The portal bifurcation and the termination of the hepatic veins: an anatomical investigation into the projection of the ultrasonographically important hepatic vessels onto the anterior body wall

1991 ◽  
Vol 13 (3) ◽  
pp. 187-195 ◽  
Author(s):  
T Sauer ◽  
H Loeweneck
2016 ◽  
Vol 170 (7) ◽  
pp. 1889-1894 ◽  
Author(s):  
Medis Çöllü ◽  
Şirin Yüksel ◽  
Başak Kumbasar Şirin ◽  
Latif Abbasoğlu ◽  
Yasemin Alanay
Keyword(s):  

2019 ◽  
Author(s):  
Rebecca J. Androwski ◽  
Nadeem Asad ◽  
Janet G. Wood ◽  
Allison Hofer ◽  
Steven Locke ◽  
...  

ABSTRACTStress influences the shape of dendritic arbors in neurons. During the stress-induced dauer stage of Caenorhabditis elegans, the IL2 neurons arborize to cover the anterior body wall. In contrast, the FLP neurons arborize to cover the anterior body wall during non-dauer development. Previous work showed that the membrane-bound receptor DMA-1 regulates FLP branching as part of a larger protein complex. Using forward genetics, we show that the IL2 neurons also use the DMA-1 complex to regulate branching. To understand the coordination of the IL2s and FLPs we conducted a time-course examination of FLPs and found previously undescribed branching patterns indicating a neighborhood effect wherein the FLPs and IL2s in the anterior have differential branching compared to the more posteriorly located PVD arborizing neurons. To determine how the IL2s and FLPs differentially regulate branching, we examined several regulators of DMA-1 localization. We show that the unfolded protein response sensor IRE-1, required for FLP branching, is only required for dauer-specific branching at elevated temperatures. Interestingly, we found that ire-1 mutants have broad, organism-wide temperature-dependent effects on dauer remodeling, suggesting a previously undescribed role for IRE-1 in phenotypic plasticity. We also found that defects in other regulators of dauer remodeling including DAF-16/FOXO, DAF-9/Cytochrome P450, and DAF-18/PTEN are required for proper IL2 arborization, but dispensable for FLP branching. Interestingly, we find that TOR adaptor protein DAF-15/RAPTOR is both required for promoting IL2 branching and inhibiting precocious development of the FLPs. Our results demonstrate specific genotypic by environmental interactions regulating dendrite arborization.SIGNIFICANCE STATEMENTNeurons have extensions called dendrites that receive information. Dendrites are often elaborately shaped with many branches. Adverse stress can reduce branching in some neurons, while increasing it in others. How stress can cause some neurons to change shape is unclear. We previously found a set of neurons in the head of the well-studied roundworm C. elegans that undergo reversible branching following exposure to specific adverse environmental conditions. Using various genetic tools, we find that branching in these neurons is controlled by a combination of branching genes common to many neuron types and others that only regulate branching in stress-responsive neurons. Our data demonstrate how experiencing stress acts through genetics pathways to cause changes to specific neurons.


1974 ◽  
Vol 52 (1) ◽  
pp. 21-22 ◽  
Author(s):  
K. A. Wright ◽  
J. Chan

A sense organ, located just posterior to the nerve ring in the ventral line of the body wall of Capillaria hepatica, is described. It consists of a pore opening through the cuticle to a cuticle-lined chamber into which four dendritic processes extend. The structure of the organ suggests that it may be chemoreceptive.


2020 ◽  
pp. 028418512092892
Author(s):  
Franz GM Poch ◽  
Beatrice Geyer ◽  
Christina A Neizert ◽  
Ole Gemeinhardt ◽  
Stefan M Niehues ◽  
...  

Background Vascular cooling effects are a well-known source for tumor recurrence in thermal in situ ablation techniques for hepatic malignancies. Microwave ablation (MWA) is an ablation technique to be considered in the treatment of malignant liver tumors. The impact of vascular cooling in MWA is still controversial. Purpose To evaluate the influence of different intrahepatic vessel types, vessel sizes, and vessel-to-antenna-distances on MWA geometry in vivo. Material and Methods Five MWAs (902–928 MHz) were performed with an energy input of 24.0 kJ in three porcine livers in vivo. MWA lesions were cut into 2-mm slices. The minimum and maximum radius of the ablation area was measured for each slice. Distances were measured from ablation center toward all adjacent hepatic vessels with a diameter of ≥1 mm and within a perimeter of 20 mm around the antenna. The respective vascular cooling effect relative to the maximum ablation radius was calculated. Results In total, 707 vessels (489 veins, 218 portal fields) were detected; 370 (76%) hepatic veins and 185 (85%) portal fields caused a cooling effect. Portal fields resulted in higher cooling effects (37%) than hepatic veins (26%, P < 0.01). No cooling effect could be observed in close proximity of vessels within the central ablation zone. Conclusion Hepatic vessels influenced MWA zones and caused a distinct cooling effect. Portal fields resulted in more pronounced cooling effect than hepatic veins. No cooling effect was observed around vessels situated within the central white zone.


Author(s):  
Abdoulaye Diakhate ◽  
Mame D. N. Guèye ◽  
Abdou K. Diallo ◽  
Mouhamadou Wade ◽  
Simon B. Ndour ◽  
...  

We report in this work, an extremely rare and major case of anterior body wall defects included ectopia cordis define by abnormal location of heart outside of the thorax. This case was diagnosed at the maternity of Ourossogui regional hospital center, in Senegal. Any scan was performed during the pregnancy. Newborn died 10 minutes after birth. Ectopia cordis is related to a possible ventral midline developmental abnormality. It’s associated to other midline abnormalities and is a part of pentalogy of Cantrell. An X-linked genetic abnormality.


1997 ◽  
Vol 78 (03) ◽  
pp. 1150-1156 ◽  
Author(s):  
Christina Jern ◽  
Heléne Seeman-Lodding ◽  
Bjӧrn Biber ◽  
Ola Winsӧ ◽  
Sverker Jern

SummaryExperimental data indicate large between-organs variations in rates of synthesis of tissue-type plasminogen activator (t-PA), which may reflect important differences in the capacity for constitutive and stimulated t-PA release from the vascular endothelium. In this report we describe a new multiple-organ experimental in vivo model for simultaneous determinations of net release/uptake rates of t-PA across the coronary, splanchnic, pulmonary, and hepatic vascular beds. In eleven intact anesthetized pigs, blood samples were obtained simultaneously from the proximal aorta, coronary sinus, pulmonary artery, and portal and hepatic veins. Plasma flows were monitored separately for each vascular region. Total plasma t-PA was determined by ELISA with a porcine t-PA standard. Regional net release/uptake rates were defined as the product of arteriovenous concentration gradients and local plasma flows. The net release of t-PA across the splanchnic vascular bed was very high, with a mean output of 1,919 ng total t-PA X min-1 (corresponding to 90 ng per min and 100 g tissue). The net coronary t-PA release was 68 ng X min-1 (30 ng X min-1 X 100 g"1)- Pulmonary net fluxes of t-PA were variable without any significant net t-PA release. The net hepatic uptake rate was 4,855 ng X min-1 (436 ng X min-1 X 100 g-1). Net trans-organ changes of active t-PA mirrored those of total t-PA. The results demonstrate marked regional differences in net release rates of t-PA in vivo. The experimental model we present offers new possibilities for evaluation of regional secretion patterns in the intact animal.


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