molecular imprinting technique
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2022 ◽  
Vol 148 ◽  
pp. 107786
Author(s):  
Saeed Azad ◽  
Mahnaz Khosravi ◽  
Alireza Nikzad ◽  
Satyendra Kumar Mishra

2021 ◽  
pp. 462579
Author(s):  
Chao Huang ◽  
Hongwei Wang ◽  
Shujuan Ma ◽  
Chunmiao Bo ◽  
Junjie Ou ◽  
...  

2021 ◽  
Vol 45 (39) ◽  
pp. 18296-18306
Author(s):  
Deniz Türkmen ◽  
Monireh Bakhshpour ◽  
Ilgım Göktürk ◽  
Süleyman Aşır ◽  
Fatma Yılmaz ◽  
...  

In this study, selective and sensitive detection of neurotransmitter dopamine from both aqueous solution and biological samples was performed by surface plasmon resonance sensor based on molecular imprinting technique.


RSC Advances ◽  
2020 ◽  
Vol 10 (19) ◽  
pp. 10980-10988 ◽  
Author(s):  
Shuang Han ◽  
Fu Teng ◽  
Yuan Wang ◽  
Liqiang Su ◽  
Qiuxue Leng ◽  
...  

In the research we designed a CEA-molecularly imprinted polymers using molecular imprinting technique with CEA tumor marker as template, boronic acid functionalized MGO as substrate for dual targeted delivery of drug to tumor cells.


AMB Express ◽  
2019 ◽  
Vol 9 (1) ◽  
Author(s):  
Junhyok Ryang ◽  
Yan Yan ◽  
Yangyang Song ◽  
Fang Liu ◽  
Tzi Bun Ng

AbstractIn this study, a single component paclitaxel was obtained from fermentation broth by molecular imprinting technique, and its antiviral, antitumor and immunomodulatory activities were studied. The results showed that paclitaxel had a good inhibitory activity on human breast cancer MCF-7 cells and showed a concentration- dependent relationship with an IC50 of about 15 μg/mL in the sulforhodamine B assay. At the same time, paclitaxel exerted a weak inhibitory activity on cervical cancer Hela cells. In addition, paclitaxel not only inhibited the invasion of HIV-1 pseudovirus into cells, but also exhibited inhibitory activity to a certain extent after viral invasion of the cells. At a paclitaxel concentration of 20 μg/mL, the inhibition of HIV-1 pseudovirus reached about 66%. The inhibition of HIV-1 protease activity was concentration-dependent. At a concentration of 20 μg/mL, the inhibitory effect of paclitaxel on HIV-1 protease was similar to that of the positive control pepstatin A, being 15.8%. The HIV-1 integrase inhibiting activity of paclitaxel was relatively weak. Paclitaxel significantly up-regulated the expression of interleukin-6.


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