Simulation and Identification of the Neck Muscle Activities During Head and Neck Flexion Whiplash

Author(s):  
Yih-Charng Deng ◽  
Joanne Fu
Ergonomics ◽  
2019 ◽  
Vol 62 (12) ◽  
pp. 1524-1533 ◽  
Author(s):  
Suwalee Namwongsa ◽  
Rungthip Puntumetakul ◽  
Manida Swangnetr Neubert ◽  
Rose Boucaut

Basal Ganglia ◽  
2012 ◽  
Vol 2 (2) ◽  
pp. 103-107 ◽  
Author(s):  
Harald Hefter ◽  
Ulrike Kahlen ◽  
Till R. Menge ◽  
Dietmar Rosenthal ◽  
Marek Moll

Author(s):  
Sang Hoon Kim ◽  
Tae Hyun Kim ◽  
Chul Won Yang ◽  
Sun A Choi ◽  
Seung Geun Yeo ◽  
...  

2019 ◽  
Vol 64 (17) ◽  
pp. 175018
Author(s):  
Molly M McCulloch ◽  
Brian M Anderson ◽  
Guillaume Cazoulat ◽  
Christine B Peterson ◽  
Abdallah S R Mohamed ◽  
...  

1998 ◽  
Vol 84 (2) ◽  
pp. 450-453 ◽  
Author(s):  
Chester A. Ray ◽  
Keith M. Hume

Ray, Chester A., and Keith M. Hume. Neck afferents and muscle sympathetic activity in humans: implications for the vestibulosympathetic reflex. J. Appl. Physiol. 84(2): 450–453, 1998.—We have shown previously that head-down neck flexion (HDNF) in humans elicits increases in muscle sympathetic nerve activity (MSNA). The purpose of this study was to determine the effect of neck muscle afferents on MSNA. We studied this question by measuring MSNA before and after head rotation that would activate neck muscle afferents but not the vestibular system (i.e., no stimulation of the otolith organs or semicircular canals). After a 3-min baseline period with the head in the normal erect position, subjects rotated their head to the side (∼90°) and maintained this position for 3 min. Head rotation was performed by the subjects in both the prone ( n = 5) and sitting ( n = 6) positions. Head rotation did not elicit changes in MSNA. Average MSNA, expressed as burst frequency and total activity, was 13 ± 1 and 13 ± 1 bursts/min and 146 ± 34 and 132 ± 27 units/min during baseline and head rotation, respectively. There were no significant changes in calf blood flow (2.6 ± 0.3 to 2.5 ± 0.3 ml ⋅ 100 ml−1 ⋅ min−1; n = 8) and calf vascular resistance (39 ± 4 to 41 ± 4 units; n = 8). Heart rate (64 ± 3 to 66 ± 3 beats/min; P = 0.058) and mean arterial pressure (90 ± 3 to 93 ± 3; P < 0.05) increased slightly during head rotation. Additional neck flexion studies were performed with subjects lying on their side ( n = 5). MSNA, heart rate, and mean arterial pressure were unchanged during this maneuver, which also does not engage the vestibular system. HDNF was tested in 9 of the 13 subjects. MSNA was significantly increased by 79 ± 12% ( P < 0.001) during HDNF. These findings indicate that neck afferents activated by horizontal neck rotation or flexion in the absence of significant force development do not elicit changes in MSNA. These findings support the concept that HDNF increases MSNA by the activation of the vestibular system.


1994 ◽  
Vol 22 (5) ◽  
pp. 586-588 ◽  
Author(s):  
S. J. Yap ◽  
R. W. Morris ◽  
D. A. Pybus

The effect of head and neck movement and Trendelenburg tilt on endotracheal tube position, relative to the carina, was studied in fifty adult patients requiring intubation for elective surgery. On average, inward movement, that is shortening of the distance between the endotracheal tube tip and the carina, resulted from neck flexion (mean = −5.5 mm), whereas outward movement occurred with neck extension (mean = 6.3 mm). Neck rotation, to right and left, and Trendelenburg tilt did not show any trend towards inward nor outward movement (mean = 0.3 mm/1.7 mm/—0.6 mm, respectively). Whilst these mean positional changes for flexion and extension confirm the findings of earlier investigations, our range of maximum inward and outward displacement for flexion (23 mm in/19 mm out), extension (21 mm in/33 mm out), rotation to right (19 mm in/17 mm out), to left (22 mm in/19 mm out) and Trendelenburg tilt (22 mm in/16 mm out) indicate that for any given postural change in any one patient, the direction and magnitude of endotracheal tube displacement is not readily predictable.


2003 ◽  
Vol 95 (4) ◽  
pp. 1560-1566 ◽  
Author(s):  
Kristina Kairaitis ◽  
Radha Parikh ◽  
Rosie Stavrinou ◽  
Sarah Garlick ◽  
Jason P. Kirkness ◽  
...  

Transmural pressure at any level in the upper airway is dependent on the difference between intraluminal airway and extraluminal tissue pressure (ETP). We hypothesized that ETP would be influenced by topography, head and neck position, resistive loading, and stimulated breathing. Twenty-eight male, New Zealand White, anesthetized, spontaneously breathing rabbits breathed via a face mask with attached pneumotachograph to measure airflow and pressure transducer to monitor mask pressure. Tidal volume was measured via integration of the airflow signal. ETP was measured with a pressure transducer-tipped catheter inserted in the tissues of the lateral (ETPlat, n = 28) and anterior (ETPant, n = 21) pharyngeal wall. Head position was controlled at 30, 50, or 70°, and the effect of addition of an external resistor, brief occlusion, or stimulated breathing was examined. Mean ETPlat was ∼0.7 cmH2O greater than mean ETPant when adjusted for degree of head and neck flexion ( P < 0.05). Mean, maximum, and minimum ETP values increased significantly by 0.7-0.8 cmH2O/20° of head and neck flexion when adjusted for site of measurement ( P < 0.0001). The main effect of resistive loading and occlusion was an increase in the change in ETPlat (maximum - minimum ETPlat) and change in ETPant at all head and neck positions ( P < 0.05). Mean ETPlat and ETPant increased with increasing tidal volume at head and neck position of 30° (all P < 0.05). In conclusion, ETP was nonhomogeneously distributed around the upper airway and increased with both increasing head and neck flexion and increasing tidal volume. Brief airway occlusion increased the size of respiratory-related ETP fluctuations in upper airway ETP.


2018 ◽  
pp. 121-228 ◽  
Author(s):  
Rui Diogo ◽  
Janine M. Ziermann ◽  
Julia Molnar ◽  
Natalia Siomava ◽  
Virginia Abdala

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