The increase in the yield of commercial timber from round timber with internal rot

The article presents information on methods bucking wood core rot. Offering flaw detector used as X-ray unit upgraded to sight through the X-ray transmission type, and it is paired with 3D scanner. The article presents the scheme bucking of stems with heart rot for different companies depending on the volume and type of products. Used peeled or not burking whips, de-pending on the technological production chain.

First Report of Internal Fruit Rot of Sweet Pepper in Korea Caused by Fusarium lactis

In June 2010, an internal fruit rot of sweet pepper (Capsicum annuum L.) fruit was found in a commercial greenhouse in Ilsan City, Korea. Disease incidence reached approximately 5% of 30 tons of harvested peppers. Affected fruits commonly did not show external symptoms. However, when the fruit was cut open, an internal rot and pinkish gray mycelium were observed on the seeds and the inner surface of fruit. Discolored soft patches, browning, and necrosis were observed on the outer surface of some fruits. Fragments (5 × 5 mm2) of the affected tissues were surface sterilized with 1% NaOCl for 30 s and then rinsed twice in sterile distilled water. The pieces were placed on water agar and incubated at 25°C for 4 to 6 days. Twenty-nine Fusarium isolates were obtained from 12 diseased fruits and maintained on synthetic low nutrient agar (SNA) at 10°C. The isolates were cultured on carnation leaf agar (CLA) and SNA at 23°C with 12 h of near-ultraviolet light per day for 14 days. Microconidia were abundant, borne in short, zig-zag chains or false heads, obovoid or clavate with a flattened base, and measured 4.3 to 7.1 × 2.2 to 3.3 μm. Macroconidia were sparse, thin walled, slender, straight to slightly curved, and measured 32 to 48 × 2.8 to 3.9 μm. Sporodochia were rare on CLA and chlamydospores were absent. The translation elongation factor 1-alpha (EF-1α) gene was amplified from four isolates (SPF01, SPF09, SPF16, and SPF22) by PCR assay using ef1 and ef2 primers (2), and the 700-bp amplification products were sequenced. The nucleotide sequences were deposited in GenBank (Accession Nos. JF411956 to JF411959). BLAST analysis showed 98% homology with the EF-1α sequence of Fusarium lactis NRRL25200 (GenBank Accession No. AF160272). All isolates were identified as F. lactis based on morphological and molecular characteristics (1). Pathogenicity tests of the four isolates were conducted by inoculating flowers on plants of the orange pepper cv. Orange Glory (3). A spore suspension was prepared by flooding 5-day-old cultures on potato dextrose agar with sterile distilled water. When the plants started to flower, each flower was inoculated by placing 20 μl of spore suspension (1 × 106 conidia/ml) on each flower. Four isolates of F. lactis were each inoculated onto three flowers on each of seven plants. Flowers from the same number of plants inoculated with sterile distilled water were used as the control treatment. Inoculated plants were kept in a greenhouse at 25°C by day and 20°C by night. Sixty days after inoculation, mature fruits were harvested and cut open to check for internal rot. Approximately 70% of inoculated fruits showed internal rot and pinkish gray mycelial growth on the inner surface of the fruits. No symptoms were observed on the control fruits. Fungal cultures resembling F. lactis were reisolated from inoculated fruits for all four isolates, fulfilling Koch's postulates. F. lactis has been reported on sweet pepper in the Netherlands and Canada (3). To our knowledge, this is the first report of internal fruit rot of sweet pepper caused by F. lactis in Korea. Although disease severity was low in this greenhouse, the economic impact on sweet pepper could be significant because the disease can reduce the quality, quantity, and market value of pepper fruits. References: (1) H. I. Nirenberg and K. O'Donnell. Mycologia 90:434, 1998. (2) K. O'Donnell et al. Proc. Natl. Acad. Sci. USA 95:2044, 1998. (3) J. Yang et al. Can. J. Plant Pathol. 31:47, 2009.

Influence of day/night humidity and cation ratios and concentration in the nutrient solution on incidence of Didymella bryoniae in glasshouse cucumbers.

The incidence of the disease on plants and fruits decreased with a lower 24 h av. RH in the range 80-87%. An increase in the electrical conductivity (EC) level of the nutrient solution from 2.5-5.0 dS/m (25 degrees C) decreased the number of diseased leaf axils at a Ca concn of 1.5 mmol/litre by 41%, but had no effect at a Ca concn of 4.5 mmol/litre. An increase in Ca concn from 1.5-4.5 mmol/litre decreased the number of diseased leaf axils at an EC level of 2.5 dS/m by 26%, but had no effect at an EC level of 5.0 dS/m. Changes in EC and Ca concn had no effect on the number of fruits with internal rot. It is concluded that to optimize disease control, high RH, low EC level and low Ca concn in the nutrient solution should be avoided. (Abstract retrieved from CAB Abstracts by CABI’s permission)

Verticillium theobromae. [Descriptions of Fungi and Bacteria].

A description is provided for Verticillium theobromae. Information is included on the disease caused by the organism, its transmission, geographical distribution, and hosts. HOSTS: On Musa; less frequently on Bambusa, Heliconia bihai and H. brasiliensis. DISEASE: 'Cigar end' of banana. On fruit in situ, necrosis begins at the pistillate end, the skin becoming folded and shrunk; the dead floral parts tend to become persistent. The powdery, greyish conidia form on the shrivelled black end of the fruit giving rise to the appearance from which the disease gets its name (11: 312). The fungus is also one of the members of a pathogenic complex which cause a rot of the crowns in the shipment of boxed bananas (42: 622; 45, 160, 2482). The internal rot on the fruit in the field is a dry one in contrast to the wet rot of Trachysphaera fructigena (CMI Descript. 229). Verticillium theobromae may also be associated with black pitting and spotting of the fruit. GEOGRAPHICAL DISTRIBUTION: Widespread in the tropics (CMI Map 146, ed. 2, 1966). TRANSMISSION: No specific studies reported, presumably air-dispersed. The fungus is a common inhabitant on plant debris in banana fields.