scholarly journals Complete gaze nystagmus and rebound nystagmus in all directions - A case with chronic toluene intoxication.

1988 ◽  
Vol 47 (3) ◽  
pp. 339-344
Author(s):  
Kanji Baba ◽  
Eiji Sakata ◽  
Tetsuzo Inoue
Keyword(s):  
Rebound Nystagmus

Rebound nystagmus in olivocerebellar atrophy: A clinicopathological correlation

1984 ◽  
Vol 15 (5) ◽  
pp. 474-477 ◽  
Author(s):  
Roberta L. Bondar ◽  
James A. Sharpe ◽  
Anthony J. Lewis
Keyword(s):  
Clinicopathological Correlation ◽  
Rebound Nystagmus

REBOUND NYSTAGMUS

Brain ◽  
1973 ◽  
Vol 96 (3) ◽  
pp. 507-526 ◽  
Author(s):  
J. D. HOOD ◽  
A. KAYAN ◽  
J. LEECH
Keyword(s):  
Rebound Nystagmus

Periodic alternating nystagmus and rebound nystagmus in spinocerebellar ataxia type 6

2003 ◽  
Vol 18 (10) ◽  
pp. 1201-1204 ◽  
Author(s):  
Takao Hashimoto ◽  
Osamu Sasaki ◽  
Kunihiro Yoshida ◽  
Yo-ichi Takei ◽  
Shu-ichi Ikeda
Keyword(s):  
Spinocerebellar Ataxia ◽  
Spinocerebellar Ataxia Type ◽  
Spinocerebellar Ataxia Type 6 ◽  
Rebound Nystagmus

Unilateral Rebound Nystagmus

2002 ◽  
Vol 956 (1) ◽  
pp. 585-587
Author(s):  
M. L. ROSENBERG ◽  
D. S. ZEE
Keyword(s):  
Rebound Nystagmus

Gaze-Evoked and Rebound Nystagmus in a Case of Migrainous Vertigo

2009 ◽  
Vol 29 (1) ◽  
pp. 26-28 ◽  
Author(s):  
Sun-Young Oh ◽  
Man-Wook Seo ◽  
Young-Hyun Kim ◽  
Kwang-Dong Choi ◽  
Dae-Seong Kim ◽  
...  
Keyword(s):  
Migrainous Vertigo ◽  
Rebound Nystagmus

scholarly journals Prevalence and Characteristics of Physiological Gaze-Evoked and Rebound Nystagmus: Implications for Testing Their Pathological Counterparts

2020 ◽  
Vol 11 ◽  
Author(s):  
Michelle Sari Ritter ◽  
Giovanni Bertolini ◽  
Dominik Straumann ◽  
Stefan Yu Bögli
Keyword(s):  
Rebound Nystagmus

Rebound Nystagmus—A Cerebellar Sign?

JAMA ◽  
1974 ◽  
Vol 227 (6) ◽  
pp. 648 ◽  
Author(s):  
James A. Sharpe
Keyword(s):  
Cerebellar Sign ◽  
Rebound Nystagmus

scholarly journals Vertical rebound nystagmus.

1986 ◽  
Vol 79 (4) ◽  
pp. 535-546
Author(s):  
Kazuhiro TERAMOTO ◽  
Kyoko OHTSU ◽  
Eiji SAKATA
Keyword(s):  
Rebound Nystagmus

scholarly journals Giant Axonal Neuropathy: Visual and Oculomotor Deficits

1980 ◽  
Vol 7 (3) ◽  
pp. 177-184 ◽  
Author(s):  
T.H. Kirkham ◽  
D. Guitton ◽  
S.G. Coupland
Keyword(s):  
Axonal Neuropathy ◽  
Central Nervous System Involvement ◽  
Motility Disorder ◽  
Ocular Motility ◽  
Giant Axonal Neuropathy ◽  
Ocular Reflex ◽  
Optokinetic Responses ◽  
Vestibulo Ocular Reflex ◽  
Male Patients ◽  
Rebound Nystagmus

SUMMARY:Giant axonal neuropathy, a generalised disorder or neurofilaments, presents as a chronic, progressive peripheral neuropathy in childhood. Evidence for central nervous system involvement is demonstrated in this study of four male patients with giant axonal neuropathy who had defective visual function and abnormal ocular motility. The visual system was studied by electroretinography, which showed normal retinal function, and by visual evoked potentials, which showed disease of both optic nerves and retrochiasmal visual pathways. The ocular motility disorder, studied by electrooculography, comprised defective pursuit, inability to maintain eccentric gaze with gaze paretic and rebound nystagmus, abnormal optokinetic responses and failure of suppression of the vestibulo-ocular reflex by fixation. These findings suggested involvement by giant axonal neuropathy of the cerebellar and brain stem pathways important in the control of ocular motility.

Clinical Significance of Rebound Nystagmus in Neuro-Otological Diagnosis

1978 ◽  
Vol 87 (2) ◽  
pp. 238-242 ◽  
Author(s):  
C. Morales-Garcia ◽  
C. Arriagada ◽  
J. L. Cardenas ◽  
J. Otte
Keyword(s):  
Clinical Analysis ◽  
Cerebellar Dysfunction ◽  
Cerebellar Sign ◽  
Cerebellar Peduncles ◽  
Postmortem Studies ◽  
Cerebellar Involvement ◽  
Almost All ◽  
The Brain ◽  
Straight Ahead ◽  
Rebound Nystagmus

The present study is concerned with a clinical analysis of 17 patients with “rebound nystagmus” examined over a period of three years. All of them have shown a short duration second degree nystagmus evoked by changes in the direction of fixation, from the lateral to straight ahead gaze. This nystagmus was a fixation nystagmus, that is to say, it was enhanced in the presence of active optic fixation and inhibited in its absence. Almost all the patients (16 out of 17) had cerebellar signs on neurological examination (in one subject rebound nystagmus was the first sign suggesting cerebellar involvement and appeared several months before any other cerebellar sign was present). Rebound nystagmus was far more common than the other neuro-otological signs suggesting cerebellar dysfunction (vestibular hyperexcitability, dysrhythmia in postcaloric nystagmus and ocular dysmetria). In three out of four patients with unilateral lesions rebound nystagmus was ipsilateral with respect to the side of the lesion. Postmortem studies were carried out upon five cases and showed either pathological changes in the cerebellum or a lesion involving the cerebellar peduncles in the brain stem.

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