scholarly journals Response of Blood Plasma Glucose, Free Fatty Acids, Triglycerides, Insulin and Food Intake to Bovine Insulin in Geese and Cockerels

1973 ◽  
Vol 52 (3) ◽  
pp. 886-892 ◽  
Author(s):  
I. Nir ◽  
V. Levy
Keyword(s):  
Fatty Acids ◽  
Food Intake ◽  
Blood Plasma ◽  
Free Fatty Acids ◽  
Plasma Glucose ◽  
Bovine Insulin

2,5-anhydro-D-mannitol: a fructose analogue that increases food intake in rats

1988 ◽  
Vol 254 (1) ◽  
pp. R150-R153 ◽  
Author(s):  
M. G. Tordoff ◽  
R. Rafka ◽  
M. J. DiNovi ◽  
M. I. Friedman
Keyword(s):  
Fatty Acids ◽  
Food Intake ◽  
Free Fatty Acids ◽  
Plasma Glucose ◽  
Ketone Bodies ◽  
Diabetic Rats ◽  
Substrate Analogues ◽  
Increase Food Intake ◽  
Related Increase

We examined the effects on food intake and plasma fuels of 2,5-anhydro-D-mannitol (2,5-AM; 2-deoxy-D-fructose), a fructose analogue that inhibits hepatocyte gluconeogenesis and glycogenolysis in vitro. 2,5-AM (50-800 mg/kg po) given to rats during the diurnal fast produced a dose-related increase in food intake during the 2 h after administration. A 200-mg/kg dose of 2,5-AM decreased plasma glucose, increased plasma ketone bodies, free fatty acids, and glycerol, and had no effect on triglycerides. Normal and diabetic rats given 2,5-AM (200 mg/kg ip) increased food intake to the same extent. These results suggest that, unlike other substrate analogues that increase food intake, 2,5-AM increases feeding by creating a metabolic state that resembles fasting.

Effect of Chemical Stimulation of the Dorsomedial Hypothalamic Nucleus on Blood Plasma Glucose, Triglycerides and Free Fatty Acids in Rats

1997 ◽  
Vol 42 (3) ◽  
pp. 195-198 ◽  
Author(s):  
Cássia Thaïs B.V Zaia ◽  
Luis C.J Gaziri ◽  
Dimas A.M Zaia ◽  
Edson Delattre ◽  
Miriam S Dolnikoff ◽  
...  
Keyword(s):  
Fatty Acids ◽  
Blood Plasma ◽  
Free Fatty Acids ◽  
Plasma Glucose ◽  
Chemical Stimulation ◽  
Hypothalamic Nucleus ◽  
Dorsomedial Hypothalamic Nucleus ◽  
Stimulation Of

scholarly journals Effect of pain related of sex and estrous cycle on blood plasma glucose, free fatty acids and corticosterone in rats

2012 ◽  
Vol 34 (4) ◽  
Author(s):  
Elzira Diniz de Moraes ◽  
Cássia Thaís Bussamra Vieira Zaia ◽  
Dimas Augusto Morozin Zaia ◽  
Angelo Alexander Torres dos Santos ◽  
Rachel Cezar de Andrade Ribeiro ◽  
...  
Keyword(s):  
Fatty Acids ◽  
Blood Plasma ◽  
Free Fatty Acids ◽  
Estrous Cycle ◽  
Plasma Glucose

Qualitative and quantitative cuanyes of lipids in experimental peritonitis

1998 ◽  
Vol 79 (5) ◽  
pp. 382-387
Author(s):  
V. A. Trofimov ◽  
A. P. Vlasov ◽  
М. М. Minnebaev
Keyword(s):  
Fatty Acids ◽  
Blood Plasma ◽  
Free Fatty Acids ◽  
Considerable Increase ◽  
Membrane Lipids ◽  
Qualitative And Quantitative ◽  
Lipid Disorders ◽  
Tissue Lipids ◽  
Organ Level ◽  
Adequate Method

The considerable increase of the free fatty acids part, phospholipid lysophorms and the decrease of the main membrane lipids level are found in tissue lipids spectrum in peritonitis. The changes in organ li pids composition in many cases are correlated with those in blood plasma. Hence, the complex estimation of blood li pids is an adequate method of determining the biochemical lipid disorders intensity on the organ level.

Metabolic concomitants of hypophagia during recovery from insulin-induced obesity in rats

1983 ◽  
Vol 245 (3) ◽  
pp. E211-E219 ◽  
Author(s):  
I. Ramirez ◽  
M. I. Friedman
Keyword(s):  
Fatty Acids ◽  
Weight Loss ◽  
Food Intake ◽  
Free Fatty Acids ◽  
Insulin Treatment ◽  
Liver Glycogen ◽  
Diabetic Rats ◽  
Acid Oxidation ◽  
Hepatic Fatty Acid Oxidation ◽  
Adipose Tissue Lipoprotein Lipase

Rats were given daily injections of protamine-zinc insulin (PZI) that increased food intake and body weight. Termination of insulin treatment resulted in transient hypophagia and weight loss. Simultaneously with the weight loss, plasma levels of glycerol, free fatty acids, glucose, and ketones increased, whereas adipose tissue lipoprotein lipase activity and liver glycogen decreased. These changes in food intake and metabolism after termination of PZI treatment were accentuated in streptozotocin-diabetic rats. Two antilipolytic drugs (nicotinic acid and 3,5-dimethylpyrazole) blocked the elevation in plasma glycerol while having no effect on food intake. A 1-day fast after termination of insulin treatment equalized insulin-treated and control groups for plasma glycerol and ketones and reversed group differences in free fatty acids; the elevation in plasma glucose persisted despite starvation. Following starvation, previously PZI-treated rats ate less than controls on refeeding. The results show that enhanced lipolysis does not invariably accompany hypophagia during excess weight loss and suggest that a disturbance in carbohydrate metabolism or an increase in hepatic fatty acid oxidation may underlie this decrease in food intake.

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