scholarly journals Slowing of Electrical Activity in Ventricular Fibrillation is Not Associated with Increased Defibrillation Energies in the Isolated Rabbit Heart

2011 ◽  
Vol 2 ◽  
Author(s):  
Jane C. Caldwell ◽  
Francis L. Burton ◽  
Stuart M. Cobbe ◽  
Godfrey L. Smith
2003 ◽  
Vol 14 (10) ◽  
pp. 1077-1084 ◽  
Author(s):  
HIDEKI HAYASHI ◽  
YASUSHI MIYAUCHI ◽  
CHUNG‐CHUAN CHOU ◽  
HRAYR S. KARAGUEUZIAN ◽  
PENG‐SHENG CHEN ◽  
...  

1957 ◽  
Vol 12 (2) ◽  
pp. 215-218 ◽  
Author(s):  
A. K. ARMITAGE ◽  
J. H. BURN ◽  
A. J. GUNNING

Circulation ◽  
1998 ◽  
Vol 98 (16) ◽  
pp. 1688-1696 ◽  
Author(s):  
Ravi Mandapati ◽  
Yukio Asano ◽  
William T. Baxter ◽  
Richard Gray ◽  
Jorge Davidenko ◽  
...  

1989 ◽  
Vol 30 (3) ◽  
pp. 327-333 ◽  
Author(s):  
L. Bååth ◽  
T. Almén

Perfusion of the isolated rabbit heart with solutions (350 mg I/ml) of the non-ionic contrast media iohexol and iopentol, both containing NaCl (20 mmol/l), caused a significantly lower frequency of ventricular fibrillation (VF) than solutions without NaCl. Iohexol or iopentol with NaCl (10 mmol/l) caused an intermediate frequency of VF. Iohexol with 10 or 20 mmol NaCl/l caused about the same frequency of VF as iohexol solutions with about the same total electrolyte concentration but with electrolyte composition as that of Krebs' solution. At 320 mg I/ml, solutions of iohexol (20 mmol NaCl/l), iodixanol (20 mmol NaCl/l) and ioxaglate (155 mmol Na/l) all produced a significantly lower frequency of VF than iohexol without NaCl. Ioxaglate caused the largest and iodixanol the smallest decrease in contractile force of the media. The investigation suggests that the small risk of VF from non-ionic monomeric media can be further reduced by adding a small amount of sodium chloride or of the electrolytes of Krebs' solution.


1964 ◽  
Vol 42 (1) ◽  
pp. 21-24 ◽  
Author(s):  
J.-C. Panisset ◽  
R. Carle ◽  
A. Beaulnes

The influence of perfusion pressure on ventricular fibrillation was studied in the isolated rabbit heart. Sudden drops in perfusion pressure inhibit ventricular fibrillation in a few minutes, whereas a slow decrease of the pressure does not stop the fibrillation, but nevertheless reduces the ventricular firing rate. High perfusion pressures facilitate the induction of fibrillation; low pressures delay the production of arrhythmias and shorten their duration. It is postulated that this effect of pressure is primarily mechanical and not strictly dependent upon changes in the coronary flow or in the myocardial temperature.


2000 ◽  
Vol 86 (1) ◽  
pp. 86-93 ◽  
Author(s):  
Jay Chen ◽  
Ravi Mandapati ◽  
Omer Berenfeld ◽  
Allan C. Skanes ◽  
José Jalife

Sign in / Sign up

Export Citation Format

Share Document