in white abdominal tissue in MetS animals.

The influence of type and level of dietary fat upon fatty acid composition in the adipose tissue of broiler chickens

1986 ◽  
Vol 14 (4) ◽  
pp. 772-772
Author(s):  
BILQUEES KHAN ◽  
SURESH PATEL ◽  
VICTOR NWOSU ◽  
ABID R. KARIM ◽  
DAVID J. MASLIN
Keyword(s):  
Adipose Tissue ◽  
Fatty Acid Composition ◽  
Fatty Acid ◽  
Acid Composition ◽  
Dietary Fat ◽  
Broiler Chickens

MODE OF ACTION OF CHOLINE: I. FATTY ACID COMPOSITION OF LIVER, SERUM, AND ADIPOSE TISSUE OF CHOLINE-DEFICIENT RATS

1966 ◽  
Vol 44 (6) ◽  
pp. 713-724 ◽  
Author(s):  
A. Chalvardjian
Keyword(s):  
Adipose Tissue ◽  
Fatty Acid Composition ◽  
Fatty Acid ◽  
Acid Composition ◽  
Dietary Fat ◽  
Fatty Acid Pattern ◽  
Albino Rats ◽  
Gas Liquid Chromatography ◽  
Serum Triglycerides ◽  
Triglyceride Fraction

Young albino rats were fed choline-deficient and choline-supplemented diets, fat-free and containing two different levels of fat (4%, 30%), for 2 weeks. The fatty acid pattern of the major lipid fractions of liver, serum, and adipose tissue was determined by thin-layer and gas–liquid chromatography. It was found that (a) no quantitative similarities in fatty acid pattern existed between hepatic triglycerides on the one hand and adipose tissue, serum triglycerides, and dietary fat on the other, but the fatty acid composition of hepatic triglycerides bore a closer resemblance to that of dietary fat when the latter was increased to 30%; (b) under the conditions of this experiment, the level of dietary fat had a more marked effect on altering the fatty acid composition of tissues, especially the triglyceride fraction, than the level of choline in the diet; and (c) a relative preponderance of 16-carbon fatty acids occurred in hepatic triglycerides of choline-deficient animals fed the fat-free and low-fat diets.

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