experimental renal hypertension
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2009 ◽  
Vol 9 (Suppl 1) ◽  
pp. P19
Author(s):  
Bianca Grosz ◽  
Stephanie Krämer ◽  
Tanja Loof ◽  
Hans H Neumayer ◽  
Yingrui Wang-Rosenke ◽  
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Circulation ◽  
1995 ◽  
Vol 92 (1) ◽  
pp. 106-113 ◽  
Author(s):  
Julian Donckier ◽  
Liviu Stoleru ◽  
Wataru Hayashida ◽  
Henri Van Mechelen ◽  
Philippe Selvais ◽  
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Hypertension ◽  
1993 ◽  
Vol 21 (5) ◽  
pp. 624-631 ◽  
Author(s):  
A Upadhya ◽  
M Samuel ◽  
R H Cox ◽  
R J Bagshaw ◽  
S Chacko

1990 ◽  
Vol 4 ◽  
pp. 109
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L.V. Podorolskaya ◽  
G.V. Andreenko ◽  
D.V. Shelkovina

1989 ◽  
Vol 108 (4) ◽  
pp. 1509-1512
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G. V. Andreenko ◽  
O. V. Shelkovina ◽  
L. V. Podorol'skaya

Physiology ◽  
1987 ◽  
Vol 2 (4) ◽  
pp. 139-141 ◽  
Author(s):  
RL Prewitt ◽  
LD Stacy ◽  
O Zenpei

Hypertensive subjects show microvascular changes that include structural decreases in the arteriolar lumen, increases in wall-to-lumen ratio, and decreases in the number of arterioles. In experimental renal hypertension, arteriolar vasoconstriction is first increased and then diminishes as structural elevations in vascular resistance develop. The small arteries may contribute significantly to the elevated vascular resistance. Whether these changes have a primary or secondary role in hypertension is still open to question.


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