scholarly journals Contralateral efferent suppression of human hearing sensitivity

2015 ◽  
Vol 8 ◽  
Author(s):  
Enzo Aguilar ◽  
Peter T. Johannesen ◽  
Enrique A. Lopez-Poveda
Keyword(s):  
Hearing Sensitivity ◽  
Human Hearing ◽  
Efferent Suppression

The Effects of Impulsive Noise Upon Human Hearing Sensitivity (8 to 20 kHz)

1981 ◽  
Vol 10 (1) ◽  
pp. 21-29 ◽  
Author(s):  
S. A. Fausti ◽  
D. A. Erickson ◽  
R. H. Frey ◽  
B. Z. Rappaport
Keyword(s):  
Impulsive Noise ◽  
Hearing Sensitivity ◽  
Human Hearing

The effects of noise upon human hearing sensitivity from 8000 to 20 000 Hz

1981 ◽  
Vol 69 (5) ◽  
pp. 1343-1349 ◽  
Author(s):  
Stephen A. Fausti ◽  
Deborah A. Erickson ◽  
Richard H. Frey ◽  
B. Z. Rappaport ◽  
M. A. Schechter
Keyword(s):  
Hearing Sensitivity ◽  
Human Hearing

Effects of steady-state noise upon human hearing sensitivity from 8000 to 20 000 Hz

AIHAJ ◽  
1980 ◽  
Vol 41 (6) ◽  
pp. 427-432 ◽  
Author(s):  
DEBORAH A. ERICKSON ◽  
STEPHEN A. FAUSTI ◽  
RICHARD H. FREY ◽  
B. Z. RAPPAPORT
Keyword(s):  
Steady State ◽  
Hearing Sensitivity ◽  
Human Hearing

Unilateral Hearing Losses Alter Loud Sound-Induced Temporary Threshold Shifts and Efferent Effects in the Normal-Hearing Ear

2001 ◽  
Vol 85 (3) ◽  
pp. 1257-1269 ◽  
Author(s):  
R. Rajan
Keyword(s):  
Hearing Loss ◽  
Normal Hearing ◽  
Unilateral Hearing Loss ◽  
Loud Tone ◽  
Loud Sound ◽  
Hearing Sensitivity ◽  
Sound Levels ◽  
Test Category ◽  
The One ◽  
Efferent Suppression

In animals with bilaterally normal hearing, olivocochlear pathways can protect the cochlea from the temporary shifts in hearing sensitivity (temporary threshold shifts; TTSs) caused by short-duration intense loud sounds. The crossed olivocochlear pathway provides protection during binaural loud sound, and uncrossed pathways protect when monaural or binaural loud sounds occur in noise backgrounds. Here I demonstrate that when there is a chronic unilateral hearing loss, effects of loud sounds, and efferent effects on loud sound, in the normal-hearing ear differ markedly from normal. Three categories of test animals with unilateral hearing loss were tested for effects at the normal-hearing ear. In all categories a monaural loud tone to the normal-hearing ear produced lower-than-normal TTSs, apparently because of a tonic re-setting of that ear's susceptibility to loud sound. Second, in the two test categories in which the hearing-loss ear was only partly damaged, binaural loud sound exacerbated TTSs in the normal-hearing ear because it caused threshold shifts that were a combination of “pure” TTSs and uncrossed efferent suppression of cochlear sensitivity. (In normal cats, this binaural tone results in crossed olivocochlear protection that reduces TTS.) Binaural loud sound did not produce such uncrossed efferent effects in the test category in which the nontest ear had suffered total hearing loss, suggesting that this uncrossed efferent effect required binaural input to the CNS. It is noteworthy that, in the absence of this uncrossed efferent suppression, the pure loud sound-alone induced TTSs after binaural exposure were low. Thus in the absence of any efferent effect, the normal-hearing cochlea had a reduced susceptibility to loud tone-induced damage. Finally, the results suggest that, with respect to cochlear actions at high sound levels, uncrossed and crossed efferent pathways may exert different effects at the one type of receptor cell.

Contralateral suppression of human hearing sensitivity in single-sided deaf cochlear implant users

2019 ◽  
Vol 373 ◽  
pp. 121-129
Author(s):  
Waldo Nogueira ◽  
Benjamin Krüger ◽  
Andreas Büchner ◽  
Enrique Lopez-Poveda
Keyword(s):  
Cochlear Implant ◽  
Hearing Sensitivity ◽  
Contralateral Suppression ◽  
Human Hearing

A Questionnaire Survey of Current Rehabilitation Practices for Adults With Normal Hearing Sensitivity Who Experience Auditory Difficulties

2020 ◽  
Vol 29 (4) ◽  
pp. 738-761
Author(s):  
Tess K. Koerner ◽  
Melissa A. Papesh ◽  
Frederick J. Gallun
Keyword(s):  
Hearing Aids ◽  
Questionnaire Survey ◽  
Patient Population ◽  
Clinical Care ◽  
Normal Hearing ◽  
Auditory Training ◽  
Training Methods ◽  
Hearing Sensitivity ◽  
Considerable Benefit ◽  
High Level

Purpose A questionnaire survey was conducted to collect information from clinical audiologists about rehabilitation options for adult patients who report significant auditory difficulties despite having normal or near-normal hearing sensitivity. This work aimed to provide more information about what audiologists are currently doing in the clinic to manage auditory difficulties in this patient population and their views on the efficacy of recommended rehabilitation methods. Method A questionnaire survey containing multiple-choice and open-ended questions was developed and disseminated online. Invitations to participate were delivered via e-mail listservs and through business cards provided at annual audiology conferences. All responses were anonymous at the time of data collection. Results Responses were collected from 209 participants. The majority of participants reported seeing at least one normal-hearing patient per month who reported significant communication difficulties. However, few respondents indicated that their location had specific protocols for the treatment of these patients. Counseling was reported as the most frequent rehabilitation method, but results revealed that audiologists across various work settings are also successfully starting to fit patients with mild-gain hearing aids. Responses indicated that patient compliance with computer-based auditory training methods was regarded as low, with patients generally preferring device-based rehabilitation options. Conclusions Results from this questionnaire survey strongly suggest that audiologists frequently see normal-hearing patients who report auditory difficulties, but that few clinicians are equipped with established protocols for diagnosis and management. While many feel that mild-gain hearing aids provide considerable benefit for these patients, very little research has been conducted to date to support the use of hearing aids or other rehabilitation options for this unique patient population. This study reveals the critical need for additional research to establish evidence-based practice guidelines that will empower clinicians to provide a high level of clinical care and effective rehabilitation strategies to these patients.

On the Potential Deterioration in Hearing Due to Hearing Aid Usage

1981 ◽  
Vol 24 (1) ◽  
pp. 3-15 ◽  
Author(s):  
Larry E. Humes ◽  
Fred H. Bess
Keyword(s):  
Hearing Aid ◽  
Threshold Shift ◽  
Temporary Threshold Shift ◽  
Hearing Sensitivity

This manuscript examines the issue of potential decline in hearing sensitivity due to hearing aid usage through an analysis of data obtained from the temporary threshold shift (TTS) paradigm. Following a critique of the traditional measures of TTS, the concept of integrated TTS (ITTS) is reviewed and data on hearing-aid-induced ITTS are presented. In addition, a series of equations relating permanent threshold shift (PTS) to a recently developed measure of noise dose (Dn) is derived and predictions for hearing-aid-induced PTS are made. Recommended gain settings established to protect the hearing of a person wearing a hearing aid from further decline following various durations of hearing aid usage are also provided.

Hearing in Mice by GSR Audiometry: II. Magnitude of Unconditioned GSR as a Function of Intensity and Frequency Interactions

1968 ◽  
Vol 11 (1) ◽  
pp. 169-178 ◽  
Author(s):  
Alan Gill ◽  
Charles I. Berlin
Keyword(s):  
Power Law ◽  
Response Magnitude ◽  
Single Units ◽  
Spectral Content ◽  
Hearing Sensitivity ◽  
Subjective Magnitude ◽  
Orderly Fashion ◽  
Viiith Nerve

The unconditioned GSR’s elicited by tones of 60, 70, 80, and 90 dB SPL were largest in the mouse in the ranges around 10,000 Hz. The growth of response magnitude with intensity followed a power law (10 .17 to 10 .22 , depending upon frequency) and suggested that the unconditioned GSR magnitude assessed overall subjective magnitude of tones to the mouse in an orderly fashion. It is suggested that hearing sensitivity as assessed by these means may be closely related to the spectral content of the mouse’s vocalization as well as to the number of critically sensitive single units in the mouse’s VIIIth nerve.

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