behavioral physiology
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Aging ◽  
2020 ◽  
Vol 12 (11) ◽  
pp. 10556-10577
Author(s):  
Wei-Hsiang Hsu ◽  
Young-Ji Shiao ◽  
Yen-Ming Chao ◽  
Yi-Jeng Huang ◽  
Yun-Lian Lin

2019 ◽  
Vol 20 (17) ◽  
pp. 4252 ◽  
Author(s):  
Solomon Zewdu Altaye ◽  
Lifeng Meng ◽  
Yao Lu ◽  
Jianke Li

Advances in instrumentation and computational analysis in proteomics have opened new doors for honeybee biological research at the molecular and biochemical levels. Proteomics has greatly expanded the understanding of honeybee biology since its introduction in 2005, through which key signaling pathways and proteins that drive honeybee development and behavioral physiology have been identified. This is critical for downstream mechanistic investigation by knocking a gene down/out or overexpressing it and being able to attribute a specific phenotype/biochemical change to that gene. Here, we review how emerging proteome research has contributed to the new understanding of honeybee biology. A systematic and comprehensive analysis of global scientific progress in honeybee proteome research is essential for a better understanding of research topics and trends, and is potentially useful for future research directions.


2019 ◽  
Vol 119 ◽  
pp. 174-183 ◽  
Author(s):  
Anete Pedro Lourenço ◽  
Juliana Ramos Martins ◽  
Fernanda Andrade Silva Torres ◽  
Aline Mackert ◽  
Luiz Roberto Aguiar ◽  
...  

Oncotarget ◽  
2017 ◽  
Vol 8 (21) ◽  
pp. 34022-34023 ◽  
Author(s):  
Maurizio Casarrubea ◽  
Andrea Santangelo ◽  
Giuseppe Crescimanno

2015 ◽  
Vol 8 (1) ◽  
Author(s):  
Mootaek Roh ◽  
Thomas J. McHugh ◽  
Kyungmin Lee

2012 ◽  
Vol 107 (7) ◽  
pp. 2008-2019 ◽  
Author(s):  
Abigail Kalmbach ◽  
Tristan Hedrick ◽  
Jack Waters

Acetylcholine profoundly affects neocortical function, being involved in arousal, attention, learning, memory, sensory and motor function, and plasticity. The majority of cholinergic afferents to neocortex are from neurons in nucleus basalis. Nucleus basalis also contains projecting neurons that release other transmitters, including GABA and possibly glutamate. Hence, electrical stimulation of nucleus basalis evokes the release of a mixture of neurotransmitters in neocortex, and this lack of selectivity has impeded research on cholinergic signaling in neocortex. We describe a method for the selective stimulation of cholinergic axons in neocortex. We used the Cre-lox system and a viral vector to express the light-activated protein channelrhodopsin-2 in cholinergic neurons in nucleus basalis and their axons in neocortex. Labeled neurons depolarized on illumination with blue light but were otherwise unchanged. In anesthetized mice, illumination of neocortex desynchronized the local field potential, indicating that light evoked release of ACh. This novel technique will enable many new studies of the cellular, network, and behavioral physiology of ACh in neocortex.


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