Development of a PANI/Fe(NO3)2 nanomaterial for reactive orange 16 (RO16) dye removal

Polyaniline–iron(ii) nitrate was prepared by the polymerization of aniline hydrochloride with Fe(NO3)2.

Preparation and Characterization of poly-aniline hydrochloride (PANI-HCL) Nano-composites by Radiation

During this search, the polymer poly-aniline hydrochloride (PANI-HCL) was prepared with different weights before and after irradiation in the form of films using the casting method. The films were recorded with the cobalt element(CO60), which sends a gama (1.2) Mev for a period of (72) hours. The structural properties of the (FT-IR) were studied. The results showed that the active groups of the recorded polymer were consistent with the chemical composition and topographic study of the prepared film surfaces. The images showed that the recorded polymer is homogeneous and its color changes after irradiation. The electrical properties included the measurement of the hall effect and the continuous electrical conductivity and the behavior of the current and voltages where the results showed that the Hall coefficient of the polymer mixture recorded before and after the irradiation are carriers of the (n-type) and through the measurement of current- voltages show results that the polymer recorded conduct the behavior of ohmic before irradiation and walk Conduction of a semiconductor after irradiation. In addition, the results of electrical conductivity were shown to be less after irradiation.

Exploring the Critical Factors Limiting Polyaniline Biocompatibility

Today, the application of polyaniline in biomedicine is widely discussed. However, information about impurities released from polyaniline and about the cytotoxicity of its precursors aniline, aniline hydrochloride, and ammonium persulfate are scarce. Therefore, cytotoxicity thresholds for the individual precursors and their combinations were determined (MTT assay) and the type of cell death caused by exposition to the precursors was identified using flow-cytometry. Tests on fibroblasts revealed higher cytotoxicity of ammonium persulfate than aniline hydrochloride. Thanks to the synergic effect, both monomers in combination enhanced their cytotoxicities compared with individual substances. Thereafter, cytotoxicity of polyaniline doped with different acids (sulfuric, nitric, phosphoric, hydrochloric, and methanesulfonic) was determined and correlated with impurities present in respective sample (HPLC). The lowest cytotoxicity showed polyaniline doped with phosphoric acid (followed by sulfuric, methanesulfonic, and nitric acid). Cytotoxicity of polyaniline was mainly attributed to the presence of residual ammonium persulfate and low-molecular-weight polar substances. This is crucial information with respect to the purification of polyaniline and production of its cytocompatible form.

Ameliorative Effect of Chronic Supplementation of Protocatechuic Acid Alone and in Combination with Ascorbic Acid in Aniline Hydrochloride Induced Spleen Toxicity in Rats

Background. Present study was designed to evaluate the protective effects of protocatechuic acid alone and in combination with ascorbic acid in aniline hydrochloride induced spleen toxicity in rats.Materials and Methods. Male Wistar rats of either sex (200–250 g) were used and divided into different groups. Spleen toxicity was induced by aniline hydrochloride (100 ppm) in drinking water for a period of 28 days. Treatment group received protocatechuic acid (40 mg/kg/day, p.o.), ascorbic acid (40 mg/kg/day, p.o.), and combination of protocatechuic acid (20 mg/kg/day, p.o.) and ascorbic acid (20 mg/kg/day, p.o.) followed by aniline hydrochloride. At the end of treatment period serum and tissue parameters were evaluated.Result. Rats supplemented with aniline hydrochloride showed a significant alteration in body weight, spleen weight, feed consumption, water intake, hematological parameters (haemoglobin content, red blood cells, white blood cells, and total iron content), tissue parameters (lipid peroxidation, reduced glutathione, and nitric oxide content), and membrane bound phosphatase (ATPase) compared to control group. Histopathology of aniline hydrochloride induced spleen showed significant damage compared to control rats. Treatment with protocatechuic acid along with ascorbic acid showed better protection as compared to protocatechuic acid or ascorbic acid alone in aniline hydrochloride induced spleen toxicity.Conclusion. Treatment with protocatechuic acid and ascorbic acid in combination showed significant protection in aniline hydrochloride induced splenic toxicity in rats.