Short adrenergic neurons and catecholamine-containing cells in vas deferens and accessory male genital glands of different mammals

1965 ◽  
Vol 66 (2) ◽  
pp. 300-320 ◽  
Author(s):  
Christer Owman ◽  
Nils O. Sj�strand
1970 ◽  
Vol 64 (3) ◽  
pp. 459-465 ◽  
Author(s):  
Ch. Owman ◽  
N.-O. Sjöberg ◽  
N. O. Sjöstrand ◽  
G. Swedin

ABSTRACT The effect of prolonged treatment with high doses of oestrogen and/or progesterone on the amount of adrenergic transmitter in the short adrenergic neurons of the male reproductive tract of castrated rats has been studied by chemical determinations and histochemical demonstration of noradrenaline. Oestrogen, progesterone, or a combination of both, had no overt effect on the total content or on the concentration of noradrenaline in the male genital organs. The results are discussed in the light of recent findings that the content of the noradrenaline transmitter in the short adrenergic neurons to the female genital tract is markedly influenced by these female sex hormones.


Medicine ◽  
2019 ◽  
Vol 98 (11) ◽  
pp. e14843 ◽  
Author(s):  
Jigang Jing ◽  
Hua Zhuang ◽  
Yan Luo ◽  
Huijiao Chen ◽  
Yaping Rao
Keyword(s):  

Author(s):  
P.E. Gibbs

Laboratory breeding of the dog-whelk, Nucella lapillus, has established that the male-sterilizing Dumpton Syndrome (DS)—underdevelopment, or non-development (aphally), of the penis, incomplete formation (non-closure) of the vas deferens, resulting in a split prostate—can be readily observed in male F1 progeny. Cultivated under high ambient concentrations of the antifouling agent tributyltin (TBT), DS-carrying females can be recognized by their lesser degree of masculinization (imposex): sterilization is thereby avoided. When Dumpton females are crossed, under high ambient TBT, with individuals from a non-DS-affected population (Bude, North Cornwall) DS is absent from both sexes. Crosses of these F1 progeny result in F2 progeny exhibiting the classic DS symptoms in both sexes. A Mendelian mechanism for DS inheritance is suggested by the data.


2009 ◽  
Vol 53 (4) ◽  
Author(s):  
C. Squillacioti ◽  
A. De Luca ◽  
S. Paino ◽  
E. Langella ◽  
N. Mirabella
Keyword(s):  

2003 ◽  
Vol 59 (9) ◽  
pp. 1999-2016 ◽  
Author(s):  
Nicola Mirabella ◽  
Caterina Squillacioti ◽  
Ettore Varricchio ◽  
Angelo Genovese ◽  
Giuseppe Paino

Author(s):  
P. E. Gibbs

Tributyltin (TBT) pollution has exterminated populations of the dog-whelk Nucella lapillus along most of the north Kent coast (Thames Estuary) but the species survives as a small enclave around the North Foreland. Males in this enclave exhibit an unusual defect involving the non-development or partial development of the genital system: about 10% lack penes, or have undersized penes, and their gonoducts (vas deferens and prostate) are incompletely developed; in some cases, spermatogenesis appears to be retarded. Laboratory-bred animals display the same characters. This deficiency (‘Dumpton Syndrome’) is manifest also in the atypical development of male sex organs on the females (‘imposex’) induced by exposure to tributyltin (TBT). The evidence points to Dumpton Syndrome being a genetic disorder which has lessened the sterilizing effect of imposex and thereby has permitted the survival of this isolated enclave in an area of high TBT pollution.


1970 ◽  
Vol 29 (1) ◽  
pp. 30-32
Author(s):  
RL Gurubacharya ◽  
SM Gurubacharya

The genitourinary tract is the most common extrapulmonary site affected by tuberculosis. The male genital organs are involved in more than 50% of patients. The epididymis is the commonest structure to be involved, followed by the seminal vesicles, prostate, testis, and the vas deferens. An isolated tuberculous orchitis without epididymal involvement is rare. This case report describes extra pulmonary tuberculosis with exclusively testicular presentation. The confirmatory diagnosis of which was made by FNAC of the testis. It provides a successful diagnosis, thereby preventing unnecessary orchidectomy. Key words: genitourinary tuberculosis, testis, USG, FNAC   doi:10.3126/jnps.v29i1.1598 J. Nepal Paediatr. Soc. Vol.29(1) p.30-32


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