scholarly journals Unique characteristics of rebound nystagmus in ENG recordings of 3 patients

2021 ◽  
Vol 80 (2) ◽  
pp. 75-86
Author(s):  
Jun-Ichi Yokota ◽  
Ayako Inoshita ◽  
Yoko Yamaguchi
Keyword(s):  
1984 ◽  
Vol 15 (5) ◽  
pp. 474-477 ◽  
Author(s):  
Roberta L. Bondar ◽  
James A. Sharpe ◽  
Anthony J. Lewis

Brain ◽  
1973 ◽  
Vol 96 (3) ◽  
pp. 507-526 ◽  
Author(s):  
J. D. HOOD ◽  
A. KAYAN ◽  
J. LEECH
Keyword(s):  

2003 ◽  
Vol 18 (10) ◽  
pp. 1201-1204 ◽  
Author(s):  
Takao Hashimoto ◽  
Osamu Sasaki ◽  
Kunihiro Yoshida ◽  
Yo-ichi Takei ◽  
Shu-ichi Ikeda

2002 ◽  
Vol 956 (1) ◽  
pp. 585-587
Author(s):  
M. L. ROSENBERG ◽  
D. S. ZEE
Keyword(s):  

2009 ◽  
Vol 29 (1) ◽  
pp. 26-28 ◽  
Author(s):  
Sun-Young Oh ◽  
Man-Wook Seo ◽  
Young-Hyun Kim ◽  
Kwang-Dong Choi ◽  
Dae-Seong Kim ◽  
...  

2020 ◽  
Vol 11 ◽  
Author(s):  
Michelle Sari Ritter ◽  
Giovanni Bertolini ◽  
Dominik Straumann ◽  
Stefan Yu Bögli
Keyword(s):  

1988 ◽  
Vol 47 (3) ◽  
pp. 339-344
Author(s):  
Kanji Baba ◽  
Eiji Sakata ◽  
Tetsuzo Inoue
Keyword(s):  

JAMA ◽  
1974 ◽  
Vol 227 (6) ◽  
pp. 648 ◽  
Author(s):  
James A. Sharpe

1986 ◽  
Vol 79 (4) ◽  
pp. 535-546
Author(s):  
Kazuhiro TERAMOTO ◽  
Kyoko OHTSU ◽  
Eiji SAKATA
Keyword(s):  

Author(s):  
T.H. Kirkham ◽  
D. Guitton ◽  
S.G. Coupland

SUMMARY:Giant axonal neuropathy, a generalised disorder or neurofilaments, presents as a chronic, progressive peripheral neuropathy in childhood. Evidence for central nervous system involvement is demonstrated in this study of four male patients with giant axonal neuropathy who had defective visual function and abnormal ocular motility. The visual system was studied by electroretinography, which showed normal retinal function, and by visual evoked potentials, which showed disease of both optic nerves and retrochiasmal visual pathways. The ocular motility disorder, studied by electrooculography, comprised defective pursuit, inability to maintain eccentric gaze with gaze paretic and rebound nystagmus, abnormal optokinetic responses and failure of suppression of the vestibulo-ocular reflex by fixation. These findings suggested involvement by giant axonal neuropathy of the cerebellar and brain stem pathways important in the control of ocular motility.


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