scholarly journals A case of cat with traumatic alopecia that died due to severe pulmonary edema after administration of triamcinolone

2020 ◽  
Vol 26 (4) ◽  
pp. 205-207
Author(s):  
Chiaki Kitanaka ◽  
Ryota Asahina ◽  
Yuta Yamamoto ◽  
Kento Mitsuhashi ◽  
Mami Hiasa ◽  
...  
Keyword(s):  
Pulmonary Edema ◽  
Severe Pulmonary Edema

Lung and chest wall impedances in the dog in normal range of breathing: effects of pulmonary edema

1992 ◽  
Vol 73 (3) ◽  
pp. 1040-1046 ◽  
Author(s):  
G. M. Barnas ◽  
D. Stamenovic ◽  
K. R. Lutchen
Keyword(s):  
Oleic Acid ◽  
Pulmonary Edema ◽  
Chest Wall ◽  
Mean Airway Pressure ◽  
Normal Range ◽  
Normal Ranges ◽  
Before And After ◽  
Measured Resistance ◽  
The Right ◽  
Severe Pulmonary Edema

We evaluated the effect of pulmonary edema on the frequency (f) and tidal volume (VT) dependences of respiratory system mechanical properties in the normal ranges of breathing. We measured resistance and elastance of the lungs (RL and EL) and chest wall of four anesthetized-paralyzed dogs during sinusoidal volume oscillations at the trachea (50–300 ml, 0.2–2 Hz), delivered at a constant mean airway pressure. Measurements were made before and after severe pulmonary edema was produced by injection of 0.06 ml/kg oleic acid into the right atrium. Chest wall properties were not changed by the injection. Before oleic acid, EL increased slightly with increasing f in each dog but was independent of VT. RL decreased slightly and was independent of VT from 0.2 to 0.4 Hz, but above 0.4 Hz it tended to increase with increasing flow, presumably due to the airway contribution. After oleic acid injection, EL and RL increased greatly. Large negative dependences of EL on VT and of RL on f were also evident, so that EL and RL after oleic acid changed two- and fivefold, respectively, within the ranges of f and VT studied. We conclude that severe pulmonary edema changes lung properties so as to make behavior VT dependent (i.e., nonlinear) and very frequency dependent in the normal range of breathing.

Severe Pulmonary Edema due to Hyponatremia after Hysteroscopy; Rapid and Full Recovery

2014 ◽  
Vol 3 (3) ◽  
pp. 375
Author(s):  
Akcan Akkaya ◽  
Ahmet Karatas ◽  
Abdullah Demirhan ◽  
Tulay Ozlu ◽  
Murat Bilgi ◽  
...  
Keyword(s):  
Pulmonary Edema ◽  
Full Recovery ◽  
Severe Pulmonary Edema

Severe Pulmonary Edema Related to Dextran 40

2002 ◽  
Vol 49 (2) ◽  
pp. 221-222 ◽  
Author(s):  
Ferit Demirkan ◽  
??akir ??nal ◽  
Emrah Arslan ◽  
Mukadder ??alko??lu ◽  
??zlem Kandemir
Keyword(s):  
Pulmonary Edema ◽  
Dextran 40 ◽  
Severe Pulmonary Edema

scholarly journals STUDIES ON PULMONARY EDEMA

1937 ◽  
Vol 66 (4) ◽  
pp. 405-411 ◽  
Author(s):  
Sidney Farber
Keyword(s):  
Guinea Pig ◽  
Pulmonary Edema ◽  
Guinea Pigs ◽  
Artificial Respiration ◽  
Vasomotor Control ◽  
Pulmonary Vessels ◽  
Cervical Vagotomy ◽  
Vasomotor Nerves ◽  
Bilateral Vagotomy ◽  
Severe Pulmonary Edema

1. Guinea pigs die shortly after bilateral cervical vagotomy, even when continuous artificial respiration effected through a tracheal cannula is carried out. Death is caused by severe pulmonary edema and congestion. 2. Direct observation of the lungs after bilateral vagotomy demonstrates that pulmonary edema develops gradually and increases slowly in amount and severity. Congestion precedes and accompanies the development of the edema. 3. Neuropathic pulmonary edema in the guinea pig is caused by disturbance to or abolition of the pulmonary vasomotor nerves. 4. The evidence obtained by experiments on animals suggests that neuropathic pulmonary edema in man is caused by disturbances, either central or peripheral, to the vasomotor control of the pulmonary vessels.

Effect of pulmonary edema on transfer of gas from acinus to airways

1994 ◽  
Vol 76 (2) ◽  
pp. 560-564 ◽  
Author(s):  
G. M. Barnas ◽  
P. B. Randalls ◽  
F. C. Forrest ◽  
B. H. Hoff ◽  
P. L. Donahue ◽  
...  
Keyword(s):  
Oleic Acid ◽  
Pulmonary Edema ◽  
Rate Constant ◽  
Airway Pressure ◽  
Esophageal Pressure ◽  
Gas Transfer ◽  
Before And After ◽  
The Right ◽  
Severe Pulmonary Edema ◽  
133Xe Washout

We directly measured the effect of progressive pulmonary edema on gas transfer from the acinus by injecting 133Xe dissolved in saline through a pulmonary artery catheter into an acinar region with occluded blood flow and measuring 133Xe washout by gamma scintillation scanning. We measured washout in six anesthetized paralyzed dogs during mechanical ventilation with O2 before and after injection of 0.6 mg/kg of oleic acid into the right atrium, which induces severe pulmonary edema within 2 h. Changes in the elastance and resistance of the lung were also calculated from measurements of airway flow, airway pressure, and esophageal pressure. Before injection of oleic acid, the monoexponential rate constant for 133Xe washout was 3.6 +/- 1.4 (SE) min-1; from this we estimated that the rate of gas transfer of 133Xe from the acini was 1.0 l/min. The rate constant decreased gradually after the injection and was correlated with increases in elastance and resistance (r = -0.66) and decreases in alveolar PO2 (r = 0.71). At 2 h after injection, the rate constant (1.2 +/- 0.8 min-1) was lower than control (P < 0.01), and the rate of gas transfer of 133Xe from the acini was < 0.32 l/min. We conclude that resistance in the acini is increased during pulmonary edema and that it is correlated, in the oleic acid model, with changes in overall lung mechanical properties.

scholarly journals Rituximab Induced Pulmonary Edema Managed with Extracorporeal Life Support

2018 ◽  
Vol 2018 ◽  
pp. 1-4
Author(s):  
Jacob R. Miller ◽  
Warren Isakow ◽  
Muhammad F. Masood ◽  
Patrick Aguilar ◽  
Kristen M. Sanfilippo ◽  
...  
Keyword(s):  
Pulmonary Function ◽  
Pulmonary Edema ◽  
Thrombotic Thrombocytopenic Purpura ◽  
Life Support ◽  
Extracorporeal Life Support ◽  
Respiratory Acidosis ◽  
High Dose ◽  
Thrombocytopenic Purpura ◽  
Cardiorespiratory Failure ◽  
Severe Pulmonary Edema

Though rare, rituximab has been reported to induce severe pulmonary edema. We describe the first report of ECLS utilization for this indication. A 31-year-old female with severe thrombotic thrombocytopenic purpura developed florid pulmonary edema after rituximab infusion. Despite advanced ventilatory settings, she developed severe respiratory acidosis and remained hypoxemic with a significant vasopressor requirement. Since her pulmonary insult was likely transient, ECLS was considered. Due to combined cardiorespiratory failure, she received support with peripheral venoarterial ECLS. During her ECLS course, she received daily plasmapheresis and high dose steroids. Her pulmonary function recovered and she was decannulated after 8 days. She was discharged after 23 days without residual sequelae.

scholarly journals STUDIES ON PULMONARY EDEMA

1937 ◽  
Vol 66 (4) ◽  
pp. 397-404 ◽  
Author(s):  
Sidney Farber
Keyword(s):  
Pulmonary Edema ◽  
Clinical Features ◽  
Postmortem Examination ◽  
Acute Pulmonary Edema ◽  
Essential Factor ◽  
Laryngeal Paralysis ◽  
Cervical Vagotomy ◽  
Vagal Innervation ◽  
Bilateral Vagotomy ◽  
Severe Pulmonary Edema

1. Bilateral cervical vagotomy in rabbits soon leads to death, usually within 8 to 24 hours. 2. Gradually increasing dyspnea, crises with expulsion of frothy, serous or sanguineous fluid from the mouth and nose, and terminal asphyxia are the important clinical features. 3. Postmortem examination reveals severe acute pulmonary edema and congestion, variable amounts of bronchopneumonia, and evidences of aspiration of food and secretions. This picture is similar to that found in the lungs in the bulbar form of poliomyelitis. 4. These changes are brought about by a combination of factors secondary to bilateral vagotomy: laryngeal paralysis (aspiration of food, slow asphyxia); loss of the vagal innervation of the lungs. 5. Laryngeal paralysis is not an essential factor in the production of severe pulmonary edema and death following bilateral cervical vagotomy. 6. To denote the pathogenesis of this type of edema, the term neuropathic pulmonary edema is employed.

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