scholarly journals Motor Restlessness

2020 ◽  
Author(s):  
Keyword(s):  
1990 ◽  
Vol 4 (1) ◽  
pp. 57-71 ◽  
Author(s):  
Britt Af Klinteberg ◽  
Daisy Schalling ◽  
David Magnusson

In a follow‐up study, teacher ratings of behaviour at 13 years of age were analysed in relation to personality factors in adults (mean age 27 years) for a group of 77 male and 84 female subjects. In the male group, ratings of Aggressiveness, Motor Restlessness, and Concentration Dificulties correlated positively with a broad psychopathy‐related factor. Furthermore, ratings of Timidity correlated positively with two anxiety factors. In the female group, ratings of Motor Restlessness correlated positively with an extraversion factor. The rated childhood behaviours and the adult personality factors were assumed to be a differential reflection of vulnerability to externalizing and internalizing psychosocial disturbances.


PEDIATRICS ◽  
1988 ◽  
Vol 82 (3) ◽  
pp. 364-366
Author(s):  
Gregory L. Kearns ◽  
Debra H. Fiser

The case of a 3-week-old male infant is described. After receiving an iatrogenic overdose of metoclopramide (1.0 mg/kg every six hours) throughout a 36-hour period for the treatment of suspected gastroesophageal reflux, he became cyanotic, lethargic, and irritable, he fed poorly, and he had diarrhea and respiratory distress. Methemoglobinemia (20.5%) and reduced oxyhemoglobin saturation (79%) were identified. The patient had an excellent clinical response following a single IV dose of methylene blue. Subsequently, methemoglobin reductase activity was normal and there was no measurable hemoglobin M. The diagnosis of methemoglobinemia should be considered in any infant receiving large doses of metoclopramide who has clinical findings of cyanosis, ashen color, or a history of lethargy and/or motor restlessness.


2016 ◽  
Vol 2016 ◽  
pp. 1-5 ◽  
Author(s):  
Daniel Kirkpatrick ◽  
Tyler Smith ◽  
Mitchell Kerfeld ◽  
Taylor Ramsdell ◽  
Hasnain Sadiq ◽  
...  

With less than 1% of patients who use benzodiazepines being affected, paradoxical responses to benzodiazepines are rare. In this case report, we outline the course of an 80-year-old female who developed a paradoxical response to benzodiazepines. Significant medical and psychiatric history includes anxiety, mood disorder, hypothyroidism, bilateral mastectomy, goiter removal, and triple bypass. The patient presented with mental status changes, anxiety, motor restlessness, and paranoia. Over time, a temporal relationship between the severity of the patient’s motor agitation and intake of alprazolam was observed. As doses of alprazolam were decreased, her motor agitation became less severe. In addition to motor agitation, the patient also demonstrated increased aggressiveness, a subjective feeling of restlessness, and increased talkativeness. As her dose of alprazolam decreased, many of the patient’s symptoms were observed to decrease. This case report also discusses theories regarding the pathophysiology of paradoxical reactions to benzodiazepines, known risk factors, and appropriate treatment.


PEDIATRICS ◽  
1972 ◽  
Vol 49 (5) ◽  
pp. 709-715
Author(s):  
Leon Eisenberg

Having reviewed what is known, what is only surmised, and what is not known about the psychopharmacology of stimulant drug use in children, the clinician must decide for which patient to use what drug for how long. Medical practice does not permit the physician the luxury of deferring decisions until knowledge is certain. His task is to weigh putative benefits against putative risks in a strategy designed to maximize the probability of improvement for a particular patient. The risks that concern the pediatrician are not only those visible in the short run during drug administration but include effects on development, effects which may not become apparent for some time after treatment has been discontinued. In the case of stimulant drugs, public controversy has centered on behavioral rather than pharmacological toxicity, both short and long run. In the short run, are the drugs being used indiscriminately to stifle independence and creativity among exceptional children? Over the long run, does childhood drug use predispose to adolescent drug addiction? Before attempting to answer these questions, let us first consider the medical indications for the use of stimulants and the mode of their administration. The clinical syndromes which respond to stimulants are characterized by motor restlessness, short attention span, poor impulse control, learning difficulties, and emotional lability. Current American Psychiatric Association diagnostic nomenclature1 includes the term: "hyperkinetic reaction of childhood" to describe this set of symptoms; the World Health Organization2 is proposing: "hyperkinetic syndrome." Both terms have the virtue of stressing the symptom constellation and of by-passing the uncertainties surrounding cause.


2012 ◽  
Vol 22 (12) ◽  
pp. 1142-1148 ◽  
Author(s):  
Amanda Freeman ◽  
Elaine Pranski ◽  
R. Daniel Miller ◽  
Sara Radmard ◽  
Doug Bernhard ◽  
...  

2010 ◽  
Vol 5 (5) ◽  
pp. 460-464 ◽  
Author(s):  
Atman Desai ◽  
David W. Nierenberg ◽  
Ann-Christine Duhaime

The authors describe the case of a 13-year-old boy who exhibited progressive disabling motor restlessness, torticollis, urinary symptoms, and confusion following a fall from a bicycle. The differential diagnosis of this striking symptom complex in this clinical context can be problematic. In this case, the symptoms ultimately appeared most consistent with severe akathisia resulting from a single administration of haloperidol used at an outside hospital to sedate the patient prior to a head CT scan. The literature on akathisia in pediatric patients, and especially in patients following acute head injury, is reviewed, with suggestions for an approach to these symptoms in this clinical setting.


2005 ◽  
Vol 20 (7) ◽  
pp. 899-901 ◽  
Author(s):  
Imad Ghorayeb ◽  
Federica Provini ◽  
Bernard Bioulac ◽  
François Tison

1976 ◽  
Vol 9 (2) ◽  
pp. 100-110 ◽  
Author(s):  
Larry P. Harris

This is a review of the pivotal position accorded attentional deficits in explaining the inferior school-related performances of the mild-to-moderate LD, ED, and MR child. Problems in defining attention are discussed and an operational definition is given which lists those behaviors commonly associated with (1) adaptation to classroom environments, which lead to (2) correct student responses to pertinent, task-relevant stimuli. Orientation response and stimulus selection theories are summarized, and three common methods of remediation — drug therapy, reduced environmental stimulation, and operant conditioning — are examined to determine their efficacy. While each method was found to reduce maladaptive behaviors (e.g., motor restlessness, eyes and ears involved off-task, irrelevant talking), none of them lead to significant increases in correct academic work. Several interpretations of this finding and its import for special educators are listed.


2016 ◽  
Vol 2016 ◽  
pp. 1-3
Author(s):  
Ali Shahriari ◽  
Maryam Khooshideh ◽  
Mahdi Sheikh

Electroconvulsive therapy (ECT) is a highly effective nonpharmacologic treatment for the management of depression and some other psychiatric disorders. Post-ECT agitation occurs in up to 12% of ECT treatments and is characterized by motor restlessness, irritability, disorientation, and panic-like behaviors. The severity of post-ECT agitation ranges from mild and self-limited to serious and severe forms requiring prompt medical intervention to protect the patient and the medical staff. In severe agitation medical management may be necessary which consists of using sedative agents, either benzodiazepines or propofol. The side-effects of these sedative agents, especially in the elderly population, necessitate finding ways that could help the prevention of the occurrence of agitation after ECT treatments. We report a 68-year-old female with major depression who was referred for ECT. She experienced severe post-ECT agitation requiring medical intervention after all ECT treatments. Administering of oral amlodipine (5 mg) one hour before ECT treatment successfully prevented the occurrence of post-ECT agitation in this patient. We briefly discuss the possible underlying mechanisms and pathophysiology of amlodipine in the prevention of post-ECT agitation.


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