scholarly journals Herpes simplex virus type 2 or human herpesvirus 8 infection and prostate cancer risk: A meta-analysis

2013 ◽  
Vol 1 (3) ◽  
pp. 433-439 ◽  
Author(s):  
XIAOXIAO GE ◽  
XIAO WANG ◽  
PENG SHEN
Keyword(s):  
Prostate Cancer ◽  
Herpes Simplex Virus ◽  
Herpes Simplex ◽  
Meta Analysis ◽  
Prostate Cancer Risk ◽  
Human Herpesvirus ◽  
Human Herpesvirus 8 ◽  
Herpesvirus 8 ◽  
Simplex Virus

scholarly journals Identification of Herpes Simplex Virus DNA and Lack of Human Herpesvirus-8 DNA in Mycosis Fungoides

2002 ◽  
Vol 82 (3) ◽  
pp. 214-216 ◽  
Author(s):  
Emel Erkek ◽  
Nilgün Sentürk ◽  
Irem Dinçer ◽  
Ali Ilgın Olut ◽  
Tanıl Kocagöz ◽  
...  
Keyword(s):  
Herpes Simplex Virus ◽  
Herpes Simplex ◽  
Mycosis Fungoides ◽  
Human Herpesvirus ◽  
Human Herpesvirus 8 ◽  
Herpesvirus 8 ◽  
Simplex Virus

scholarly journals Risk of Bacterial Vaginosis Among Women With Herpes Simplex Virus Type 2 Infection: A Systematic Review and Meta-analysis

2015 ◽  
Vol 212 (1) ◽  
pp. 8-17 ◽  
Author(s):  
A. Esber ◽  
R. D. Vicetti Miguel ◽  
T. L. Cherpes ◽  
M. A. Klebanoff ◽  
M. F. Gallo ◽  
...  
Keyword(s):  
Systematic Review ◽  
Herpes Simplex Virus ◽  
Herpes Simplex ◽  
Bacterial Vaginosis ◽  
Virus Type ◽  
Herpes Simplex Virus Type ◽  
Meta Analysis ◽  
Simplex Virus

scholarly journals Herpes Simplex Virus Type 1 and Type 2 Infection Increases Atherosclerosis Risk: Evidence Based on a Meta-Analysis

2016 ◽  
Vol 2016 ◽  
pp. 1-9 ◽  
Author(s):  
Yu peng Wu ◽  
Dan dan Sun ◽  
Yun Wang ◽  
Wen Liu ◽  
Jun Yang
Keyword(s):  
Herpes Simplex Virus ◽  
Herpes Simplex ◽  
Virus Type ◽  
Herpes Simplex Virus Type ◽  
Subgroup Analysis ◽  
Meta Analysis ◽  
Simplex Virus ◽  
Hsv 1

Objective.The aim of our study was to evaluate the relation of herpes simplex virus type 1 (HSV-1) and type 2 (HSV-2) infection with the risk of atherosclerosis (AS).Methods.A systematic literature search was performed through three electronic databases. The pooled odds ratio (OR) and corresponding 95% confidence interval (CI) were used to assess the effect of HSV-1 and HSV-2 infection on AS risk.Results.17 studies were available for meta-analysis of HSV-1 infection and AS risk and seven studies for meta-analysis of HSV-2 infection and AS risk. Subjects exposed to HSV-1 infection exhibited an increased risk of AS (OR = 1.77; 95% CI: 1.40–2.23;P<0.001). And consistent elevated AS risks for HSV-1 positive subjects were found in all subgroup analysis of disease type, region, male proportion, and age. HSV-2 positive subjects demonstrated significantly increased AS risk (OR = 1.37; 95% CI: 1.13–1.67;P<0.005). In subgroup analysis, elevated AS risks were only observed in myocardial ischemia group, male proportion >60% group, and age ≤60-year-old group.Conclusion.Our meta-analysis indicated that HSV-1 and HSV-2 infection could increase the risk of contracting AS.

scholarly journals Mechanisms of Viral Infections Associated with HIV

2011 ◽  
Vol 23 (1) ◽  
pp. 130-136 ◽  
Author(s):  
S.M. Tugizov ◽  
J.Y. Webster-Cyriaque ◽  
S. Syrianen ◽  
A. Chattopadyay ◽  
H. Sroussi ◽  
...  
Keyword(s):  
Herpes Simplex ◽  
Epstein Barr Virus ◽  
Human Herpesvirus ◽  
Human Herpesvirus 8 ◽  
Viral Pathogens ◽  
Herpesvirus 8 ◽  
Varicella Zoster ◽  
Barr Virus ◽  
Epstein Barr ◽  
Simplex Virus

HIV infection is commonly associated with activation and dissemination of several other viral pathogens, including herpes simplex virus 1/2, human cytomegalovirus, human herpesvirus 8, Epstein-Barr virus, Varicella Zoster virus, and human papillomavirus, which behave as opportunistic agents and cause various diseases in immunocompromised hosts. The increased frequency and severity of diseases caused by these viruses in HIV-infected individuals is due mainly to dysfunction of both the adaptive and innate immune responses to viral pathogens. In addition, molecular interactions between HIV and these opportunistic viruses are likely to play critical roles in the progression of disease, including neoplasia. This report reviews the critical aspects of HIV interaction with opportunistic viruses, including Epstein-Barr virus, human cytomegalovirus, herpes simplex virus, Varicella Zoster virus, human herpesvirus 8, and human papillomavirus.

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