scholarly journals Dietary methionine deficiency reduces laying performances of female common ducks and impacts traits of interest of their mule ducklings

2019 ◽  
Vol 98 (11) ◽  
pp. 5590-5600
Author(s):  
L Bodin ◽  
A Sécula ◽  
H Chapuis ◽  
A Cornuez ◽  
M Lessire ◽  
...  
1950 ◽  
Vol 186 (1) ◽  
pp. 377-385
Author(s):  
A.E. Denton ◽  
J.N. Williams ◽  
C.A. Elvehjem

2018 ◽  
Vol 66 (1) ◽  
pp. 52-65 ◽  
Author(s):  
Tao Ruan ◽  
Lingjun Li ◽  
Yingnan Lyu ◽  
Qin Luo ◽  
Bangyuan Wu

The aim of this study was to investigate the effects of methionine (Met) deficiency on antioxidant functions (in the duodenal, jejunal and ileal mucosa) and apoptosis in the duodenum, jejunum and ileum of broiler chickens. A total of 120 one-day-old Cobb broilers were divided into two groups and fed a Metdeficient diet and a control diet, respectively, for six weeks. The activities of superoxide dismutase (SOD), catalase (CAT) and glutathione peroxidase (GSH-Px), the ability to inhibit hydroxyl radicals, and glutathione (GSH) content were significantly decreased in the Met-deficient group compared to the control. In contrast, malondialdehyde (MDA) content was significantly higher in the Met-deficient group. As measured by terminal deoxynucleotidyl transferase 2’-deoxyuridine 5’- triphosphate dUTP nick end-labelling (TUNEL) and flow cytometry (FCM), the percentages of apoptotic cells were significantly increased. In conclusion, dietary Met deficiency can cause oxidative stress and then induce increased apoptosis in the intestine. Oxidative stress contributes to intestinal apoptosis. This results in the impairment of local intestinal mucosal immunity due to oxidative stress and apoptosis in the small intestine. The results of this study provide new experimental evidence for understanding the negative effects of Met deficiency on mucosal immunity or the functions of other immune tissues.


1993 ◽  
Vol 4 (11) ◽  
pp. 610-617 ◽  
Author(s):  
Hisashi Shinozuka ◽  
Masaaki Masuhara ◽  
Yoshitsugu Kubo ◽  
Sikandar L. Katyal

1966 ◽  
Vol 123 (1) ◽  
pp. 17-24 ◽  
Author(s):  
Soussan Mohajer ◽  
Janis Gabliks

The role of methionine in poliovirus infection in HeLa and monkey kidney cells was investigated by using the methionine analogue l-ethionine. In the presence of 2.0 x 10–3 and 4.0 x 10–3 moles ethionine, the growth of HeLa and monkey kidney cells was significantly inhibited. Under the same experimental conditions, ethionine had no significant effect on the biosynthesis of two strains of poliovirus (Mahoney and Lansing) in HeLa cells, whereas in primary monkey kidney cells, it markedly inhibited the biosynthesis of the Lansing strain of poliovirus. HeLa cells partly depleted of their intracellular amino acids did not change the rate of viral biosynthesis. The inhibitory effect of ethionine on cell growth and viral biosynthesis was reversed by addition of an excess of l-methionine.


2007 ◽  
Vol 53 (3) ◽  
pp. 767-776 ◽  
Author(s):  
Helieh S. Oz ◽  
Theresa S. Chen ◽  
Manuela Neuman

Sign in / Sign up

Export Citation Format

Share Document