Acid-Base Balance and Body Temperature Regulation of Heat Stressed Psammomys obesus (Gerbillinae): The Effect of Bicarbonate Loss via Saliva Spreading

1983 ◽  
Vol 56 (3) ◽  
pp. 389-396 ◽  
Author(s):  
J. Marder ◽  
I. Porat ◽  
P. Raber ◽  
J. H. Adler
1956 ◽  
Vol 186 (1) ◽  
pp. 31-34 ◽  
Author(s):  
D. Robert Axelrod ◽  
David E. Bass

Dogs were cooled in an ice-water bath, and plasma electrolytes were measured at heart temperatures of 38°C, 28°C and 25°C. A ‘cold acidosis’ occurred during hypothermia that is attributable largely to temperature-influenced physico-chemical factors related to the buffer systems. A slight respiratory depression is of greater importance in decreasing plasma ph at lower body temperature than at normal body temperature.


1982 ◽  
Vol 52 (1) ◽  
pp. 114-118 ◽  
Author(s):  
D. G. Davies ◽  
J. L. Thomas ◽  
E. N. Smith

Pulmonary ventilation and arterial blood acid-base balance were measured in six unanesthetized alligators, Alligator mississipiensis, at 15, 25, and 35 degree C. The animals exhibited pronounced ventilatory responses to hypercapnia at all temperatures studied. Arterial PCO2 increased and pH decreased with increases in body temperature during both normocapnia and hypercapnia. The fractional dissociation of imidazole (alpha Pr) remained constant with changes in body temperature during normocapnia, but increased with temperature during hypercapnia. Ventilatory sensitivity, defined as delta (VE/VO2/delta (alpha Pr), was independent of body temperature. We conclude that the control of breathing in the alligator is a physiological defense of alpha Pr and that ventilatory responses occur following nontemperature-induced changes in blood acid-base balance, which tend to return alpha Pr to a normal value.


1970 ◽  
Vol 218 (2) ◽  
pp. 600-606 ◽  
Author(s):  
BJ Howell ◽  
FW Baumgardner ◽  
K Bondi ◽  
H Rahn

2014 ◽  
Vol 84 (3-4) ◽  
pp. 0206-0217 ◽  
Author(s):  
Seyedeh-Elaheh Shariati-Bafghi ◽  
Elaheh Nosrat-Mirshekarlou ◽  
Mohsen Karamati ◽  
Bahram Rashidkhani

Findings of studies on the link between dietary acid-base balance and bone mass are relatively mixed. We examined the association between dietary acid-base balance and bone mineral density (BMD) in a sample of Iranian women, hypothesizing that a higher dietary acidity would be inversely associated with BMD, even when dietary calcium intake is adequate. In this cross-sectional study, lumbar spine and femoral neck BMDs of 151 postmenopausal women aged 50 - 85 years were measured using dual-energy x-ray absorptiometry. Dietary intakes were assessed using a validated food frequency questionnaire. Renal net acid excretion (RNAE), an estimate of acid-base balance, was then calculated indirectly from the diet using the formulae of Remer (based on dietary intakes of protein, phosphorus, potassium, and magnesium; RNAERemer) and Frassetto (based on dietary intakes of protein and potassium; RNAEFrassetto), and was energy adjusted by the residual method. After adjusting for potential confounders, multivariable adjusted means of the lumbar spine BMD of women in the highest tertiles of RNAERemer and RNAEFrassetto were significantly lower than those in the lowest tertiles (for RNAERemer: mean difference -0.084 g/cm2; P=0.007 and for RNAEFrassetto: mean difference - 0.088 g/cm2; P=0.004). Similar results were observed in a subgroup analysis of subjects with dietary calcium intake of >800 mg/day. In conclusion, a higher RNAE (i. e. more dietary acidity), which is associated with greater intake of acid-generating foods and lower intake of alkali-generating foods, may be involved in deteriorating the bone health of postmenopausal Iranian women, even in the context of adequate dietary calcium intake.


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