scholarly journals Genetic map of seven polymorphic markers comprising a single linkage group on rat Chromosome 5

1993 ◽  
Vol 4 (11) ◽  
pp. 670-675 ◽  
Author(s):  
Ellen A. Goldmuntz ◽  
Elaine F. Remmers ◽  
Hongbin Zha ◽  
Peter Mathern ◽  
Ying Du ◽  
...  
Genomics ◽  
1992 ◽  
Vol 14 (3) ◽  
pp. 618-623 ◽  
Author(s):  
Elaine F. Remmers ◽  
Ellen A. Goldmuntz ◽  
Joseph M. Cash ◽  
Leslie J. Crofford ◽  
Barbara Misiewicz-Poltorak ◽  
...  

1993 ◽  
Vol 31 (9-10) ◽  
pp. 441-448 ◽  
Author(s):  
Peter Mathern ◽  
Ellen A. Goldmuntz ◽  
Hongbin Zha ◽  
Ying Du ◽  
Leslie J. Crofford ◽  
...  

1994 ◽  
Vol 5 (9) ◽  
pp. 538-541 ◽  
Author(s):  
H. Zha ◽  
E. F. Remmers ◽  
Y. Du ◽  
E. A. Goldmuntz ◽  
P. Mathern ◽  
...  

1995 ◽  
Vol 6 (1) ◽  
pp. 25-30 ◽  
Author(s):  
G. E. Truett ◽  
H. J. Jacob ◽  
J. Miller ◽  
G. Drouin ◽  
N. Bahary ◽  
...  
Keyword(s):  

Genetics ◽  
1996 ◽  
Vol 142 (4) ◽  
pp. 1277-1288
Author(s):  
Stephen L Johnson ◽  
Michael A Gates ◽  
Michele Johnson ◽  
William S Talbot ◽  
Sally Horne ◽  
...  

Abstract The ease of isolating mutations in zebrafish will contribute to an understanding of a variety of processes common to all vertebrates. To facilitate genetic analysis of such mutations, we have identified DNA polymorphisms closely linked to each of the 25 centromeres of zebrafish, placed centromeres on the linkage map, increased the number of mapped PCR-based markers to 652, and consolidated the number of linkage groups to the number of chromosomes. This work makes possible centromere-linkage analysis, a novel, rapid method to assign mutations to a specific linkage group using half-tetrads.


Genome ◽  
1993 ◽  
Vol 36 (4) ◽  
pp. 712-724 ◽  
Author(s):  
Dave Pilgrim

A genetic approach was taken to identify new transposable element Tc1 -dependent polymorphisms on the left end of linkage group III in the nematode Caenorhabditis elegans. The cloning of the genomic DNA surrounding the Tc1 allowed the selection of overlapping clones (from the collection being used to assemble the physical map of the C. elegans genome). A contig of approximately 600–800 kbp in the region has been identified, the genetic map of the region has been refined, and 10 new RFLPs as well as at least four previously characterized genetic loci have been positioned onto the physical map, to the resolution of a few cosmids. This analysis demonstrated the ability to combine physical and genetic mapping for the rapid analysis of large genomic regions (0.5–1 Mbp) in genetically amenable eukaryotes.Key words: Caenorhabditis elegans, genome analysis, RFLP, physical map, genetic map.


1995 ◽  
Vol 6 (7) ◽  
pp. 459-463 ◽  
Author(s):  
E. A. Goldmuntz ◽  
E. F. Remmers ◽  
Y. Du ◽  
H. Zha ◽  
P. Mathern ◽  
...  

Genomics ◽  
1990 ◽  
Vol 6 (1) ◽  
pp. 94-99 ◽  
Author(s):  
Settara C. Chandrasekharappa ◽  
Michelle S. Rebelsky ◽  
Thomas A. Firak ◽  
Michelle M. Le Beau ◽  
Carol A. Westbrook

Genomics ◽  
2006 ◽  
Vol 87 (1) ◽  
pp. 1-29 ◽  
Author(s):  
June E. Swinburne ◽  
Mike Boursnell ◽  
Gemma Hill ◽  
Louise Pettitt ◽  
Twink Allen ◽  
...  

Genetics ◽  
1997 ◽  
Vol 146 (3) ◽  
pp. 1049-1060 ◽  
Author(s):  
David P King ◽  
Martha Hotz Vitaterna ◽  
Anne-Marie Chang ◽  
William F Dove ◽  
Lawrence H Pinto ◽  
...  

Clock is a semidominant mutation identified from an N-ethyl-N-nitrosourea mutagenesis screen in mice. Mice carrying the Clock mutation exhibit abnormalities of circadian behavior, including lengthening of endogenous period and loss of rhythmicity. To identify the gene affected by this mutation, we have generated a high-resolution genetic map (>1800 meioses) of the Clock locus. We report that Clock is 0.7 cM distal of Kit on mouse chromosome 5. Mapping shows that Clock lies within the W19H deletion. Complementation analysis of different Clock and W19H compound genotypes indicates that the Clock mutation behaves as an antimorph. This antimorphic behavior of Clock strongly argues that Clock defines a gene centrally involved in the mammalian circadian system.


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