Mortality Rates of a Canada Goose Population in New Zealand

1968 ◽  
Vol 32 (2) ◽  
pp. 256 ◽  
Author(s):  
M. J. Imber ◽  
G. R. Williams
Author(s):  
Rajani P. Agadi ◽  
A. S. Talawar

In the present paper we consider an application of stochastic differential equation to model age-specific mortalities. We use New Zealand mortality data for the period 1948–2015 to fit the model. The point predictions of mortality rates at ages 40, 60 and 80 are quite good, almost undistinguishable from the true mortality rates observed.  


2018 ◽  
Vol 19 (3-4) ◽  
pp. 285-293 ◽  
Author(s):  
Maize C. Cao ◽  
Andrew Chancellor ◽  
Alison Charleston ◽  
Mike Dragunow ◽  
Emma L. Scotter

1999 ◽  
Vol 26 (1) ◽  
pp. 75 ◽  
Author(s):  
Richard P. Heyward ◽  
Grant L. Norbury

The incidence of secondary poisoning was determined by using radio-telemetry to assess the survival of 68 ferrets and 21 cats on two treatment sites and one control site in the dry tussock grasslands of New Zealand. The treatment sites were aerially poisoned with 1080-coated carrot baits (0.02% wt/wt) to control rabbits. The control site was not poisoned. Ferrets and cats were monitored at two-weekly intervals for at least 1 month before, and 2 months after the poison operations. Muscle samples from ferrets and cats that died within 50 days of poisoning on the treatment sites were assayed for 1080. In all, 7–11% (n = 28) of ferrets on one site and 8–15% (n = 26) of ferrets at the other site apparently died of secondary 1080 poisoning. Natural mortality rates of ferrets were 46–81% per annum. While we have evidence that secondary poisoning of cats does occur, we monitored insufficient numbers of cats to reliably estimate mortality rates. Declines in predator numbers are commonly observed after rabbit poisoning. This study indicates that secondary poisoning contributes to these declines.


2006 ◽  
Vol 80 (1) ◽  
pp. 33-40 ◽  
Author(s):  
N.E. Davis

AbstractWaterfowl from Lake Wanaka and the Waitaki Lakes watershed of New Zealand's South Island were surveyed to find natural hosts with adult echinostomes and schistosomes to provide sufficient numbers of eggs for laboratory studies. The Canada goose (Branta canadensis) was found to host an echinostome determined to be a New Zealand strain of Echinostoma revolutum. The New Zealand scaup (Aythya novaeseelandia) concurrently hosts three species of echinostomes (E. revolutum, Echinoparyphium cinctum and E. recurvatum) plus two species of avian schistosomes (Trichobilharzia sp. and Dendritobilharzia pulverulenta). In the Canada goose and the New Zealand scaup, adult gravid echinostomes predominated over juveniles. In other waterfowl surveyed, very few echinostomes were found, with juveniles predominating.


2014 ◽  
Vol 2014 ◽  
pp. 1-6
Author(s):  
Brian Cox ◽  
Chih-Wei Liu ◽  
Mary J. Sneyd ◽  
Claire M. Cameron

Background. Non-Hodgkin lymphoma (NHL) incidence rates have increased considerably in New Zealand.Methods. Incidence and mortality rates for NHL from 1981 to 2010 were calculated. Trends in age-specific rates were analysed and age-period-cohort models fitted to explore generation-specific changes in incidence and mortality.Results. NHL incidence increased by 67% for men and 74% for women between the 1981–1985 and 2006–2010 time periods in New Zealand. For women born about 1936 and men born about 1946, NHL incidence and mortality have diverged suggesting an improved prognosis for recent generations.Conclusion. The strong generation effects suggest that an exposure before 25 years of age is of major importance in determining the lifetime risk of NHL in New Zealand. NHL incidence rates in New Zealand will continue to increase in the future and probably more in females than males, as generations with increased risk age. Current hypotheses for the cause of NHL do not explain the trends observed. A decline in the prevalence of a protective factor may have also contributed to these trends. Examination of trends for subtypes of NHL and innovative testable hypotheses that may explain these trends are needed.


Author(s):  
Victor Khou ◽  
Nicole L De La Mata ◽  
Rachael L Morton ◽  
Patrick J Kelly ◽  
Angela C Webster

Abstract Background Withdrawal from renal replacement therapy is common in patients with end-stage kidney disease (ESKD), but end-of-life service planning is challenging without population-specific data. We aimed to describe mortality after treatment withdrawal in Australian and New Zealand ESKD patients and evaluate death-certified causes of death. Methods We performed a retrospective cohort study on incident patients with ESKD in Australia, 1980–2013, and New Zealand, 1988–2012, from the Australian and New Zealand Dialysis and Transplant registry. We estimated mortality rates (by age, sex, calendar year and country) and summarized withdrawal-related deaths within 12 months of treatment modality change. Certified causes of death were ascertained from data linkage with the Australian National Death Index and New Zealand Mortality Collection database. Results Of 60 823 patients with ESKD, there were 8111 treatment withdrawal deaths and 26 207 other deaths over 381 874 person-years. Withdrawal-related mortality rates were higher in females and older age groups. Rates increased between 1995 and 2013, from 1142 (95% confidence interval 1064–1226) to 2706/100 000 person-years (95% confidence interval 2498–2932), with the greatest increase in 1995–2006. A third of withdrawal deaths occurred within 12 months of treatment modality change. The national death registers reported kidney failure as the underlying cause of death in 20% of withdrawal cases, with other causes including diabetes (21%) and hypertensive disease (7%). Kidney disease was not mentioned for 18% of withdrawal patients. Conclusions Treatment withdrawal represents 24% of ESKD deaths and has more than doubled in rate since 1988. Population data may supplement, but not replace, clinical data for end-of-life kidney-related service planning.


2002 ◽  
Vol 29 (4) ◽  
pp. 323 ◽  
Author(s):  
Peter Caley ◽  
L. M. McElrea ◽  
Jim Hone

Life-table data from feral ferret populations in New Zealand were analysed to estimate their mortality rates, and to test for any additive effect of Mycobacterium bovis infection on observed mortality rates. The observed instantaneous mortality rate was best estimated by modelling mortality as a 2-phase step model with different rates for juveniles (μ1 = 1.45 year–1, 95% C.I. 1.2–1.7 year–1) and adults (μ2 = 0.55 year–1, 95% C.I. 0.4–0.9 year–1). This corresponds to a survival probability of 0.25 during the first year of life, rising to 0.55 year–1 thereafter, and a life expectancy of 0.95 years. At a population level, no additional mortality due to M. bovis infection was observed, suggesting either that the rate of disease-induced mortality was negligible, or that it was compensatory with natural mortality.


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