scholarly journals Muscle Mechanics and Electromyography

Author(s):  
Sarmad Shams ◽  
Muhammad Asif ◽  
Samreen Hussain
Keyword(s):  
1967 ◽  
Vol 1 (3) ◽  
pp. 202-202
Author(s):  
Mouazza M Jarmakani ◽  
Madison S Spach ◽  
Sam B Edwards ◽  
Ramon V Canent ◽  
M Paul Capp ◽  
...  

Author(s):  
D. C. S. White
Keyword(s):  

1996 ◽  
Vol 81 (3) ◽  
pp. 1323-1330 ◽  
Author(s):  
E. Saugen ◽  
N. K. Vollestad

The effect of repetitive isometric knee extensions on the energy cost of contraction was examined. The rate of temperature rise (dT/dt) was determined in test contractions at 30 and 50% of maximal voluntary contraction (MVC) force before and during 30% MVC repetitive isometric exercise (RIE) to exhaustion and regularly in a 30-min postexercise recovery period (n = 9). Pulmonary O2 uptake and muscle temperature (Tmus) were determined at regular intervals. During the 30% MVC test contractions, dT/dt was 5.6 +/- 0.6 mK/s in unfatigued muscle, increasing linearly by 68% during exercise. In the 50% MVC test contractions, dT/dt rose by 84% from 9.8 +/- 1.1 mK/s. dT/dt determined during test contractions at both force levels did not decrease significantly throughout the 30-min postexercise recovery period. The rise in dT/dt was paralleled by 76% increased in O2 uptake. In contrast, Tmus rose initially and then leveled off. The present data indicate that RIE induced a gradual rise in the rate of energy turnover associated with isometric force production. Neither increased Tmus nor recruitment of less economic type II fibers can fully explain the increased energy cost. We suggest that energetic changes may occur at the cellular level and argue that this may be associated with the changes in muscle mechanics occurring during fatigue from submaximal voluntary RIE.


2019 ◽  
Author(s):  
Andrea Zonnino ◽  
Daniel R. Smith ◽  
Peyton L. Delgorio ◽  
Curtis L. Johnson ◽  
Fabrizio Sergi

AbstractNon-invasive in-vivo measurement of individual muscle force is limited by the infeasibility of placing force sensing elements in series with the musculo-tendon structures. At the same time, estimating muscle forces using EMG measurements is prone to inaccuracies, as EMG is not always measurable for the complete set of muscles acting around the joints of interest. While new methods based on shear wave elastography have been recently proposed to directly characterize muscle mechanics, they can only be used to measure muscle forces in a limited set of superficial muscles. As such, they are not suitable to study the neuromuscular control of movements that require coordinated action of multiple muscles.In this work, we present multi-muscle magnetic resonance elastography (MM-MRE), a new technique capable of quantifying individual muscle force from the complete set of muscles in the forearm, thus enabling the study of the neuromuscular control of wrist movements. MM-MRE integrates measurements of joint torque provided by an MRI-compatible instrumented handle with muscle-specific measurements of shear wave speed obtained via MRE to quantify individual muscle force using model-based estimator.A single-subject pilot experiment demonstrates the possibility of obtaining measurements from individual muscles and establishes that MM-MRE has sufficient sensitivity to detect changes in muscle mechanics following the application of isometric joint torque with self-selected intensity.


2010 ◽  
Vol 24 (S1) ◽  
Author(s):  
C Subah Packer ◽  
Alexa M Boice ◽  
Leeanna M Meulen ◽  
Kathy Coy ◽  
Richard G Peterson

eLife ◽  
2018 ◽  
Vol 7 ◽  
Author(s):  
William A Kronert ◽  
Kaylyn M Bell ◽  
Meera C Viswanathan ◽  
Girish C Melkani ◽  
Adriana S Trujillo ◽  
...  

K146N is a dominant mutation in human β-cardiac myosin heavy chain, which causes hypertrophic cardiomyopathy. We examined how Drosophila muscle responds to this mutation and integratively analyzed the biochemical, physiological and mechanical foundations of the disease. ATPase assays, actin motility, and indirect flight muscle mechanics suggest at least two rate constants of the cross-bridge cycle are altered by the mutation: increased myosin attachment to actin and decreased detachment, yielding prolonged binding. This increases isometric force generation, but also resistive force and work absorption during cyclical contractions, resulting in decreased work, power output, flight ability and degeneration of flight muscle sarcomere morphology. Consistent with prolonged cross-bridge binding serving as the mechanistic basis of the disease and with human phenotypes, 146N/+ hearts are hypercontractile with increased tension generation periods, decreased diastolic/systolic diameters and myofibrillar disarray. This suggests that screening mutated Drosophila hearts could rapidly identify hypertrophic cardiomyopathy alleles and treatments.


Author(s):  
Justin Fernandez ◽  
Kumar Mithraratne ◽  
Massoud Alipour ◽  
Geoffrey Handsfield ◽  
Thor Besier ◽  
...  

1989 ◽  
Vol 142 (1) ◽  
pp. 17-29 ◽  
Author(s):  
C. J. PENNYCUICK ◽  
M. R. FULLER ◽  
LYNNE McALLISTER

Two Harris' hawks were trained to fly along horizontal and climbing flight paths, while carrying loads of various masses, to provide data for estimating available muscle power during short flights. The body mass of both hawks was about 920 g, and they were able to carry loads up to 630 g in horizontal flight. The rate of climb decreased with increasing all-up mass, as also did the climbing power (product of weight and rate of climb). Various assumptions about the aerodynamic power in low-speed climbs led to estimates of the maximum power output of the flight muscles ranging from 41 to 46 W. This, in turn, would imply a stress during shortening of around 210 kPa. The effects of a radio package on a bird that is raising young should be considered in relation to the food load that the forager can normally carry, rather than in relation to its body mass.


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