Mom's Smoking Linked to Hearing Defect

Science News ◽  
1993 ◽  
Vol 144 (2) ◽  
pp. 23
Author(s):  
K. A. Fackelmann
Keyword(s):  
PEDIATRICS ◽  
1967 ◽  
Vol 40 (5) ◽  
pp. 875-880
Author(s):  
Richard L. Goode ◽  
F. Mark Rafaty ◽  
F. Blair Simmons

The clinical course of hearing loss associated with retinitis pigmentosa is outlined in four brief case summaries. This incidence of hearing loss in retinitis pigmentosa is 10% and occurs several years before clinical visual abnormalities. A battery of audiometric tests all suggest that the hearing defect is within the cochlea and that it is not rapidly progressive. Onset age has not been established. The employment of more than routine hearing tests, testing of other family members, electroretinograms, and historical information about certain features of familial vision are useful in establishing early diagnosis.


1982 ◽  
Vol 21 (3) ◽  
pp. 197-203 ◽  
Author(s):  
Moshe Feinmesser ◽  
Lilly Tell ◽  
Haya Levi
Keyword(s):  

2017 ◽  
Vol 2(5) (2017) ◽  
pp. 149-155
Author(s):  
Liudmyla Stasiuk ◽  
◽  
Maryna Sliusar ◽  
Keyword(s):  

Author(s):  
Dr. Kshitiz Chourasia ◽  
Dr. Kumar Girendrae ◽  
Dr. Vinod Patel ◽  
Dr. Mudra Khare

Background: A retrospective and prospective study including LPA /AFB CULTURE proven cases of MDR TB. Data will be collected from the patient attending the OPD & IPD in the Dept. of Respiratory Medicine Index Medical College, Indore. Result: Out of 226 patients, 183 patients continue on treatment and 8 patients get cure and 12 patients dead and 12 patients defaulted the treatment. Out of 226 patients, the present study shows that Gastrointestinal side effects are the most common 42.5%. 12 patients have renal toxicity and 8 patients have Arthalgia and 6 patients have cutaneous reactions and 14 patients developed hepatitis and 7 patients have some psychiatric illenss and 7 patients developed hearing defect. Conclusion: Overall, these findings emphasize the importance of continuing the systematic surveillance of M. tuberculosis isolates to monitor the trends of drug resistance in different patient categories as well as its association with  HIV across the country to timely modify and strengthen the national programs in order to prevent the emergence of MDR-TB strains and avert the threat of XDR-TB. Keywords: Profile, second Line Drug & TB.


Neurosurgery ◽  
1984 ◽  
Vol 15 (1) ◽  
pp. 108-110 ◽  
Author(s):  
Diana L. Ross ◽  
John M. Tew ◽  
Corning Benton ◽  
Craig Eisentrout

Abstract A 5-year-old girl presented with headache, vomiting, flushing, ptosis, and paroxysmal tachycardia. The neurological findings were partial motor and sensory left trigeminal palsy, left conductive hearing defect, and left cerebellar deficit. The radiological and neuropathological findings were typical for trigeminal schwannoma, which is rare in childhood. The present patient is the youngest yet recorded.


1975 ◽  
Vol 78 (4) ◽  
pp. 705-713 ◽  
Author(s):  
B. M. Goslings ◽  
R. Djokomoeljanto ◽  
R. Hoedijono ◽  
H. Soepardjo ◽  
A. Querido

ABSTRACT An audiometric study was carried out in a community of 642 subjects severely affected by endemic goitre and cretinism. Hearing loss was measured in 34 out of 41 subjects diagnosed as cretins, 92 normal subjects aged 5–20 years from the same community and 54 subjects (also of 5–20 years) living in a nearby control area without endemic goitre. The excess number of hearing defects in the endemic area seems to be entirely due to the process that leads to cretinism. There is no reason to describe deafness and deafmutism in an area with severe endemic goitre as a separate entity. The hearing defect showed a definite greater loss in the higher frequencies than in the lower frequencies and was found in 92 % of the cretins. Deafmutism was present in 5, a loss of more than 60 db in 8, a loss of 40–60 db in 10 cretins. A loss of 20–30 db was found in 2.2 % of normal subjects in the endemic area and 1.8 % of those living in the control area. It is concluded that audiometry is a simple and significant test to establish the presence of the neurological form of endemic cretinism, which is the most prevalent form in most endemias. The differential diagnosis and pathogenesis of the described hearing defect are discussed.


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