plasma acth level
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2004 ◽  
Vol 80 (4) ◽  
pp. 481-488 ◽  
Author(s):  
Toshihiro Nakashima ◽  
Mariko Akamatsu ◽  
Akikazu Hatanaka ◽  
Toshikazu Kiyohara

1996 ◽  
Vol 19 (9) ◽  
pp. 1244-1246 ◽  
Author(s):  
Kenji YAMADA ◽  
Takeharu MIURA ◽  
Yoshihiro MIMAKI ◽  
Yutaka SASHIDA

1983 ◽  
Vol 95 (3) ◽  
pp. 351-353 ◽  
Author(s):  
S. B. Seredenin ◽  
Yu. A. Blednov ◽  
B. A. Badyshtov ◽  
N. M. Shevchenko

1981 ◽  
Vol 240 (4) ◽  
pp. E441-E446 ◽  
Author(s):  
G. B. Makara ◽  
E. Stark ◽  
M. Karteszi ◽  
M. Palkovits ◽  
G. Rappay

The effects of destroying the paraventricular nucleus (PVN) of the rat hypothalamus on pituitary-adrenal function were studied. Four days after PVN lesions were placed with a rotating knife, the basal plasma corticosterone level was normal, but the corticosterone response to electrical stimulation of the medial basal hypothalamus, surgical trauma, and ether-venesection stress was significantly inhibited. Four and 8 days after PVN lesioning and adrenalectomy, the basal plasma ACTH level was lower, and the rise of plasma ACTH level elicited by a 3-min ether inhalation was significantly smaller than in the adrenalectomized controls. Corticotropin-releasing factor (CRF) activity in the stalk-median eminence extracts from PVN-lesioned rats was significantly less than in the control extracts. The weight of the adrenals was decreased by both 2 and 4 wk after PVN destruction, and 2 wk after hemiadrenalectomy, the compensatory adrenal hypertrophy was inhibited. The plasma corticosterone response to ether-venesection stress was inhibited only temporarily because it returned to normal by the end of the 4th postoperative week. The results are consistent with the hypothesis that a substantial portion of CRF-containing fibers in the stalk-median eminence region either originate from or run though the PVN or its immediate vicinity.


1980 ◽  
Vol 93 (2) ◽  
pp. 129-133 ◽  
Author(s):  
M. Kárteszi ◽  
G. B. Makara ◽  
E. Stark

Abstract. The effect of ether stress on the release of immunoreactive ACTH was studied in rats with an antero-lateral cut around the medial basal hypothalamus. Ether failed to raise the plasma ACTH level of rats in which an antero-lateral hypothalamic cut and adrenalectomy had been performed 7 to 8 days previously. Plasma ACTH was also unchanged in rats exposed to ether 2 h after an antero-lateral cut. These data suggest that intact neural pathways entering the medial basal hypothalamus from the antero-lateral direction are necessary for the ACTH releasing action of ether stress.


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