characterization of the adrenal gland pathology of carney complex, and molecular genetics of the disease.

1998 ◽  
Vol 24 (3-4) ◽  
pp. 863-864 ◽  
Author(s):  
Lawrence S. Kirschner ◽  
Susan E. Taymans ◽  
Constantine A. Stratakis
2020 ◽  
Vol 92 (12) ◽  
pp. 185-194
Author(s):  
S. A. Buryakina ◽  
N. V. Tarbaeva ◽  
N. N. Volevodz ◽  
G. G. Karmazanovsky ◽  
L. D. Kovalevich ◽  
...  

The adrenal incidentaloma is a lesion of a different etiology and found incidentally in patients who underwent a diagnostic study not about the disease of this organ. Lesions can be both hormonally inactive and hormonally active, can arise from different zones of the adrenal gland or have non-specific organ affiliation, can be benign or malignant. Computed tomography characterization of these lesions, especially the differential diagnosis of benign and malignant, is extremely important for the correct diagnosis in order to provide adequate management of the patient. The article presents the key computed tomography criteria that allow radiologist to characterize the lesion most accurately and consider appropriate diagnosis.


1995 ◽  
Vol 62 (2) ◽  
pp. 196-208
Author(s):  
A. D'Amico ◽  
V. Ficarra ◽  
F. Mastroeni ◽  
G. Caleffi ◽  
A. Porcaro ◽  
...  

The use of cytogenetics in the characterization of bladder tumours has made it possible to demonstrate that chromosomal alterations are correlated with stage and grade of the tumour and have a predictive value as regards both tumour recurrences and progression. In the last decade the chromosomes involved in the main aberrations have been identified, and a negative prognostic significance has been suggested for some chromosomal aberrations. The knowledge of cytogenetics has been deepened by the sophisticated methods of molecular genetics, that have discovered many oncogenes and suppressor genes probably involved in the development of bladder tumours. The most characteristic molecular alterations of these tumours are losses of genetic information on chromosomes 9, 11 and 17, as a consequence of deletions and/or mutations. Such alterations probably cause the loss and/or the inactivation of suppressor genes (partly hypothetic still) and could represent important predictive factors of tumour progression.


Endocrinology ◽  
1992 ◽  
Vol 130 (1) ◽  
pp. 475-483 ◽  
Author(s):  
A Larcher ◽  
C Delarue ◽  
F Homo-Delarche ◽  
S Kikuyama ◽  
G Kupryszewski ◽  
...  

Radiographics ◽  
2006 ◽  
Vol 26 (6) ◽  
pp. 1811-1824 ◽  
Author(s):  
Semin Chong ◽  
Kyung Soo Lee ◽  
Ha Young Kim ◽  
Yoon Kyung Kim ◽  
Byung-Tae Kim ◽  
...  

2006 ◽  
Vol 83 (3-4) ◽  
pp. 189-199 ◽  
Author(s):  
Sosipatros A. Boikos ◽  
Constantine A. Stratakis

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