scholarly journals Aflatoxin Effects in White Leghorn Chicken s Selected for Response to Sheep Erythrocyte Antigen

1985 ◽  
Vol 64 (6) ◽  
pp. 1071-1076 ◽  
Author(s):  
C.O. UBOSI ◽  
W.B. GROSS ◽  
P.B. HAMILTON ◽  
M. EHRICH ◽  
P.B. SIEGEL
1985 ◽  
Vol 64 (6) ◽  
pp. 1065-1070 ◽  
Author(s):  
C.O. UBOSI ◽  
P.B. HAMILTON ◽  
E.A. DUNNINGTON ◽  
P.B. SIEGEL

1972 ◽  
Vol 51 (1) ◽  
pp. 313-320 ◽  
Author(s):  
J.A. Asmar ◽  
P.L. Pellett ◽  
Nur Hariri ◽  
M.D. Hariri

1970 ◽  
Vol 1 (3) ◽  
pp. 288-292
Author(s):  
S. P. Chan ◽  
W. A. Hook ◽  
W. Turner ◽  
M. A. Chirigos

Infection of mice with the murine sarcoma virus (Moloney) markedly suppressed the humoral antibody response to sheep erythrocyte antigen injected 10 days after infection, when tumor size was maximal, and on day 26, when primary tumors had partially regressed. Humoral antibody response was also inhibited when antigen was injected at the time secondary tumors and metastases were evident. No significant suppression of humoral antibody was seen when mice were injected with sheep erythrocyte antigen 5 days after virus infection. Inhibition of the cellular immune response of murine sarcoma virus (Moloney)-infected mice, as measured by the increased survival time of skin grafts, was also determined. Mice that were infected 5 days prior to grafting demonstrated prolonged survival of grafts, suggesting a suppression of cellular immunity. These mice had a graft survival time 14 days greater than noninfected controls. No significant prolongation of graft survival was seen in mice grafted at the times of maximum primary tumor growth, of primary tumor regression, or when secondary tumors had appeared.


1942 ◽  
Vol 84 (3) ◽  
pp. 295-306 ◽  
Author(s):  
Norman M. Nelson

2018 ◽  
Vol 53 (3) ◽  
pp. 247
Author(s):  
Snehal D. Patil ◽  
F.P. Savaliya ◽  
A.B. Patel ◽  
H.I. Paleja ◽  
R.S. Joshi ◽  
...  

1994 ◽  
Vol 142 (1) ◽  
pp. 153-160 ◽  
Author(s):  
S M Kisliuk ◽  
R Grossmann

Abstract The influence of low humidity during incubation (LHI) on arginine vasotocin (AVT) secretion in male and female White Leghorn chicken embryos and chicks was investigated. LHI embryos were incubated from day 8 (E8) to E18 at a relative humidity of 32–35%, while control embryos were incubated at 58–62%. In the control groups, plasma AVT gradually increased from E15 to E18, increased sharply during hatching and returned to the El8 level within 4 h. LHI did not alter the level of AVT until E18. LHI E19 males had concentrations of AVT twice as high as those of control males (39 ± 3·9 vs 15 ± 1·6 pmol/l, P<0·001). Augmentation persisted during hatching and 4 h afterwards. In LHI females additional elevation reached significant levels only at 4 h after hatching (58 ± 13 vs 16 ± 1·7 pmol/l, P<0·01). Deprivation of water caused an increase in plasma sodium concentrations in the 2-day-old chicks of the control groups, while no significant changes were observed in the LHI group. Deprivation of water together with a high environmental temperature caused an additional increase in AVT only in the LHI groups (42 ± 5·2 vs 22 ± 3·9 pmol/l, P<0·01 for males; 32 ± 3·4 vs 19 ± 2·8 pmol/l, P<0·01 for females). The results clearly showed that LHI leads to a more pronounced response of the AVT system on subsequent osmotic and non-osmotic challenges, especially in males. Embryonal activation of the AVT system probably helps the organism to cope with hatching and posthatching stresses. Journal of Endocrinology (1994) 142, 153–160


2007 ◽  
Vol 21 (2) ◽  
pp. 110-120 ◽  
Author(s):  
Douglas P. Whiteside ◽  
Ian K. Barker ◽  
Peter D. Conlon ◽  
Angelo Tesoro ◽  
Jake J. Thiessen ◽  
...  

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