Heart Rate Variability Reflects Similar Cardiac Autonomic Function in Explosive and Aerobically Trained Athletes

Autonomic cardiac function can be indirectly detected non-invasively by measuring the variation in microtiming of heart beats by a method known as heart rate variability (HRV). Aerobic training for sport is associated with reduced risk for some factors associated with cardiovascular diseases (CVD), but effects on autonomic function in different athlete types are less known.To compare cardiac autonomic modulation using a standard protocol and established CVD risk factors in highly trained intercollegiate athletes competing in aerobic, explosive, and cross-trained sports. A total of 176 college athletes were categorized in distinct sports as explosive (EA), aerobic (AA), or cross-trained (mixed) athletes. Eight different HRV measures obtained at rest were compared across training type and five health factors: systolic (SBP), diastolic blood pressure (DBP), body weight (BW), sex, and race. All athletic types shared favorable HRV measures that correlated with low CVD risk factors and indicated normal sympathovagal balance. A significant correlation was reported between DBP and pNN50 (% RR intervals > 50 ms) (β = −0.214, p = 0.011) and between BW and low-frequency (LF) power (β = 0.205, p = 0.006). Caucasian and African American athletes differed significantly (p < 0.05) with respect to four HRV variables: pNN50, HF power, LF power, and LF/HF ratios. Explosive, aerobic and mixed athletes had similar cardiovascular and autonomic HRV results in all eight HRV parameters measured. All athletes reported LF and pNN50 values that were significantly correlated with two CVD risk factors: DBP and BW. Compared with Caucasian teammates, African American athletes demonstrated lower LF/HF and higher pNN50, indicating an even more favorable resting sympathovagal activity and healthy CV function.

Rebooting the Politics of the Sports Melodrama: Creed vs Rocky

This chapter describes Creed as a sequel-reboot which functions as a politically ambivalent, but textually reverent, reboot of the Rocky franchise (1976–90). It asserts that Creed is a film that at one and the same time celebrates the franchise’s deployment of the tropes of the boxing picture and male melodrama, while also updating the racial and sexual politics of the series. Developing ideas on the way film reboots mediate the tension between familiarity and novelty, the chapter demonstrates how Creed rewrites aspects of the original Rocky films so as to create a new political narrative, one that explicitly challenges stereotypes of African-American athletes.

Royce White, DeMar DeRozan and Media Framing of Mental Health in the NBA

This study analyzed media framing of two players from the National Basketball Association (NBA), Royce White and DeMar DeRozan, who both publicly addressed their mental health concerns within the last decade. Due to societal stigmas, such a public conversation was once a rarity in the NBA, making their discussion of mental health a worthy area of examination. A media framing analysis of 58 press articles for White and 42 articles for DeRozan revealed six frames for each athlete used to discuss their situation. Despite both being African American athletes playing the same sport, they were covered differently. Analysis discovered that frames for White, a first-year player who wanted changes in the system of how the NBA handled mental health, most commonly focused on him and his specific situation, while frames for DeRozan, an NBA All-Star who positioned himself solely as an advocate, most frequently discussed shedding the stigma of mental health and the increased awareness surrounding the topic. Additionally, while there were frames critical of White, there were no such frames for DeRozan. Still, for both players, this type of coverage humanizes the experience of mental health and leads to important insights about changes in masculinity among athletes.

Tau pathology in the medial temporal lobe of athletes with chronic traumatic encephalopathy: a chronic effects of neurotrauma consortium study

Chronic traumatic encephalopathy (CTE) is a progressive neurodegenerative condition associated with repetitive traumatic brain injury (rTBI) seen in contact-sport athletes and military personnel. The medial temporal lobe (MTL; i.e., hippocampus, subiculum, and entorhinal and perirhinal cortices) memory circuit displays tau lesions during the pathological progression of CTE. We examined MTL tissue obtained from 40 male Caucasian and African American athletes who received a postmortem CTE neuropathological diagnosis defined as stage II, III, or IV. Sections were immunolabeled using an early (AT8) or a late (TauC3) marker for pathological tau and for amyloid beta (Aβ) species (6E10, Aβ1–42 and thioflavin S). Stereological analysis revealed that stage III had significantly less AT8-positive neurons and dystrophic neurites than stage IV in all MTL regions except hippocampal subfield CA3, whereas significantly more AT8-positive neurons, dystrophic neurites, and neurite clusters were found in the perirhinal cortex, entorhinal cortex, hippocampal CA1, and subiculum of CTE stage III compared with stage II. TauC3-positive pathology was significantly higher in the perirhinal and subicular cortex of stage IV compared to stage III and the perirhinal cortex of stage III compared to stage II. AT8-positive neurite clusters were observed in stages III and IV, but virtually absent in stage II. When observed, Aβ pathology appeared as amyloid precursor protein (APP)/Aβ (6E10)-positive diffuse plaques independent of region. Thioflavine S labeling, did not reveal evidence for fibril or neuritic pathology associated with plaques, confirming a diffuse, non-cored plaque phenotype in CTE. Total number of AT8-positive profiles correlated with age at death, age at symptom onset, and time from retirement to death. There was no association between AT8-positive tau pathology and age sport began, years played, or retirement age, and no difference between CTE stage and the highest level of sport played. In summary, our findings demonstrate different tau profiles in the MTL across CTE stages, proffering CA3 tau pathology and MTL dystrophic neurite clusters as possible markers for the transition between early (II) and late (III/IV) stages, while highlighting CTE as a progressive noncommunicative tauopathy.

It’s All We’ve Got

Increased media coverage of college and professional college shaped beliefs about the benefits and risks of youth football. The importance attributed to high school football in schools and communities contributed to the expansion of football at the little league level. Football among elementary and middle school children increasingly served as a feeder system for the high school level of play. In addition, the appeal of future access to social and financial resources, including the hope of landing a college football scholarship and a potential professional career, became increasingly prominent in the latter half the twentieth century. The possibility of accessing higher education through football influenced how parents and players weighed the risks and benefits of the sport at the high school level and younger. The ways football improved perceived access to higher social standing and higher education contributed in part to the changing racial demographics of tackle football, particularly with the increasing involvement of African American athletes. Meanwhile, sportscasters’ glorification of “big hits” fostered celebration of football’s dangers even as sports organizers claimed both educational and physical benefits for the youth sport.