amyloid precursor protein ectodomain Latest Research Papers

Peptides of Presenilin-1 Bind the Amyloid Precursor Protein Ectodomain and Offer a Novel and Specific Therapeutic Approach to Reduce ß-Amyloid in Alzheimer’s Disease

PLoS ONE ◽

10.1371/journal.pone.0122451 ◽

2015 ◽

Vol 10 (4) ◽

pp. e0122451 ◽

Cited By ~ 4

Author(s):

Nazneen N. Dewji ◽

S. Jonathan Singer ◽

Eliezer Masliah ◽

Edward Rockenstein ◽

Mihyun Kim ◽

...

Keyword(s):

Alzheimer’S Disease ◽

Alzheimer's Disease ◽

Amyloid Precursor Protein ◽

Therapeutic Approach ◽

Precursor Protein ◽

Presenilin 1 ◽

Amyloid Precursor Protein Ectodomain

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Establishment and characterization of RNA-edited serotonin 2C receptor isoform cell models and alteration of amyloid precursor protein ectodomain secretion in HEK293 APPSwe cells

Human Cell ◽

10.1007/s13577-011-0014-3 ◽

2011 ◽

Vol 24 (2) ◽

pp. 104-111 ◽

Cited By ~ 1

Author(s):

Lingchen Guo ◽

Jing Zhang ◽

Qixin Yan ◽

Ming Yin

Keyword(s):

Amyloid Precursor Protein ◽

Precursor Protein ◽

Cell Models ◽

Serotonin 2C Receptor ◽

Amyloid Precursor Protein Ectodomain

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Faculty Opinions recommendation of The secreted beta-amyloid precursor protein ectodomain APPs alpha is sufficient to rescue the anatomical, behavioral, and electrophysiological abnormalities of APP-deficient mice.

Faculty Opinions – Post-Publication Peer Review of the Biomedical Literature ◽

10.3410/f.1088987.542124 ◽

2007 ◽

Author(s):

Karl-Peter Giese

Keyword(s):

Amyloid Precursor Protein ◽

Beta Amyloid ◽

Precursor Protein ◽

Deficient Mice ◽

Amyloid Precursor Protein Ectodomain

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The Secreted -Amyloid Precursor Protein Ectodomain APPs Is Sufficient to Rescue the Anatomical, Behavioral, and Electrophysiological Abnormalities of APP-Deficient Mice

Journal of Neuroscience ◽

10.1523/jneurosci.1026-07.2007 ◽

2007 ◽

Vol 27 (29) ◽

pp. 7817-7826 ◽

Cited By ~ 224

Author(s):

S. Ring ◽

S. W. Weyer ◽

S. B. Kilian ◽

E. Waldron ◽

C. U. Pietrzik ◽

...

Keyword(s):

Amyloid Precursor Protein ◽

Precursor Protein ◽

Deficient Mice ◽

Amyloid Precursor Protein Ectodomain

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Regulation of the amyloid precursor protein ectodomain shedding by the 5-HT4receptor and Epac

FEBS Letters ◽

10.1016/j.febslet.2005.01.010 ◽

2005 ◽

Vol 579 (5) ◽

pp. 1136-1142 ◽

Cited By ~ 40

Author(s):

Sylvain Robert ◽

Marjorie Maillet ◽

Eric Morel ◽

Jean-Marie Launay ◽

Rodolphe Fischmeister ◽

...

Keyword(s):

Amyloid Precursor Protein ◽

Precursor Protein ◽

Ectodomain Shedding ◽

Protein Ectodomain Shedding ◽

Amyloid Precursor Protein Ectodomain

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Posttranslational Modifications of Amyloid Precursor Protein: Ectodomain Phosphorylation and Sulfation

Alzheimer's Disease ◽

10.1385/1-59259-195-7:149 ◽

2003 ◽

pp. 149-168 ◽

Cited By ~ 2

Author(s):

Jochen Walter ◽

Christian Haass

Keyword(s):

Amyloid Precursor Protein ◽

Posttranslational Modifications ◽

Precursor Protein ◽

Amyloid Precursor Protein Ectodomain

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Constitutive shedding of the amyloid precursor protein ectodomain is up-regulated by tumour necrosis factor-α converting enzyme

Biochemical Journal ◽

10.1042/0264-6021:3570787 ◽

2001 ◽

Vol 357 (3) ◽

pp. 787 ◽

Cited By ~ 48

Author(s):

Barbara E. SLACK ◽

Leona K. MA ◽

Ching Ching SEAH

Keyword(s):

Amyloid Precursor Protein ◽

Tumour Necrosis Factor ◽

Tumour Necrosis ◽

Precursor Protein ◽

Converting Enzyme ◽

Tumour Necrosis Factor Α ◽

Factor Α ◽

Amyloid Precursor Protein Ectodomain ◽

Necrosis Factor

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Muscarinic m1 receptor agonists increase the secretion of the amyloid precursor protein ectodomain

Life Sciences ◽

10.1016/s0024-3205(97)00038-6 ◽

1997 ◽

Vol 60 (13-14) ◽

pp. 985-991 ◽

Cited By ~ 63

Author(s):

Dorothea M. Müller ◽

Klaus Mendla ◽

Steven A. Farber ◽

Roger M. Nitsch

Keyword(s):

Amyloid Precursor Protein ◽

Precursor Protein ◽

Receptor Agonists ◽

M1 Receptor ◽

Amyloid Precursor Protein Ectodomain

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Serotonin 5-HT2a and 5-HT2c Receptors Stimulate Amyloid Precursor Protein Ectodomain Secretion

Journal of Biological Chemistry ◽

10.1074/jbc.271.8.4188 ◽

1996 ◽

Vol 271 (8) ◽

pp. 4188-4194 ◽

Cited By ~ 142

Author(s):

Roger M. Nitsch ◽

Meihua Deng ◽

John H. Growdon ◽

Richard J. Wurtman

Keyword(s):

Amyloid Precursor Protein ◽

Precursor Protein ◽

Amyloid Precursor Protein Ectodomain

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amyloid precursor protein ectodomain
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H-INDEX

Peptides of Presenilin-1 Bind the Amyloid Precursor Protein Ectodomain and Offer a Novel and Specific Therapeutic Approach to Reduce ß-Amyloid in Alzheimer’s Disease

Establishment and characterization of RNA-edited serotonin 2C receptor isoform cell models and alteration of amyloid precursor protein ectodomain secretion in HEK293 APPSwe cells

Faculty Opinions recommendation of The secreted beta-amyloid precursor protein ectodomain APPs alpha is sufficient to rescue the anatomical, behavioral, and electrophysiological abnormalities of APP-deficient mice.

The Secreted -Amyloid Precursor Protein Ectodomain APPs Is Sufficient to Rescue the Anatomical, Behavioral, and Electrophysiological Abnormalities of APP-Deficient Mice

Regulation of the amyloid precursor protein ectodomain shedding by the 5-HT4receptor and Epac

Posttranslational Modifications of Amyloid Precursor Protein: Ectodomain Phosphorylation and Sulfation

Constitutive shedding of the amyloid precursor protein ectodomain is up-regulated by tumour necrosis factor-α converting enzyme

Muscarinic m1 receptor agonists increase the secretion of the amyloid precursor protein ectodomain

Serotonin 5-HT2a and 5-HT2c Receptors Stimulate Amyloid Precursor Protein Ectodomain Secretion

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amyloid precursor protein ectodomainRecently Published Documents

TOTAL DOCUMENTS

H-INDEX

Peptides of Presenilin-1 Bind the Amyloid Precursor Protein Ectodomain and Offer a Novel and Specific Therapeutic Approach to Reduce ß-Amyloid in Alzheimer’s Disease

Establishment and characterization of RNA-edited serotonin 2C receptor isoform cell models and alteration of amyloid precursor protein ectodomain secretion in HEK293 APPSwe cells

Faculty Opinions recommendation of The secreted beta-amyloid precursor protein ectodomain APPs alpha is sufficient to rescue the anatomical, behavioral, and electrophysiological abnormalities of APP-deficient mice.

The Secreted -Amyloid Precursor Protein Ectodomain APPs Is Sufficient to Rescue the Anatomical, Behavioral, and Electrophysiological Abnormalities of APP-Deficient Mice

Regulation of the amyloid precursor protein ectodomain shedding by the 5-HT4receptor and Epac

Posttranslational Modifications of Amyloid Precursor Protein: Ectodomain Phosphorylation and Sulfation

Constitutive shedding of the amyloid precursor protein ectodomain is up-regulated by tumour necrosis factor-α converting enzyme

Muscarinic m1 receptor agonists increase the secretion of the amyloid precursor protein ectodomain

Serotonin 5-HT2a and 5-HT2c Receptors Stimulate Amyloid Precursor Protein Ectodomain Secretion

amyloid precursor protein ectodomain
Recently Published Documents