scholarly journals Effects of nicarbazin and hot temperature on evaporatative water loss, acid-base balance, body temperature and carbin dioxide exhalation in adult roosters

1994 ◽  
Vol 7 (1) ◽  
pp. 97-101
Author(s):  
B. D. Lee ◽  
S. K. Lee ◽  
W. J. Hyun
1956 ◽  
Vol 186 (1) ◽  
pp. 31-34 ◽  
Author(s):  
D. Robert Axelrod ◽  
David E. Bass

Dogs were cooled in an ice-water bath, and plasma electrolytes were measured at heart temperatures of 38°C, 28°C and 25°C. A ‘cold acidosis’ occurred during hypothermia that is attributable largely to temperature-influenced physico-chemical factors related to the buffer systems. A slight respiratory depression is of greater importance in decreasing plasma ph at lower body temperature than at normal body temperature.


1982 ◽  
Vol 52 (1) ◽  
pp. 114-118 ◽  
Author(s):  
D. G. Davies ◽  
J. L. Thomas ◽  
E. N. Smith

Pulmonary ventilation and arterial blood acid-base balance were measured in six unanesthetized alligators, Alligator mississipiensis, at 15, 25, and 35 degree C. The animals exhibited pronounced ventilatory responses to hypercapnia at all temperatures studied. Arterial PCO2 increased and pH decreased with increases in body temperature during both normocapnia and hypercapnia. The fractional dissociation of imidazole (alpha Pr) remained constant with changes in body temperature during normocapnia, but increased with temperature during hypercapnia. Ventilatory sensitivity, defined as delta (VE/VO2/delta (alpha Pr), was independent of body temperature. We conclude that the control of breathing in the alligator is a physiological defense of alpha Pr and that ventilatory responses occur following nontemperature-induced changes in blood acid-base balance, which tend to return alpha Pr to a normal value.


1970 ◽  
Vol 218 (2) ◽  
pp. 600-606 ◽  
Author(s):  
BJ Howell ◽  
FW Baumgardner ◽  
K Bondi ◽  
H Rahn

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