scholarly journals The Predictive Value Of Gamma-Glutamyl Transferase In Acute Myocardial Infarction And Ischemic Stroke

2011 ◽  
Vol 21 (2) ◽  
pp. 49-53
Author(s):  
Bahar Emen ◽  
Hüseyin Can ◽  
Adife Çetintürk Üstündağ ◽  
Mert Özbakkaloğlu ◽  
Yusuf Adnan Güçlü
Keyword(s):  
Myocardial Infarction ◽  
Acute Myocardial Infarction ◽  
Ischemic Stroke ◽  
Predictive Value ◽  
Gamma Glutamyl Transferase ◽  
Gamma Glutamyl ◽  
Glutamyl Transferase

scholarly journals Serum gamma-glutamyl transferase is a predictor of mortality in patients with acute myocardial infarction

Medicine ◽  
2018 ◽  
Vol 97 (29) ◽  
pp. e11393 ◽  
Author(s):  
Jae Gyung Kim ◽  
Kiyuk Chang ◽  
Eun Ho Choo ◽  
Jong-Min Lee ◽  
Ki-Bae Seung
Keyword(s):  
Myocardial Infarction ◽  
Acute Myocardial Infarction ◽  
Gamma Glutamyl Transferase ◽  
Gamma Glutamyl ◽  
Glutamyl Transferase

scholarly journals Increased concentration of serum gamma-glutamyl transferase in ischemic stroke patients

2021 ◽  
Vol 7 (2) ◽  
pp. 71
Author(s):  
Pinki Tak ◽  
TejaRam Kalirawna ◽  
Jitendra Rohilla ◽  
ShyamSunder Bairwa ◽  
SunilKumar Gothwal ◽  
...  
Keyword(s):  
Ischemic Stroke ◽  
Gamma Glutamyl Transferase ◽  
Stroke Patients ◽  
Gamma Glutamyl ◽  
Glutamyl Transferase

scholarly journals Gamma-Glutamyl Transferase Levels in Patients with Acute Ischemic Stroke

2014 ◽  
Vol 2014 ◽  
pp. 1-4 ◽  
Author(s):  
Nurbanu Gurbuzer ◽  
Eren Gozke ◽  
Zeliha Ayhan Basturk
Keyword(s):  
Ischemic Stroke ◽  
Acute Ischemic Stroke ◽  
Gamma Glutamyl Transferase ◽  
Gamma Glutamyl ◽  
Cerebrovascular Risk ◽  
The Mean ◽  
And Gender ◽  
Glutamyl Transferase ◽  
Relationship Of ◽  
The Relationship

Objective. The aim of this study was to investigate the relationship between gamma-glutamyl transferase (GGT) levels, cerebrovascular risk factors, and distribution of cerebral infarct areas in patients with acute ischemic stroke (AIS). Patients and Methods. Sixty patients with AIS and 44 controls who had not cerebrovascular disease were included in the study. The patients were divided into four groups according to the location of the infarct area and evaluated as for GGT levels and the presence of diabetes mellitus (DM), hypertension (HT), and hyperlipidemia (HL). Results. The frequency of DM, HT, and HL and gender distributions were similar. The mean GGT levels were significantly higher in patients with AIS and those with relatively larger areas of infarction (P<0.05). Increased mean GGT levels were found in the subgroup with hypertension, higher LDL-cholesterol, and triglyceride levels among cases with AIS (P<0.05). Conclusion. Higher GGT levels in AIS patients reinforce the relationship of GGT with inflammation and oxidative stress. The observation of higher GGT levels in patients with relatively larger areas of infarction is indicative of a positive correlation between increases in infarct areas and elevated GGT levels.

scholarly journals Association between Serum Gamma-glutamyl transferase and Intracranial Arterial Calcification in Acute Ischemic Stroke Subjects

2019 ◽  
Vol 9 (1) ◽  
Author(s):  
Tao Yao ◽  
Jing Li ◽  
Qi Long ◽  
Gang Li ◽  
Yanbin Ding ◽  
...  
Keyword(s):  
Ischemic Stroke ◽  
Acute Ischemic Stroke ◽  
Arterial Calcification ◽  
Gamma Glutamyl Transferase ◽  
Intracranial Artery ◽  
Gamma Glutamyl ◽  
Risk Of Death ◽  
All Cause Mortality ◽  
Male Patients ◽  
Glutamyl Transferase

AbstractIntracranial artery calcification (IAC) is an important risk factor for cerebral infarction and a key biomarker for intracranial artery stenosis. Gamma-glutamyl transferase (GGT) has been independently associated with increased cardiovascular events and coronary calcification. Our study assessed whether GGT is an independent factor for IAC in acute ischemic stroke (AIS) patients. This cross-sectional study involved a total of 754 patients with AIS (mean age: 65 ± 13.2 years). All the patients had received brain computed tomography angiography (CTA) examination to evaluate IAC. Further, serum GGT levels and other biochemical parameters were analyzed. The average GGT level in patients who died was also significantly increased (37.0 ± 26.8 vs 29.0 ± 21.5 U/L, p = 0.012). Partial correlation analysis showed that serum GGT levels were associated with NIHSS score at admission after adjustment for age and gender was considered (r = 0.150, p = 0.001). Logistic regression analysis showed that serum GGT levels independently predicted all-cause mortality (OR = 1.036, 95% CI: 1.014–1.060, p = 0.002), NIHSS scores (β = 0.051, 95% CI: 0.020–0.082, p = 0.001) and IAC scores (β = 0.006, 95% CI: 0.003–0.014, p = 0.005) in male patients. Each SD (standard deviation) increase of serum GGT levels was also associated with risk of all-cause mortality (OR = 2.272, 95% CI: 1.364–3.787, P = 0.002). GGT levels in patients with severe IAC were significantly elevated (37.6 ± 33.6 vs 28.6 ± 19.2, p < 0.001). However, serum GGT levels could not independently predict the severity of IAC in AIS patients. Our study identified that serum GGT levels were significantly elevated in patients who died, and the GGT levels had a certain association with the risk of death and IAC in male patients.

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