scholarly journals Cystic Change

2020 ◽  
Author(s):  
Keyword(s):  
2018 ◽  
Vol 2018 ◽  
pp. 1-5
Author(s):  
Raoul Verzijl ◽  
Pim J. Bongers ◽  
Geetha Mukerji ◽  
Ozgur Mete ◽  
Karen M. Devon ◽  
...  

A 71-year-old man with known history of atrial fibrillation (treated with routine rivaroxaban therapy) was found to have incidental biochemical elevated calcium and parathyroid hormone (PTH) levels. His physical examination demonstrated the presence of a palpable right neck mass. Subsequent imaging studies revealed a large parathyroid mass as well as multiple bone lesions, raising the suspicion of parathyroid carcinoma. The anticoagulant therapy was stopped 5 days prior to his elective surgery. The night before his elective surgery, he presented to the emergency room with profound hypocalcemia. The surgery was postponed and rescheduled after calcium correction. Intraoperative findings and detailed histopathological examination revealed an infarcted 4.0 cm parathyroid adenoma with cystic change. His bony changes were related to brown tumors associated with long-standing hyperparathyroidism. Autoinfarction of a large parathyroid adenoma causing severe hypocalcemia is a rare phenomenon and may be considered in patients with large parathyroid adenomas after withdrawal of anticoagulants.


2008 ◽  
Vol 69 (9) ◽  
pp. 2347-2351
Author(s):  
Kazuhiro KATSUHARA ◽  
Shinya HARA ◽  
Yota YAMAMOTO ◽  
Shigeharu UEDA ◽  
Kenji NOBUHARA ◽  
...  

Author(s):  
P. Jane Clarke ◽  
R. Fiddes

Benign conditions of the breast are very common, but they cause great anxiety, often leading the patient to be concerned that she has breast cancer. Symptoms may include: (1) a mass in the breast, commonly due to fibroadenoma, benign cystic change, or macrocysts; (2) mastalgia; and (3) discharge from the nipple, which may be caused by hyperprolactinaemia, intraduct papilloma, or duct ectasia. Management involves exclusion of malignancy, often by triple assessment of any palpable abnormality (for clinical examination, radiological and pathological assessment, see ...


Chest Imaging ◽  
2019 ◽  
pp. 493-497
Author(s):  
Brett W. Carter

Glandular enlargement in the mediastinum is usually due to hyperplasia of thymus in the anterior mediastinum or thyroid enlargement with intramediastinal growth. Thymic enlargement is typically due to rebound hyperplasia associated with chemotherapy, radiation therapy, and stresses. Rebound thymic hyperplasia manifests as diffuse, symmetric enlargement of the thymus. MRI may be helpful in distinguishing thymic hyperplasia from neoplastic involvement of the thymus, as the former lose signal on opposed-phase T1-weighted MR imaging. Thyroid goiters may originate in the neck and migrate into the mediastinum or arise from an ectopic focus of mediastinal thyroid tissue. Goiters manifest as mediastinal masses that are similar in appearance to the thyroid gland, with intrinsic hyperdensity, hyperenhancement, foci of calcification and cystic change.


2019 ◽  
Vol 15 (1) ◽  
Author(s):  
Naruhiko Ueda ◽  
Tatsurou Tanaka ◽  
Masafumi Oda ◽  
Nao Wakasugi-Sato ◽  
Shinobu Matsumoto-Takeda ◽  
...  

Abstract Background The purpose of the present study was to describe the CT imaging findings of normal incisive canals and incisive canal cysts and propose cut-off values to differentiate between them. Methods A total of 220 normal subjects and 40 patients with incisive canal cysts on multi-detector row computed tomography (MDCT) were retrospectively analyzed. The shapes, sizes, anatomic variations, Hounsfield scale values, and so on of maxillary incisive canals and the sizes and Hounsfield scale values of maxillary incisive canal cysts were analyzed. Results A significant difference in sizes of maxillary incisive canals in normal subjects was found between males and females. The sizes of maxillary incisive canals were significantly wider during aging, but shapes, anatomic variations, and Hounsfield scale values in the maxillary incisive canals were not significantly different with aging. A significant difference in sizes but not Hounsfield scale values was found between normal maxillary incisive canals and maxillary incisive canal cysts. Based on a cut-off of over 6 mm in the width of incisive canals, maxillary incisive canal cysts could not be appropriately diagnosed for subjects over 60 years of age. Over 60 years of age, maxillary incisive canal cysts could be appropriately diagnosed based on a cut-off of over 7.1 mm in width of incisive canals. When maxillary incisive canals of the hourglass types were seen on sagittal sections, significantly more patients had maxillary incisive canal cysts than other types. Conclusion In coincidentally diagnosing asymptomatic incisive canal cysts on imaging, we should apply different cut-offs for the size of the maxillary incisive canal for patients over and under 60 years of age. Specifically, the cut-offs for the long axis of maxillary incisive canal cysts were 7.1 mm for patients over 60 years of age and 6.0 mm for those under 60 years of age. In addition, we should pay attention to wider canals with hourglass shapes as indicative of cystic change of maxillary incisive canals.


Author(s):  
Albert C. M. Ong ◽  
Timothy Ellam

Autosomal dominant polycystic kidney disease (ADPKD) is responsible for up to 10% of prevalent patients with end-stage renal disease (ESRD). It is characterized by the enlargement of multiple bilateral renal cysts, present in almost all patients by their fifth decade. Loin pain is a common symptom that may be caused by cyst growth, intracyst haemorrhage, nephrolithiasis, or infection. Gross haematuria is also a common feature, but usually settles spontaneously. Excretory impairment develops after extensive cystic change has occurred and progresses to ESRD in half of all affected patients by the age of 60. However, the onset of cystic change and rate of renal functional decline are highly variable between individuals. ADPKD associated with the PKD1 gene has an earlier average age of cyst development and ESRD than PKD2, but the two cannot be distinguished on clinical grounds. Polycystins 1 and 2 are expressed in various organs and extrarenal disease may be the presenting feature. Intracranial aneurysms are five times more common in patients with ADPKD, but rupture is infrequent. Liver cysts are present in most patients and may be complicated by haemorrhage or infection, though liver failure is very rare. Massive hepatic cystic disease is confined to women, reflecting stimulatory effects of oestrogen on hepatic cyst growth. Cardiovascular disease is the leading cause of death in ADPKD and vascular dysfunction is present in many patients even before the development of excretory impairment. However, despite the multisystem manifestations of ADPKD, survival from ESRD is better for patients with ADPKD than for other non-diabetic causes of kidney failure.


2019 ◽  
Vol 14 (11) ◽  
pp. 1315-1319 ◽  
Author(s):  
Tatsunori Asada ◽  
Takayuki Yamada ◽  
Reiko Kumano
Keyword(s):  

2020 ◽  
Vol 216 (4) ◽  
pp. 152832
Author(s):  
Masakazu Toya ◽  
Yuichi Yamada ◽  
Ryohei Yokoyama ◽  
Kenichi Taguchi ◽  
Kazuki Nabeshima ◽  
...  

Sign in / Sign up

Export Citation Format

Share Document